scholarly journals Association Between Admission Serum Phosphate Level and All-Cause Mortality Among Patients with Spontaneous Intracerebral Hemorrhage

2021 ◽  
Vol Volume 14 ◽  
pp. 3739-3746
Author(s):  
Yu Hong ◽  
Xian-Hui Wang ◽  
Yi-Tong Xiong ◽  
Jie Li ◽  
Chun-feng Liu
Keyword(s):  
Intracerebral Hemorrhage ◽  
Serum Phosphate ◽  
Phosphate Level ◽  
Serum Phosphate Level ◽  
Spontaneous Intracerebral Hemorrhage ◽  
All Cause Mortality

Effects of serum phosphate level on formation of incisor dentine in hypophosphatemic mice

1996 ◽  
Vol 25 (4) ◽  
pp. 182-187 ◽  
Author(s):  
Y. Masatomi ◽  
Y. Nakagawa ◽  
Y. Kanamoto ◽  
S. Sobue ◽  
T. Ooshima
Keyword(s):  
Serum Phosphate ◽  
Phosphate Level ◽  
Serum Phosphate Level

Prognostic value of serum phosphate level in adult patients resuscitated from cardiac arrest

Resuscitation ◽  
2018 ◽  
Vol 128 ◽  
pp. 56-62 ◽  
Author(s):  
Yong Hun Jung ◽  
Byung Kook Lee ◽  
Kyung Woon Jeung ◽  
Chun Song Youn ◽  
Dong Hun Lee ◽  
...  
Keyword(s):  
Cardiac Arrest ◽  
Prognostic Value ◽  
Serum Phosphate ◽  
Adult Patients ◽  
Phosphate Level ◽  
Serum Phosphate Level

How do we sense phosphate to regulate serum phosphate level?

2019 ◽  
Vol 38 (1) ◽  
pp. 1-6
Author(s):  
Seiji Fukumoto ◽  
Yuichi Takashi ◽  
Maria K. Tsoumpra ◽  
Shun Sawatsubashi ◽  
Toshio Matsumoto
Keyword(s):  
Serum Phosphate ◽  
Phosphate Level ◽  
Serum Phosphate Level

scholarly journals Relation Between Serum Phosphate Level and Cardiovascular Event Rate in People With Coronary Disease

Circulation ◽  
2005 ◽  
Vol 112 (17) ◽  
pp. 2627-2633 ◽  
Author(s):  
Marcello Tonelli ◽  
Frank Sacks ◽  
Marc Pfeffer ◽  
Zhiwei Gao ◽  
Gary Curhan
Keyword(s):  
Coronary Disease ◽  
Cardiovascular Event ◽  
Event Rate ◽  
Serum Phosphate ◽  
Phosphate Level ◽  
Serum Phosphate Level

Serum phosphate level in burn patients

Burns ◽  
2010 ◽  
Vol 36 (7) ◽  
pp. 1112-1115 ◽  
Author(s):  
Shahriar Loghmani ◽  
Mohammad Reza Maracy ◽  
Reza Kheirmand
Keyword(s):  
Serum Phosphate ◽  
Phosphate Level ◽  
Serum Phosphate Level ◽  
Burn Patients

scholarly journals Deterioration of Cerebral Oxygenation by Aortic Arch Calcification Progression in Patients Undergoing Hemodialysis: A Cross-Sectional Study

2017 ◽  
Vol 2017 ◽  
pp. 1-6 ◽  
Author(s):  
Kiyonori Ito ◽  
Susumu Ookawara ◽  
Tomohisa Okochi ◽  
Yuichiro Ueda ◽  
Masaya Kofuji ◽  
...  
Keyword(s):  
Aortic Arch ◽  
Cerebral Oxygenation ◽  
Univariate Analysis ◽  
Multivariate Logistic Regression Analysis ◽  
Serum Phosphate ◽  
Phosphate Level ◽  
Serum Phosphate Level ◽  
History Of ◽  
Grade 3 ◽  
Aortic Arch Calcification

Background. Near-infrared spectroscopy revealed that the regional saturation of oxygen (rSO2) in cerebral tissue is lower in hemodialysis (HD) patients than in healthy subjects. However, no study has examined the changes in cerebral oxygenation by aortic arch calcification (AAC) progression in HD patients. Methods. A total of 104 HD patients were divided into four groups by AAC grade determined using chest radiography: 23 patients at grade 0, 24 at grade 1, 30 at grade 2, and 27 at grade 3. Differences in clinical parameters, including cerebral rSO2, among AAC grades were investigated and atherosclerotic parameters affecting cerebral rSO2 values were identified. Results. Cerebral rSO2 significantly decreased as AAC progressed (AAC grade 3 versus grade 0, p<0.01 versus grade 1, p<0.05). Multivariate logistic regression analysis was performed using parameters with p values < 0.20 in univariate analysis between cerebral rSO2 values less than the mean and atherosclerotic parameters. AAC grades 2 and 3, serum phosphate level, and history of smoking were factors associated with the cerebral rSO2 decrease. Conclusions. Cerebral rSO2 significantly decreased as AAC progressed and was independently associated with higher AAC grade, serum phosphate level, and history of smoking.

scholarly journals Genotype and Phenotype Analysis in X-Linked Hypophosphatemia

2021 ◽  
Vol 9 ◽  
Author(s):  
Peong Gang Park ◽  
Seon Hee Lim ◽  
HyunKyung Lee ◽  
Yo Han Ahn ◽  
Hae Il Cheong ◽  
...  
Keyword(s):  
Serum Phosphate ◽  
Hypophosphatemic Rickets ◽  
Phosphate Level ◽  
Serum Phosphate Level ◽  
Onset Age ◽  
Presenting Symptoms ◽  
Truncating Mutation ◽  
Mutation Group

Background: X-linked hypophosphatemia (XLH) is the most frequent form of hypophosphatemic rickets and is caused by mutations in the PHEX gene. We analyzed genotype-phenotype correlations in XLH patients with proven PHEX mutations.Methods:PHEX mutations were detected in 55 out of 81 patients who clinically presented with hypophosphatemic rickets. The patients were grouped into nontruncating (n = 9) and truncating (n = 46) mutation groups; their initial presentation as well as long-term clinical findings were evaluated according to these groups.Results: Initial findings, including presenting symptoms, onset age, height standard deviation scores (SDS), and laboratory tests, including serum phosphate level and tubular resorption of phosphate, were not significantly different between the two groups (onset age: nontruncating mutation group, 2.0 years, truncating mutation group, 2.2 years; height SDS: nontruncating mutation group, −1.9, truncating mutation group, −1.7; serum phosphate: nontruncating mutation group, 2.5 mg/dL, truncating mutation group, 2.6 mg/dL). However, at their last follow-up, the serum phosphate level was significantly lower in patients with truncating mutations (nontruncating mutation group: 3.2 mg/dl, truncating mutation group: 2.3 mg/dl; P = 0.006). Additionally, 62.5% of patients with truncating mutations developed nephrocalcinosis at their last follow-up, while none of the patients with nontruncating mutations developed nephrocalcinosis (P = 0.015). Orthopedic surgery due to bony deformations was performed significantly more often in patients with truncating mutations (52.3 vs. 10.0%, P = 0.019).Conclusion: Although considerable inconsistency exists regarding the correlation of truncating mutations and their disease phenotype in several other studies, we cautiously suggest that there would be genotype-phenotype correlation in some aspects of disease manifestation after long-term follow-up. This information can be used when consulting patients with confirmed XLH regarding their disease prognosis.

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