scholarly journals Association between dental pulp stones and calcifying nanoparticles

2011 ◽  
pp. 109 ◽  
Author(s):  
Yang ◽  
Zeng ◽  
Yang ◽  
Zhang ◽  
Gong ◽  
...  
2012 ◽  
Vol 2012 ◽  
pp. 1-4 ◽  
Author(s):  
Mohita Marwaha ◽  
Radhika Chopra ◽  
Payal Chaudhuri ◽  
Atul Gupta ◽  
Jayna Sachdev

Pulp stones are foci of calcification or discrete calcifications in the dental pulp. They are frequently found on bitewing and periapical radiographs, but their occurrence in entire dentition is unusual. We are reporting four cases in which the occurrence of pulp stones ranged from their presence in just primary teeth (Cases 1 and 2) to involvement of young permanent teeth also (Case 3) and even unerupted permanent teeth (Case 4). In all the cases, dental, medical, and family histories as well as the findings from the clinical examination of the patient were not contributory. Histopathological report revealed true denticle. Metabolic evaluation of patients through liver function test, kidney function test, and blood investigation did not show any metabolic disorders. Patients were also evaluated for any systemic, syndromic, or genetic involvement, but this was also noncontributing. Therefore, it is suggested that these unusual cases may be of idiopathic origin.


2007 ◽  
Vol 86 (10) ◽  
pp. 951-955 ◽  
Author(s):  
K.M. Galler ◽  
A. Yasue ◽  
A.C. Cavender ◽  
P. Bialek ◽  
G. Karsenty ◽  
...  

The molecular mechanisms that maintain the equilibrium of odontoblast progenitor cells in dental pulp are unknown. Here we tested whether homeostasis in dental pulp is modulated by Twist-1, a nuclear protein that partners with Runx2 during osteoblast differentiation. Our analysis of Twist-1(+/−) mice revealed phenotypic changes that involved an earlier onset of dentin matrix formation, increased alkaline phosphatase activity, and pulp stones within the pulp. RT-PCR analyses revealed Twist-1 expression in several adult organs, including pulp. Decreased levels of Twist-1 led to higher levels of type I collagen and Dspp gene expression in perivascular cells associated with the pulp stones. In mice heterozygous for both Twist-1 and Runx2 inactivation, the phenotype of pulp stones appeared completely rescued. These findings suggest that Twist-1 plays a key role in restraining odontoblast differentiation, thus maintaining homeostasis in dental pulp. Furthermore, Twist-1 functions in dental pulp are dependent on its interaction with Runx2.


2013 ◽  
Vol 24 (3) ◽  
pp. 393
Author(s):  
Neeta Sharma
Keyword(s):  

2002 ◽  
Vol 109 (1) ◽  
pp. 56-60 ◽  
Author(s):  
Piranit N. Kantaputra ◽  
Akira Kinoshita ◽  
Chanin Limwonges ◽  
Oranud Praditsup ◽  
Norio Niikawa

Diseases ◽  
2019 ◽  
Vol 7 (3) ◽  
pp. 50 ◽  
Author(s):  
Ali Alsweed ◽  
Randa Farah ◽  
Satheeshkumar PS ◽  
Rafat Farah

Background: This study sought to determine the prevalence of carotid artery calcifications (CACs) and pulp stones detected on panoramic radiographs (PRs) and ascertain their correlation. Methods: A total of 2013 digital PRs were retrospectively retrieved and thoroughly examined to determine the prevalence of CACs and pulp stones, their correlation with patient age and gender, and the relationship between the presence of pulps stones and radiographically detectable CACs. Results: The prevalence of CACs on PRs was 2.0%; the prevalence of pulp stones was 4.6%. There was no statistical relationship between pulp stones and CACs (p = 0.714). Older patients exhibited a significantly higher prevalence of CACs than younger patients (p < 0.001); pulp stones were statistically more prevalent in younger patients than older patients (p = 0.001). There were no significant differences between male and females in terms of the prevalence of either CACs or pulp stones (p = 0.087 and p = 0.278, respectively). Conclusions: Dentists should be trained to detect CACs on PRs belonging to patients older than 40 to exclude the presence of CACs. Moreover, pulp stones do not function as a diagnostic marker for CACs.


Health ◽  
2015 ◽  
Vol 07 (03) ◽  
pp. 336-345 ◽  
Author(s):  
Fatemeh Ezoddini-Ardakani ◽  
Seyedeh Mahdieh Nemayandeh ◽  
Seyed Mahmood Sadrbafghi ◽  
Sedigheh Hajihashemi ◽  
Mahmood Emami ◽  
...  

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