scholarly journals Conductivity Properties of Selected Aliphatic Monocarboxylic Acid Anions in Water at 298.15 K

2020 ◽  
pp. 10007-10027
Author(s):  
Zdzisław Kinart ◽  
Keyword(s):  
Monocarboxylic Acid

Uptake of phenoxyacetic acid derivatives into Caco-2 cells by the monocarboxylic acid transporters

2009 ◽  
Vol 189 (2) ◽  
pp. 102-109 ◽  
Author(s):  
Osamu Kimura ◽  
Kensuke Tsukagoshi ◽  
Tetsuya Endo
Keyword(s):  
Monocarboxylic Acid ◽  
Phenoxyacetic Acid

HPLC enantiomer separation of a chiral 1,4-dihydropyridine monocarboxylic acid

2004 ◽  
Vol 35 (2) ◽  
pp. 259-266 ◽  
Author(s):  
Michael Lämmerhofer ◽  
Olle Gyllenhaal ◽  
Wolfgang Lindner
Keyword(s):  
Enantiomer Separation ◽  
Monocarboxylic Acid

Changes in the Blood of the Rat induced by the Monocarboxylic Acid of Cyanocobalamin obtained by Fermentation (anti-Vitamin B12)

Nature ◽  
1962 ◽  
Vol 194 (4829) ◽  
pp. 689-690 ◽  
Author(s):  
E. CSÁNYI ◽  
M. A. KELEMEN ◽  
J. BORSY
Keyword(s):  
Vitamin B12 ◽  
Monocarboxylic Acid

Monocarboxylic Acid Transport

2013 ◽  
pp. 1611-1643 ◽  
Author(s):  
Andrew P. Halestrap
Keyword(s):  
Monocarboxylic Acid ◽  
Acid Transport

Regional blood-brain barrier transport of ketone bodies in portacaval-shunted rats

1991 ◽  
Vol 261 (5) ◽  
pp. E647-E652 ◽  
Author(s):  
R. A. Hawkins ◽  
A. M. Mans
Keyword(s):  
Blood Brain Barrier ◽  
Ketone Bodies ◽  
Monocarboxylic Acid ◽  
Brain Barrier ◽  
Substantial Contribution ◽  
Sham Operation ◽  
Acid System ◽  
Portacaval Shunting ◽  
Blood Brain ◽  
The Brain

The permeability of the blood-brain barrier to ketone bodies, substrates of the monocarboxylic acid carrier, was measured in individual brain structures of control and portacaval-shunted rats. The measurements were made 5-7 wk after the shunt or sham operation by quantitative autoradiography. Portacaval shunting caused the permeability to ketone bodies to decrease throughout the brain by approximately 70%. There was a striking change in the transport pattern in the cerebral cortex; deeper cortical layers were affected more than superficial layers. Ketone body consumption by brain is limited by the transport capacity of the monocarboxylic acid system. Therefore, in portacaval-shunted rats the very low activity of this system makes it unlikely that ketone bodies can make a substantial contribution during situations such as fasting. Likewise, other substrates of the monocarboxylic acid system, e.g., lactate and pyruvate, will have greatly restricted access to the brain after portacaval shunting. If the carrier is symmetrical, another consequence will be that exit of endogenously produced lactate will be retarded.

Sign in / Sign up

Export Citation Format

Share Document