scholarly journals COVID-19 induced liver function abnormality associates with age

Aging ◽  
2020 ◽  
Vol 12 (14) ◽  
pp. 13895-13904
Author(s):  
Shasha Li ◽  
Jinsong Li ◽  
Zhenhua Zhang ◽  
Lin Tan ◽  
Tuo Shao ◽  
...  
Keyword(s):  
Liver Function ◽  
Liver Function Abnormality

scholarly journals Hyperferritinemia and Hyperuricemia May Be Associated with Liver Function Abnormality in Obese Adolescents

PLoS ONE ◽  
2012 ◽  
Vol 7 (10) ◽  
pp. e48645 ◽  
Author(s):  
Solomon Chih Cheng Chen ◽  
Ya Fang Huang ◽  
Jung Der Wang
Keyword(s):  
Liver Function ◽  
Liver Function Abnormality ◽  
Obese Adolescents

Liver function abnormality & Vestibular symptoms with Azithromycin

2011 ◽  
Vol 1 (6) ◽  
pp. 205-206
Author(s):  
Dr. Parveen Kumar Sharma ◽  
◽  
Dr. Rekha Bansal
Keyword(s):  
Liver Function ◽  
Liver Function Abnormality ◽  
Vestibular Symptoms

scholarly journals Correction: Hyperferritinemia and Hyperuricemia May Be Associated with Liver Function Abnormality in Obese Adolescents

PLoS ONE ◽  
2017 ◽  
Vol 12 (3) ◽  
pp. e0173849
Author(s):  
Solomon Chih Cheng Chen ◽  
Ya Fang Huang ◽  
Jung Der Wang
Keyword(s):  
Liver Function ◽  
Liver Function Abnormality ◽  
Obese Adolescents

Acetaminophen: Macula densa hypersecretory activity by induced hepatotoxicity

1987 ◽  
Vol 45 ◽  
pp. 934-935
Author(s):  
S.S. Poolsawat ◽  
C.A. Huerta ◽  
S.TY. Lae ◽  
G.A. Miranda
Keyword(s):  
Cell Proliferation ◽  
Liver Function ◽  
Chronic Inflammation ◽  
Foam Cell ◽  
Term Effect ◽  
Short Term ◽  
Drug Induced ◽  
Experimental Induction ◽  
Tissue Alterations ◽  
Toxic Responses

Introduction. Experimental induction of altered histology by chemical toxins is of particular importance if its outcome resembles histopathological phenomena. Hepatotoxic drugs and chemicals are agents that can be converted by the liver into various metabolites which consequently evoke toxic responses. Very often, these drugs are intentionally administered to resolve an illness unrelated to liver function. Because of hepatic detoxification, the resulting metabolites are suggested to be integrated into the macromolecular processes of liver function and cause an array of cellular and tissue alterations, such as increased cytoplasmic lysis, centrilobular and localized necroses, chronic inflammation and “foam cell” proliferation of the hepatic sinusoids (1-4).Most experimentally drug-induced toxicity studies have concentrated primarily on the hepatic response, frequently overlooking other physiological phenomena which are directly related to liver function. Categorically, many studies have been short-term effect investigations which seldom have followed up the complications to other tissues and organs when the liver has failed to function normally.

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