Erratum toRevised Tectonic Forecast of Global Shallow Seismicity Based on Version 2.1 of the Global Strain Rate Map: Table 1

Author(s):  
Peter Bird ◽  
Corné Kreemer
Circulation ◽  
2007 ◽  
Vol 116 (suppl_16) ◽  
Author(s):  
Michael Dandel ◽  
Hans Lehmkuhl ◽  
Christoph Knosalla ◽  
Roland Hetzer

Background: Non-Doppler based two-dimensional (2D) strain imaging can reveal wall motion alterations not visible by conventional echocardiography (ECHO) and like tissue Doppler imaging (TDI) it can detect myocardial deformation (regional shortening and thickening) in the absence of visible ventricular wall displacement. Because TDI is dependent on the direction of Doppler angle of incidence in relation to myocardial motion, in our attempt to assess of the diagnostic value of strain and strain rate changes for early detection of transplant coronary arteriopathy (TCA), we focused our attention on the angle independent 2D strain imaging. Methods: In 68 heart recipients with normal LV wall motion and ejection fraction in conventional ECHO, 2D strain LV wall motion analysis were additionally performed before each follow-up cardiac catheterization. Circumferential, radial and longitudinal strain and strain rate were calculated from parasternal short axes and apical (3- and 4-chamber) views, respectively. 2D strain parameters were tested for relationships with angiographic findings. Results: In comparison with patients without TCA, those with angiographic TCA showed lower global systolic strain rate (radial, circumferential and longitudinal) values and longer systolic times measured from onset of contraction to the peak of systolic strain (p<0.01). For radial peak systolic global strain rate values below 1.1/s we found a 93.3% likelihood of angiographic TCA in general, regardless of the presence or absence of focal stenoses on main coronaries. Regional strain and strain rate analyses showed differences in contraction asynchrony and dyssynergy indexes between patients with and without focal stenoses (p<0.01). For longitudinal midsystolic dyssynchrony index values >0.5 we found an 88.2% likelihood of relevant focal coronary stenoses (>50% narrowing). Conclusions: In heart allografts with apparently normal LV kinetics in conventional ECHO, simple global strain rate measurements allow early TCA prediction, but without the ability to differentiate between diffuse TCA and predominantly focal coronary stenoses. Such differentiation is possible with more complex regional strain analyses using contraction asynchrony and dyssynergy indexes.


2014 ◽  
Vol 15 (10) ◽  
pp. 3849-3889 ◽  
Author(s):  
Corné Kreemer ◽  
Geoffrey Blewitt ◽  
Elliot C. Klein

2010 ◽  
Vol 11 (9) ◽  
pp. 743-751 ◽  
Author(s):  
M. Kasner ◽  
R. Gaub ◽  
D. Sinning ◽  
D. Westermann ◽  
P. Steendijk ◽  
...  

2011 ◽  
Vol 29 (4) ◽  
pp. 404-410 ◽  
Author(s):  
Koichi Kimura ◽  
Katsu Takenaka ◽  
Aya Ebihara ◽  
Tomoko Okano ◽  
Kansei Uno ◽  
...  

2000 ◽  
Vol 52 (10) ◽  
pp. 765-770 ◽  
Author(s):  
Corné Kreemer ◽  
John Haines ◽  
William E. Holt ◽  
Geoffrey Blewitt ◽  
David Lavallee

Circulation ◽  
2007 ◽  
Vol 116 (suppl_16) ◽  
Author(s):  
Takeshi Takamura ◽  
Kaoru Dohi ◽  
Katsuya Onishi ◽  
Naoki Fujimoto ◽  
Tairo Kurita ◽  
...  

Background: We tested the hypothesis that global strain rate imaging can quantify and stratify the severity of left ventricular (LV) relaxation abnormality in patients with left ventricular hypertrophy (LVH) ranged from normal to reduced LV systolic function independently from longitudinal (L), circumferential (C), and radial axes (R). Methods: Fifty-seven patients with hypertensive LVH and thirty age matched controls (Control, EF 65 ± 5 %) had echo-study with speckle tracking strain and strain rate imaging from (L), (C), and (R). LVH were divided into two groups; normal EF (LVH-NEF) defined as EF ≥ 55% (n = 35, EF 64 = 5 %), and systolic dysfunction (LVH-SD) defined as EF < 55% (n = 22, EF 48 ± 8 %). Global peak systolic strain (PSS) and peak relaxation rate (PRR) were used as indices of global LV contraction and relaxation, respectively (Vivid 7 and EchoPAC, GE Electronic). Results: PSS was maintained in LVH-NEF but reduced in LVH-SD from all three perpendicular axes. PRR (L) was impaired in LVH-NEF and was further decreased in LVH-SD (0.95 ± 0.33* and 0.58 ± 0.24* † 1/s, *p <0.05 vs. Control and † p <0.05 vs. LVH-NEF, respectively)compared to Control (1.14 ± 0.30 1/s). PRR (C) was maintained in LVH-NEF but reduced in LVH-SD (1.24 ± 0.50 and 0.73 ± 0.36 1/s*, p <0.05 vs. Control) compared to Control (1.30 ± 0.48 1/s). PRR (R) was impaired in both LVH-NEF and LVH-SD in the same degrees (-1.53 ± 0.60* and -1.27 ± 0.64* 1/s, p <0.05 vs. Control: -2.08 ± 0.84 1/s). Conclusion: Speckle tracking strain rate imaging quantified and stratified the severity of LV relaxation abnormality in patients with LVH ranged from normal to reduced LV systolic function independently from all three perpendicular ventricular axes.


2020 ◽  
Vol 21 (Supplement_1) ◽  
Author(s):  
A Stoylen ◽  
H E Molmen ◽  
H E Dalen

Abstract Background As the Left ventricular (LV) apex is stationary, and maximum velocities are in the mitral plane, peak systolic annular velocity (S") normalized for wall length (WL) is a measure of peak systolic strain rate (Fig. 1), as is strain rate measured by tissue Doppler. As the normalization presumably corrects for the size of the heart (and thus body), this should reduce biological variability, but this assumption has previously been shown to be faulty for annular displacement (MAPSE) vs global longitudinal strain. Methods The HUNT study examined 1266 subjects without evidence of heart disease, from a mixed urban / rural population of North Tøndelag county in Norway. Annular systolic velocity was measured by pulsed-wave Tissue Doppler in the septal, lateral, anterior and inferior points, and averaged. Wall lengths was measured in a straight line in the same points, and S" was normalized (S"/WL) and averaged. Segmental systolic strain rate was also measured by combined speckle tracking / Tissue Doppler and averaged to a global value (GLSR). Results are given in table 1. All three measures declined with increasing age (R was -0.23, -0.29 and -0.29, respectively, all p &lt; 0.001) The over-all relative standard deviations were similar for S", normalized S" and global strain rate. Both S"/WL and GLSR correlated negatively with BSA, R was - 0.22 and 0.17, respectively (p &lt; 0.001), while S" showed a modest positive correlation; R = 0.13, (p &lt; 0.001) Conclusion Normalizing peak systolic mitral annular velocity for length do not reduce biological variability, and introduces a systematic error in the body size relation, due to the one-dimensional nature of the normalisation, and the three-dimensional nature of LV deformation. This is in accordance with what has previously been shown for global longitudinal strain vs. annular plane displacement. Table 1 Age (years) S" (cm/s) S"/WL (/s) GLSR (/s) &lt; 40 9.1 (1.2) 0.93 (0.13) 1.08 (0.12) 40 - 60 8.3 (1.3) 0.86 (0.13) 1.03 (0.12) &gt; 60 7.7 (1.3) 0.81 (0.14) 0.98 (0.14) All 8.4 (1.4) 0.87 (0.14) 1.03 (0.13) Relative SD (%) 16.7 16.1 12.6 Mitral annular velocity (S"), normalised velocity (S"/WL) and Global strain by age groups. (Standard deviations in parentheses) Abstract 1025 Figure. Fig. 1


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