Intravenous infusion of 40% urotropine solution in gynecology

2021 ◽  
Vol 20 (6) ◽  
pp. 645-645
Author(s):  
B. Tarlo
Keyword(s):  
Intravenous Infusion ◽  
Large Material ◽  
Severe Spasm

Having tested these infusions on a very large material, Quack (Zentr. F. Gyn., 1923, No. 50-51) found that 1) prophylactic infusions of 5 cubic meters. sant. 40% urotropine solution after surgery leads to severe spasm of the bladder, which is why it should not be used

A user-centered evaluation of three intravenous infusion pumps

2004 ◽  
Author(s):  
Roger Gagnon ◽  
Jason Laberge ◽  
Allison Lamsdale ◽  
Jonathan Histon ◽  
Carl Hudson ◽  
...  
Keyword(s):  
Intravenous Infusion ◽  
Infusion Pumps ◽  
User Centered

Technology characteristics predicting end user acceptance of smart intravenous infusion pumps

2005 ◽  
Author(s):  
Tosha B. Wetterneck ◽  
Pascale Carayon ◽  
Folasade Sobande ◽  
Ann Schoofs Hundt
Keyword(s):  
Intravenous Infusion ◽  
User Acceptance ◽  
End User ◽  
Infusion Pumps

Efficacy of octreotide (Octrade) for prevention of pancreatitis after endoscopic retrograde cholangiopancreatography

2020 ◽  
Vol 1 (30) ◽  
pp. 30-36
Author(s):  
E. A. Krylova ◽  
D. V. Aleinik
Keyword(s):  
Endoscopic Retrograde Cholangiopancreatography ◽  
Intravenous Infusion ◽  
Pancreatic Enzyme ◽  
Enzyme Secretion ◽  
Continuous Intravenous Infusion ◽  
Endoscopic Retrograde ◽  
Pancreatic Enzyme Secretion

The article presents the results of a study of the effectiveness of the use of an inhibitor of pancreatic enzyme secretion of octreotide (Octrade) for the prevention of pancreatitis after endoscopic retrograde cholangiopancreatography (ERCP). It was shown that the administration of Octrade at a dose of 0.3 mg in 500 ml of 0.9 % NaCl by continuous intravenous infusion for 7 hours and then 0.1 mg of Octrade subcutaneously at 6 and 12 hours after the end of intravenous infusion significantly reduced the frequency of pancreatitis (4.0 % and 22.2 %; p < 0.05) and hyperamylasemia (8.0 % and 25.9 %; p < 0.05) after ERCP. It is concluded that Octrade is effective in preventing the development of pancreatitis and hyperamilasemia after ERCP.

Blood Ethanol Levels Following Rapid Intravenous Infusion

1971 ◽  
Vol 32 (3) ◽  
pp. 741-747 ◽  
Author(s):  
John W. Dundee ◽  
M. Isaac ◽  
J. Taggart
Keyword(s):  
Intravenous Infusion

IMITATION OF THE ADRENO-CORTICAL RESPONSE TO SURGERY BY INTRAVENOUS INFUSION OF SYNTHETIC HUMAN ACTH

1973 ◽  
Vol 72 (1) ◽  
pp. 75-80
Author(s):  
Henrik Kehlet ◽  
Christian Binder ◽  
Christen Engbæk
Keyword(s):  
Intravenous Infusion ◽  
Time Course ◽  
Cortical Response ◽  
Secretion Rate ◽  
Major Surgery ◽  
Acth Secretion ◽  
Dose Rates

ABSTRACT The concentration of plasma corticosteroids was followed during major surgery and during the infusion of synthetic human ACTH at dose rates varying from 2400 ng to 15 000 ng per hour. The results showed that the time course of plasma corticosteroids during major surgery was intermediate between that obtained during the infusion of 7500 and 15 000 ng synthetic human ACTH per hour. This gives an estimated ACTH secretion rate during major surgery of between 7500 ng and 15 000 ng per hour.

ADRENERGIC CONTROL MECHANISM FOR ACTH SECRETION IN MAN

1973 ◽  
Vol 74 (2) ◽  
pp. 263-270 ◽  
Author(s):  
Yoshikatsu Nakai ◽  
Hiroo Imura ◽  
Teruya Yoshimi ◽  
Shigeru Matsukura
Keyword(s):  
Intravenous Infusion ◽  
Human Subjects ◽  
Blocking Agent ◽  
Inhibitory Effect ◽  
Plasma Acth ◽  
Acth Secretion ◽  
Glucose Levels ◽  
Alpha Receptors ◽  
Normal Human ◽  
Adrenergic Blocking

ABSTRACT In order to determine if an adrenergic mechanism is involved in the secretion of corticotrophin (ACTH), the effect of adrenergic-blocking or -stimulating agent on plasma ACTH, cortisol and glucose levels was studied in normal human subjects. The intravenous infusion of methoxamine, an alpha adrenergic-stimulating agent, caused a rise in plasma ACTH and cortisol. This increase in plasma ACTH and cortisol was significantly inhibited by the simultaneous administration of phentolamine, an alpha adrenergic-blocking agent, in combination with methoxamine. The intravenous infusion of propranolol, a beta adrenergic-blocking agent, caused no significant change in plasma ACTH and cortisol, although it enhanced the plasma ACTH response to insulin-induced hypoglycaemia. On the other hand, alpha adrenergicblockade by intravenous infusion of phentolamine significantly suppressed the plasma ACTH response to insulin-induced hypoglycaemia. These studies suggest a stimulatory effect of alpha receptors and a possible inhibitory effect of beta receptors on ACTH secretion in man.

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