GSM and DCS Wireless Communication Signals: Combined Chronic Toxicity/Carcinogenicity Study in the Wistar Rat

2007 ◽  
Vol 168 (4) ◽  
pp. 480-492 ◽  
Author(s):  
Paul Smith ◽  
Niels Kuster ◽  
Sven Ebert ◽  
Hans-Jörg Chevalier
Keyword(s):  
Wireless Communication ◽  
Chronic Toxicity ◽  
Wistar Rat ◽  
Communication Signals ◽  
Carcinogenicity Study

Carcinogenicity study of GSM and DCS wireless communication signals in B6C3F1 mice

2007 ◽  
Vol 28 (3) ◽  
pp. 173-187 ◽  
Author(s):  
Thomas Tillmann ◽  
Heinrich Ernst ◽  
Sven Ebert ◽  
Niels Kuster ◽  
Wolfgang Behnke ◽  
...  
Keyword(s):  
Wireless Communication ◽  
Communication Signals ◽  
Carcinogenicity Study ◽  
B6c3f1 Mice

The Effects of 884 MHz GSM Wireless Communication Signals on Self-reported Symptom and Sleep (EEG)- An Experimental Provocation Study

PIERS Online ◽  
2007 ◽  
Vol 3 (7) ◽  
pp. 1148-1150 ◽  
Author(s):  
Bengt B Arnetz ◽  
Torbjorn Akerstedt ◽  
Lena Hillert ◽  
Arne Lowden ◽  
N. Kuster ◽  
...  
Keyword(s):  
Wireless Communication ◽  
Sleep Eeg ◽  
Communication Signals

scholarly journals Reevaluation of Hepatocellular Neoplasms in CD-1 Mice from a 2-year Oral Carcinogenicity Study with Permethrin

2018 ◽  
Vol 47 (1) ◽  
pp. 11-17 ◽  
Author(s):  
Erin M. Quist ◽  
Gary A. Boorman ◽  
John M. Cullen ◽  
Robert R. Maronpot ◽  
Amera K. Remick ◽  
...  
Keyword(s):  
Chronic Toxicity ◽  
Expert Panel ◽  
Biological Significance ◽  
Male Mice ◽  
Male And Female ◽  
Female Mice ◽  
Carcinogenicity Study ◽  
Dose Dependent Increase ◽  
Dose Dependent ◽  
The Continuum

A 24-month oral carcinogenicity study of permethrin was conducted by feeding male and female CD-1 mice diets containing concentrations of 0, 20, 500, and 2,000 ppm of permethrin (males) or 0, 20, 2,500, and 5,000 ppm of permethrin (females). After approximately two years on study, surviving mice were sacrificed for the evaluation of chronic toxicity and/or carcinogenicity. An expert panel of pathologists was convened as a Pathology Working Group (PWG) to review coded liver histology sections from male and female mice and to classify all liver neoplasms according to current nomenclature and diagnostic criteria guidelines. The PWG results indicate that permethrin induced a significant dose-dependent increase in the incidence of hepatocellular neoplasms in treated female mice ( p < .01) as well as a nonstatistically significant increase in the incidence of hepatocellular tumors in treated male mice. Given the continuum of the diagnoses of adenoma and carcinoma, and the difficulty in distinguishing some of the lesions, it is appropriate to consider only the combined incidences of hepatocellular tumors (adenoma and/or carcinoma) for biological significance and risk assessment.

Two-year oral chronic toxicity and carcinogenicity study in rats of diets fumigated with methyl bromide

1990 ◽  
Vol 28 (2) ◽  
pp. 109-119 ◽  
Author(s):  
K. Mitsumori ◽  
K. Maita ◽  
T. Kosaka ◽  
T. Miyaoka ◽  
Y. Shirasu
Keyword(s):  
Methyl Bromide ◽  
Chronic Toxicity ◽  
Carcinogenicity Study

scholarly journals Parathyroid Gland Carcinoma in a Wistar Rat

2003 ◽  
Vol 40 (2) ◽  
pp. 203-206 ◽  
Author(s):  
V. Pace ◽  
S. Scarsella ◽  
E. Perentes
Keyword(s):  
Proliferating Cell Nuclear Antigen ◽  
Proliferative Activity ◽  
Wistar Rat ◽  
Anaplastic Carcinoma ◽  
Nuclear Antigen ◽  
Ki 67 ◽  
Carcinogenicity Study ◽  
Thyroid Transcription Factor ◽  
Gland Carcinoma ◽  
Proliferating Cell

An anaplastic carcinoma was found in one of the two parathyroids of a 2-year-old male Wistar rat, which was sacrificed at the end of a carcinogenicity study. Morphologically, it was characterized by the presence of nodular areas of pleomorphic and dense cells with numerous atypical mitoses and large regions of smaller and dark monomorphic cells devoid of mitoses and forming small cystic spaces. Local invasion of the capsule and pronounced compression of the parenchyma of the thyroid gland were observed. Immunohistochemically, the tumor was markedly positive for the parathyroid hormone and negative for the thyroid transcription factor. The proliferative activity was assessed by immunostaining the endogenous cell proliferation associated-antigen Ki-67, and the proliferating cell nuclear antigen. The diagnosis of carcinoma of the parathyroid was made on the basis of microscopic and immunohistochemical findings.

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