scholarly journals Crassulacean Acid Metabolism May Alleviate Production of Reactive Oxygen Species in a Facultative CAM Plant, the Common Ice PlantMesembryanthemum crystallinumL.

2010 ◽  
Vol 13 (3) ◽  
pp. 256-260 ◽  
Author(s):  
Haruki Sunagawa ◽  
John Cushman ◽  
Sakae Agarie
Keyword(s):  
Reactive Oxygen Species ◽  
Crassulacean Acid Metabolism ◽  
Acid Metabolism ◽  
Reactive Oxygen ◽  
Oxygen Species ◽  
The Common ◽  
Cam Plant

Role of mitochondrial complexes I and II, reactive oxygen species and arachidonic acid metabolism in deoxycholate-induced apoptosis

2002 ◽  
Vol 177 (2) ◽  
pp. 129-144 ◽  
Author(s):  
Delon Washo-Stultz ◽  
Cara L Crowley-Weber ◽  
Katerina Dvorakova ◽  
Carol Bernstein ◽  
Harris Bernstein ◽  
...  
Keyword(s):  
Reactive Oxygen Species ◽  
Arachidonic Acid ◽  
Acid Metabolism ◽  
Reactive Oxygen ◽  
Arachidonic Acid Metabolism ◽  
Oxygen Species ◽  
Mitochondrial Complexes ◽  
Induced Apoptosis

scholarly journals Recent Studies on the Therapeutic and Prophylactic Effect of Some Agents on Experimental Induced Parkinson Disease Models Review

2021 ◽  
pp. 1-18
Author(s):  
I. M. Hassan ◽  
A. M. Abdullahi
Keyword(s):  
Reactive Oxygen Species ◽  
Review Paper ◽  
Neuronal Cells ◽  
Reactive Oxygen ◽  
The Body ◽  
Prophylactic Effect ◽  
Oxygen Species ◽  
Unique Role ◽  
The Central Nervous System ◽  
The Common

Neurodegeneration include disease of the central nervous system (CNS) such as Parkinson’s disease (PD). The sign of this disease include destruction of dopaminergic neuronal cells. Main causes of this disease are unknown, but several studies reported polygenic causes. These include free radicals, malfunction of mitochondria, protein oxidation and DNA denaturation. Several reports implicated the involvement of oxygen species and stresses cause by over accumulation of reactive oxygen species in the contribution to the cascade that resulted in diminishing of dopamine cells destruction in this disease. Generally, natural defensive substances such as bio-cartelized (proteineous and non proteineous) from natural or synthetic sources played a unique role in protecting the susceptible neuronal cells from the adverse effects of the reactive oxygen species. In most cases antioxidant enzyme production by the body system diminished with advance age. Hence, treatment with antioxidant potential agent or administration of antioxidant substance with the common drugs used in the treatment of PD may yield positive result in protecting neurons from degenerative changes caused by reactive oxygen species. Recent studies on the therapeutic and prophylactic effect of some agents on experimental induced PD summarized in this review paper.

Effects of reactive oxygen species on arachidonic acid metabolism in rabbit platelets

1993 ◽  
Vol 15 (1) ◽  
pp. 101-104 ◽  
Author(s):  
Toshiki Sumiya ◽  
Yohko Fujimoto ◽  
Hiroko Nishida ◽  
Yuriyo Morikawa ◽  
Satotu Sakuma ◽  
...  
Keyword(s):  
Reactive Oxygen Species ◽  
Arachidonic Acid ◽  
Acid Metabolism ◽  
Reactive Oxygen ◽  
Arachidonic Acid Metabolism ◽  
Oxygen Species ◽  
Rabbit Platelets

Mitochondria as a source of reactive oxygen species

2009 ◽  
pp. c3 ◽  
Author(s):  
Helena M. Cochemé ◽  
Michael P. Murphy
Keyword(s):  
Reactive Oxygen Species ◽  
Reactive Oxygen ◽  
Oxygen Species

Clinical aspects of reactive oxygen and nitrogen species

2004 ◽  
Vol 71 ◽  
pp. 121-133 ◽  
Author(s):  
Ascan Warnholtz ◽  
Maria Wendt ◽  
Michael August ◽  
Thomas Münzel
Keyword(s):  
Oxidative Stress ◽  
Reactive Oxygen Species ◽  
Risk Factor ◽  
Endothelial Dysfunction ◽  
Vascular Tissue ◽  
Rapid Development ◽  
Reactive Oxygen ◽  
Clinical Aspects ◽  
No Production ◽  
Oxygen Species

Endothelial dysfunction in the setting of cardiovascular risk factors, such as hypercholesterolaemia, hypertension, diabetes mellitus and chronic smoking, as well as in the setting of heart failure, has been shown to be at least partly dependent on the production of reactive oxygen species in endothelial and/or smooth muscle cells and the adventitia, and the subsequent decrease in vascular bioavailability of NO. Superoxide-producing enzymes involved in increased oxidative stress within vascular tissue include NAD(P)H-oxidase, xanthine oxidase and endothelial nitric oxide synthase in an uncoupled state. Recent studies indicate that endothelial dysfunction of peripheral and coronary resistance and conductance vessels represents a strong and independent risk factor for future cardiovascular events. Ways to reduce endothelial dysfunction include risk-factor modification and treatment with substances that have been shown to reduce oxidative stress and, simultaneously, to stimulate endothelial NO production, such as inhibitors of angiotensin-converting enzyme or the statins. In contrast, in conditions where increased production of reactive oxygen species, such as superoxide, in vascular tissue is established, treatment with NO, e.g. via administration of nitroglycerin, results in a rapid development of endothelial dysfunction, which may worsen the prognosis in patients with established coronary artery disease.

Fas ligand expression in rat kupffer cells in response to lipopolysaccharide is mediated by reactive oxygen species

2001 ◽  
Vol 120 (5) ◽  
pp. A361-A361
Author(s):  
K UCHIKURA ◽  
T WADA ◽  
Z SUN ◽  
S HOSHINO ◽  
G BULKLEY ◽  
...  
Keyword(s):  
Reactive Oxygen Species ◽  
Kupffer Cells ◽  
Fas Ligand ◽  
Reactive Oxygen ◽  
Oxygen Species ◽  
Ligand Expression
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