Exemplar Abstract for Helicobacter pylori (Fox et al. 1988) Goodwin et al. 1989, Campylobacter pyloridis (sic) (Fox et al. 1988) Marshall et al. 1985 emend. Fox et al. 1989, Campylobacter pylori corrig. (Fox et al. 1988) Marshall et al. 1985 emend. Fox et al. 1989 and Campylobacter pylori pylori Fox et al. 1988.

2003 ◽  
Author(s):  
Charles Thomas Parker ◽  
Dorothea Taylor ◽  
George M Garrity
Keyword(s):  
Helicobacter Pylori ◽  
Campylobacter Pylori ◽  
Campylobacter Pyloridis

Helicobacter pylori (Campylobacter pylori) and duodenal ulcer

1990 ◽  
Vol 153 (2) ◽  
pp. 66-67 ◽  
Author(s):  
C Stewart Goodwin ◽  
Andrew Gordon ◽  
Valerie Burke
Keyword(s):  
Duodenal Ulcer ◽  
Helicobacter Pylori ◽  
Campylobacter Pylori

scholarly journals Theory of Additional Corpus Biopsy for Epidemiology and Diagnosis of Helicobacter Pylori

2017 ◽  
Vol 1 (1) ◽  
pp. 01-04
Author(s):  
Vijay Kumar Bontha ◽  
Swathi Goli ◽  
Prasad Garrepally
Keyword(s):  
Helicobacter Pylori ◽  
Acute Infection ◽  
Colorectal Polyps ◽  
Campylobacter Pylori ◽  
Causative Role ◽  
Type B ◽  
Ulcer Disease ◽  
Acute Gastritis ◽  
H Pylori ◽  
Type B Gastritis

Background: There is still debate on the best sites for biopsy- based tests of Helicobacter pylori infection in patients with gastritis. This study was designed to determine if it is important to add corpus biopsies to the routine antral ones for identification of H. pylori, especially in case of gastric atrophy and/or intestinal metaplasia. Methods: A causative role is now accepted for Helicobacter (formerly Campylobacter) pylori in type B gastritis, and evidence is accumulating that H. pylori infection plays a major contributory role in peptic ulcer disease. Preliminary studies have reported that the prevalence of H pylori infection increases with age, but detailed information on the prevalence of the bacteria in any defined population and on the factors that may influence the pattern of distribution remains scanty. Results: Up to 85% of people infected with H. pylori never experience symptoms or complications. Acute infection may appear as an acute gastritis with abdominal pain (stomach ache) or nausea. Where this develops into chronic gastritis, the symptoms, if present, are often those of nonulcer dyspepsia: stomach pains, nausea, bloating, belching, and sometimes vomiting or black stool. Conclusion: H. pylori has been associated with colorectal polyps and colorectal cancer. It may also be associated with eye disease.

Some Ultrastructural Aspects of Chronic Gastritis

1990 ◽  
Vol 48 (3) ◽  
pp. 198-199
Author(s):  
Hieronim Bartel ◽  
Stanislaw Orkisz ◽  
Jan Chojnacki
Keyword(s):  
Chronic Gastritis ◽  
Gastric Epithelium ◽  
Ultrastructural Changes ◽  
Campylobacter Pylori ◽  
Surface Epithelium ◽  
Biopsy Material ◽  
Campylobacter Pyloridis ◽  
Semithin Sections ◽  
Ultrathin Sections ◽  
Epithelium Cells

Despite numerous observations and publications on the aethiology and pathogenesis of chronic gastritis many questions remain unanswered (1,2,3,4,6). Views on the aethiolcgy of chronic gastritis changed when in 1982 Campylobacter pyloridis (later named Campylobacter pylori CP) was first isolated in Western Australia from the stomach of patients with gastritis lesions and peptic ulceration (5).The aim of this study, carried out with cooperation with gastroenterologist, has been to investigate the ultrastructural changes in gastric surface epithelium and lamina propria in selected patients endoscopicaUy diagnosed gastritis.Observations have been carried out on 20 patients (10 men and 10 women). Biopsy material was obtained during endoscopy. In cases when the presence of bacteria has been confirmed further E M investigations have been introduced. Semithin sections for LM observations and ultrathin sections for EM were prepared according to routine EM technique.CP was found both on the airface of gastric epithelium cells, and what has not been reported so far, inside the lumen of the fundic glands (Fig. 1).

scholarly journals Helicobacter pylori Infection in Persons with Intellectual Disability in Residential Care in Israel

2001 ◽  
Vol 1 ◽  
pp. 264-268 ◽  
Author(s):  
Joav Merrick ◽  
Shoshana Aspler ◽  
Inessa Dubman
Keyword(s):  
Helicobacter Pylori ◽  
Mental Retardation ◽  
Intellectual Disability ◽  
Drug Treatment ◽  
Residential Care ◽  
Treatment Success ◽  
Campylobacter Pylori ◽  
Persistent Symptoms ◽  
Dyspeptic Symptoms ◽  
H Pylori

Helicobacter pylori (formerly Campylobacter pylori) was identified in 1982 by researchers from Australia as a pathogenic factor in peptic ulcer disease. Due to the few studies on H. pylori infection conducted in the population of persons with intellectual disability it was decided to conduct a clinical study in Israel. The purpose of the study was to determine the occurrence of H. pylori infection in persons who presented with severe dyspeptic symptoms and to monitor clinically the effect of treatment. The Division for Mental Retardation in Israel provides service to 6,022 persons in 53 residential care centers and 1 in central Israel was selected for this pilot study. The study has been performed since 1999 and each patient who came to the medical clinic of the institution with severe dyspeptic symptoms was examined clinically and a blood specimen drawn for IgG antibodies to H. pylori (ELISA, Pharmatop Millenia). In case of positive serology, triple drug treatment (amoxycillin, metronidazole, and pantoprazole or omepra-zole) was initiated for 1 week. Since 1999 a total of 43 persons (total population in care was 224) had severe dyspeptic symptoms and 42 persons (98%, 26 males, 16 females, mean age 45 years, mean institutionalization 20 years) had Helicobacter infection. All patients were treated for 1 week, but six patients received an extra month of omeprazole due to persistent symptoms. At follow-up, clinically all patients had improvement and only seven still had minor complaints (83% treatment success). Persons with developmental disability, intellectual disability, or mental retardation in residential care presenting with severe dyspeptic symptoms had a high incidence of H. pylori infection. Therefore, we recommend serology or urea breath investigations in this population presenting with dyspeptic symptoms and triple drug treatment for 1 week in case of positive findings.

scholarly journals Helicobacter (Campylobacter) pylori and Acid Peptic Diseases

1990 ◽  
Vol 4 (6) ◽  
pp. 237-242 ◽  
Author(s):  
Sigmund Kradjen ◽  
Philip Sherman
Keyword(s):  
Gastric Cancer ◽  
Helicobacter Pylori ◽  
Peptic Ulcer ◽  
Campylobacter Pylori ◽  
Gram Negative Bacteria ◽  
Breath Tests ◽  
Gram Negative ◽  
Ulcer Disease ◽  
Biopsy Specimens ◽  
Diagnostic Approaches

Helicobacter pylori is a spiral-shaped Gram-negative bacteria implicated as a cause of histological gastritis, contributing to peptic ulcer disease and perhaps playing a role in gastric cancer in humans. The organism is found worldwide; the prevalence of infection increases with age; and colonization probably persists for life. Diagnostic approaches chat have been used include tissue stains, culture of stomach biopsy specimens, labelled-urea breath tests and serology. It is too early to advocate treatment for all infected individuals; the benefits and risks have yet to be carefully studied and assessed.

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