scholarly journals Orbito cerebral complications of poly sinusitis

2017 ◽  
Vol 1 (2) ◽  
Author(s):  
Soulé Alamou ◽  
Lisette YehouessI ◽  
Chakiratou Abouki ◽  
Olivier Biaou ◽  
Hermann Ngoufo ◽  
...  
Keyword(s):  
Cerebral Complications

Determination of the content of neurospecific enolase in blood for diagnosis of early postoperative cerebral complications after removal of meningioma

2019 ◽  
Vol 17 (7) ◽  
pp. 149-153
Author(s):  
A. S. Kurakina ◽  
◽  
N. A. Schelchkova ◽  
I. V. Mukhina ◽  
V. N. Grigoreva ◽  
...  
Keyword(s):  
Cerebral Complications

Intra-Abdominal Hypertension: Pulmonary and Cerebral Complications

2008 ◽  
Vol 40 (4) ◽  
pp. 1190-1192 ◽  
Author(s):  
V. Vegar-Brozovic ◽  
J. Brezak ◽  
I. Brozovic
Keyword(s):  
Cerebral Complications ◽  
Abdominal Hypertension

Preventing Cerebral Complications of Cardiac Surgery

1991 ◽  
pp. 219-227
Author(s):  
Marc I. Chimowitz ◽  
Anthony J. Furlan
Keyword(s):  
Cardiac Surgery ◽  
Cerebral Complications

Serum S100B Protein Could Help to Detect Cerebral Complications Associated with Extracorporeal Membrane Oxygenation (ECMO)

2013 ◽  
Vol 20 (3) ◽  
pp. 367-374 ◽  
Author(s):  
Duc Nam Nguyen ◽  
Luc Huyghens ◽  
Francis Wellens ◽  
Johan Schiettecatte ◽  
Johan Smitz ◽  
...  
Keyword(s):  
Extracorporeal Membrane Oxygenation ◽  
S100b Protein ◽  
Serum S100b ◽  
Membrane Oxygenation ◽  
Cerebral Complications

scholarly journals Subclinical Cerebral Complications After Coronary Artery Bypass Grafting

1998 ◽  
Vol 55 (5) ◽  
pp. 618 ◽  
Author(s):  
Ritva Vanninen ◽  
Marja Äikiä ◽  
Mervi Könönen ◽  
Kaarina Partanen ◽  
Harri Tulla ◽  
...  
Keyword(s):  
Coronary Artery ◽  
Coronary Artery Bypass Grafting ◽  
Coronary Artery Bypass ◽  
Artery Bypass ◽  
Bypass Grafting ◽  
Artery Bypass Grafting ◽  
Cerebral Complications

scholarly journals Hypertension and Cerebral Microangiopathy (Cerebral Small Vessel Disease): Genetic and Epigenetic Aspects of Their Relationship

Acta Naturae ◽  
2018 ◽  
Vol 10 (2) ◽  
pp. 4-15 ◽  
Author(s):  
L. A. Dobrynina ◽  
M. R. Zabitova ◽  
L. A. Kalashnikova ◽  
E. V. Gnedovskaya ◽  
M. A. Piradov
Keyword(s):  
Antihypertensive Therapy ◽  
Small Vessel Disease ◽  
Association Studies ◽  
Cerebral Small Vessel Disease ◽  
Cerebral Injury ◽  
Small Vessel ◽  
Genome Wide Association Studies ◽  
Vessel Disease ◽  
Cerebral Complications ◽  
Cerebral Microangiopathy

Hypertension (HT) and its cerebral complications are extremely vexing medical and social problems. Despite the obvious association between hypertension and the clinical and neuroimaging features of cerebral microangiopathy (CMA) (also known as cerebral small vessel disease), the causal links between them remain ambiguous. Besides, antihypertensive therapy as the only way to manage these patients does not always prevent brain damage. Knowledge about the key factors and mechanisms involved in HT and CMA development is important for predicting the risk of cerebral complications and developing new approaches to their prevention and treatment. At present, genome-wide association studies and other approaches are used to investigate the common hereditary mechanisms of HT and CMA development, which will explain a large number of CMA cases not associated with hypertension, lack of a correlation between HT severity and the degree of cerebral injury, and failure of antihypertensive therapy to prevent CMA progression. Epigenetic markers likely play a modulating role in the development of these diseases.

Abstract 18110: Impact of Stroke Severity on Clinical Outcomes in Patients who had Infective Endocarditis With Cerebral Complications

Circulation ◽  
2014 ◽  
Vol 130 (suppl_2) ◽  
Author(s):  
Ryosuke Murai ◽  
Shunsuke Funakoshi ◽  
Shuichiro Kaji ◽  
Yasuhiro Sasaki ◽  
Kitae Kim ◽  
...  
Keyword(s):  
Infective Endocarditis ◽  
Clinical Outcomes ◽  
Neurological Deficit ◽  
Treatment Strategy ◽  
Conventional Treatment ◽  
Hospital Death ◽  
Stroke Severity ◽  
Severe Stroke ◽  
Cerebral Complications ◽  
Stroke Group

Background: The treatment strategy in active infective endocarditis (IE) with cerebral complications still remains unclear. We sought to analyze the association of the neurological deficit level with the clinical outcomes in IE patients with stroke. Methods and Results: Clinical data were retrospectively reviewed in 141 consecutive patients with active left-sided IE with cerebral complications. To evaluate the severity of stroke, the National Institute of Health Stroke Scale (NIHSS) was assessed in all patients on admission. There were 116 (82%) patients with cerebral infarction including 9 hemorrhagic infarctions and 57 (40%) patients with cerebral hemorrhage. We divided the patients according to NIHSS; severe stroke group (NIHSS>16: n=19) and non-severe stroke group (NIHSS≤16: n=122). Early surgery in the active phase (within 2 weeks after the initial diagnosis) was performed in 64 patients (6 severe stroke group and 58 non-severe stroke group), and the conventional treatment strategy was applied in 77 patients (13 severe stroke group and 64 non-severe stroke group). In the conventional treatment group, 37 patients (36 severe stroke group and 1 non-severe stroke group) underwent late surgical intervention. A mean follow-up period was 4.5 years. In-hospital death was significantly lower in non-severe stroke group (12% versus 53%, p<0.001). In addition, the freedom rate from IE-related death was significantly higher in patients with non-severe stroke than those with severe stroke (84±4 % versus 37±13 % at 5-year, p<0.001). Multivariate Cox proportional hazard analysis showed that NIHSS (HR=1.074; 95% CI 1.042-1.107: p<0.001), logistic EuroSCORE (HR=1.026; 95% CI 1.010-1.042: p=0.002), conventional treatment (HR=3.478; 95% CI 1.477-8.193: p=0.004), and aortic valve involvement (HR=3.091; 95% CI 1.460-6.546: p=0.003) were significantly associated with IE-related mortality (p<0.001). Conclusions: Severity of stroke was strongly associated with clinical outcomes in IE patients with cerebral complications. Therapeutic strategy for IE patients with stroke might have to be customized according to severity of neurological deficit.

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