scholarly journals Epiphyseal Closure of Femur, Tibia and Fibula of the Paca (Cuniculus Paca, Linnaeus, 1766)

Author(s):  
Lippi ICC ◽  
Oliveira RGS ◽  
Smargiassi NF ◽  
Machado MRF ◽  
Sasahara THC ◽  
...  
Keyword(s):  
1972 ◽  
Vol 34 (1) ◽  
pp. 42-45 ◽  
Author(s):  
D. L. Grant ◽  
H. J. Tuma ◽  
R. C. Covington ◽  
A. D. Dayton

1986 ◽  
Vol 15 (6) ◽  
pp. 1259-1262 ◽  
Author(s):  
Julie Prendiville ◽  
E.A. Bingham ◽  
D. Burrows
Keyword(s):  

2001 ◽  
Vol 26 (1) ◽  
pp. 13-16 ◽  
Author(s):  
S. HOUSHIAN ◽  
T. IPSEN

Twelve metacarpals and two phalanges in 14 patients (nine males and five females) were lengthened between August 1992 and March 1999 by the callus distraction technique using a small external fixator (Orthofix®). The indications were traumatic amputation (10 cases), aplasia (three cases) and hypoplasia owing to premature epiphyseal closure of the metacarpal (one case). All patients were reviewed with a median follow-up time of 39 (range, 9–88) months. The median age of the patients was 15 (range, 4–39) years. The median lengthening was 21 (range, 13–34) mm which was a median of 82% (range, 38–162) of the original bone length. The median times for callus distraction and consolidation were 4.5 and 7.5 weeks respectively. All except one patient tolerated the procedure well and were satisfied with the results. Functional improvement was seen in 13 out of 14 patients, but non-union was observed in one patient.


1997 ◽  
Vol 22 (8) ◽  
pp. 567-568
Author(s):  
C. VAN DE WIELE ◽  
K. OSMANAGAOGLU ◽  
K. AUDENAERT ◽  
R. A. DIERCKX

Foot & Ankle ◽  
1983 ◽  
Vol 3 (5) ◽  
pp. 297-298 ◽  
Author(s):  
Elias D. Sedlin

A case of Freiberg's Infraction of the third metatarsal which demonstrates early epiphyseal closure in this condition is presented. The etiology of this phenomenon is unclear.


1991 ◽  
Vol 260 (3) ◽  
pp. E471-E476 ◽  
Author(s):  
V. Gilsanz ◽  
T. F. Roe ◽  
J. Antunes ◽  
M. Carlson ◽  
M. L. Duarte ◽  
...  

Reductions in peak bone mass at skeletal maturity may increase the risk for the subsequent development of osteoporosis. Although changes in calcium intake can modify the rate of decline in bone density in the mature skeleton, longitudinal assessments of the effect of dietary calcium supplementation during skeletal growth on peak bone mass have not been done in humans or experimental animals. Thus quantitative computed tomography (QCT) was used to monitor changes in vertebral bone density at 6-wk intervals during growth from 8 wk of age until skeletal maturity at 35 wk in male New Zealand White rabbits maintained on diets containing 0.15% (low Ca), 0.45% (normal Ca), or 1.35% (high Ca) calcium. Serum parathyroid hormone (PTH) and calcitriol levels increased, and renal calcium excretion decreased in low Ca compared with normal Ca; in contrast, serum calcitriol levels decreased and renal calcium excretion increased from control values in high Ca. Vertebral bone density by QCT did not differ during growth between high Ca and normal Ca, and peak values at epiphyseal closure also did not differ in these two groups. Vertebral bone density was lower, however, throughout the study in low Ca, and peak values at epiphyseal closure remained below those in either normal Ca or high Ca. Quantitative bone histology revealed decreases in cortical thickness in the third lumbar vertebra in low Ca, whereas trabecular bone area did not differ among groups; there was no histological evidence of osteomalacia in low Ca. Thus dietary calcium restriction during growth reduces peak bone mass at skeletal maturity, but raising dietary calcium intake above normal levels does not increase peak bone mass in this experimental model.


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