scholarly journals Salicylic acid and hydrogen peroxide accumulation in relation to hydrolyte leakage in barley plants challenged with Cochliobolus sativus

2018 ◽  
Vol 46 (4) ◽  
pp. 650-657
Author(s):  
A. Al-Daoude ◽  
E. Al-Shehadah ◽  
A. Shoaib ◽  
M. Jawhar ◽  
M.I.E. Arabi
Keyword(s):  
Hydrogen Peroxide ◽  
Salicylic Acid ◽  
Cochliobolus Sativus ◽  
Hydrogen Peroxide Accumulation

scholarly journals Salicylic acid-mediated hydrogen peroxide accumulation and protection against Cd toxicity in rice leaves

2009 ◽  
Vol 329 (1-2) ◽  
pp. 327-337 ◽  
Author(s):  
Yun-Yang Chao ◽  
Chao-Yeh Chen ◽  
Wen-Dar Huang ◽  
Ching Huei Kao
Keyword(s):  
Hydrogen Peroxide ◽  
Salicylic Acid ◽  
Cd Toxicity ◽  
Hydrogen Peroxide Accumulation ◽  
Rice Leaves

scholarly journals Feedback Control of the Arabidopsis Hypersensitive Response

2004 ◽  
Vol 17 (4) ◽  
pp. 357-365 ◽  
Author(s):  
Chu Zhang ◽  
Annie Tang Gutsche ◽  
Allan D. Shapiro
Keyword(s):  
Hydrogen Peroxide ◽  
Salicylic Acid ◽  
Pseudomonas Syringae ◽  
Hypersensitive Response ◽  
Ion Leakage ◽  
Wild Type ◽  
Regulatory Circuits ◽  
Acid Accumulation ◽  
Salicylic Acid Accumulation ◽  
Hydrogen Peroxide Accumulation

The plant hypersensitive response (HR) to avirulent bacterial pathogens results from programmed cell death of plant cells in the infected region. Ion leakage and changes in signaling components associated with HR progression were measured. These studies compared Arabidopsis mutants affecting feedback loops with wild-type plants, with timepoints taken hourly. In response to Pseudomonas syringae pv. tomato DC3000·avrB, npr1-2 mutant plants showed increased ion leakage relative to wild-type plants. Hydrogen peroxide accumulation was similar to that in wild type, but salicylic acid accumulation was reduced at some timepoints. With DC3000·avrRpt2, similar trends were seen. In response to DC3000·avrB, ndr1-1 mutant plants showed more ion leakage than wild-type or npr1-2 plants. Hydrogen peroxide accumulation was delayed by approximately 1 h and reached half the level seen with wild-type plants. Salicylic acid accumulation was similar to npr1-2 mutant plants. With DC3000·avrRpt2, ndr1-1 mutant plants showed no ion leakage, no hydrogen peroxide accumulation, and minimal salicylic acid accumulation. Results with a ndr1-1 and npr1-2 double mutant were similar to ndr1-1. A model consistent with these data is presented, in which one positive and two negative regulatory circuits control HR progression. Understanding this circuitry will facilitate HR manipulation for enhanced disease resistance.

scholarly journals Arabidopsis NATA1 acetylates putrescine and decreases defense-related hydrogen peroxide accumulation

2016 ◽  
pp. pp.00446.2016 ◽  
Author(s):  
Yann-Ru Lou ◽  
Melike Bor ◽  
Jian Yan ◽  
Aileen S Preuss ◽  
Georg Jander
Keyword(s):  
Hydrogen Peroxide ◽  
Hydrogen Peroxide Accumulation

scholarly journals Roles of intracellular hydrogen peroxide accumulation in abscisic acid signaling in Arabidopsis guard cells

2011 ◽  
Vol 168 (16) ◽  
pp. 1919-1926 ◽  
Author(s):  
Rayhanur Jannat ◽  
Misugi Uraji ◽  
Miho Morofuji ◽  
Mohammad Muzahidul Islam ◽  
Rachel E. Bloom ◽  
...  
Keyword(s):  
Hydrogen Peroxide ◽  
Abscisic Acid ◽  
Guard Cells ◽  
Abscisic Acid Signaling ◽  
Hydrogen Peroxide Accumulation ◽  
Intracellular Hydrogen Peroxide

Induction of tolerance to cryogenic protocols in Solanum tuberosum by salicylic acid is mediated by enzymatic antioxidant activity and hydrogen peroxide

2021 ◽  
pp. 1-10
Author(s):  
Diana Rocio Ruiz-Sáenz ◽  
Humberto Antonio López-Delgado ◽  
Diana Daniela Ayala Hernández ◽  
Carlos Trejo ◽  
Martha Elena Mora-Herrera ◽  
...  
Keyword(s):  
Hydrogen Peroxide ◽  
Antioxidant Activity ◽  
Salicylic Acid ◽  
Solanum Tuberosum ◽  
Enzymatic Antioxidant ◽  
Induction Of Tolerance

Evaluation of Oxidative Stress potential of some Nsaıds against Hydrogen Peroxide in experimental animal

2021 ◽  
pp. 6194-6200
Author(s):  
KM Diksha Singh ◽  
Vikram Singh ◽  
Adity Singh ◽  
Vipin Kesharwani
Keyword(s):  
Oxidative Stress ◽  
Hydrogen Peroxide ◽  
Salicylic Acid ◽  
Liver Steatosis ◽  
Chronic Kidney Failure ◽  
Acetyl Salicylic Acid ◽  
Control Group ◽  
Experimental Protocol ◽  
Examination Result ◽  
Experimental Protocols

Background: Oxidative stress is imbalance between aggressive and defensive system. Overproduction of oxidative stress contribute in pathogenesis of many diseasesincluding Parkinsonism, Alzheimer diseases, apoptosis, hepatic fibrosis ,chronic kidney failure and liver steatosis etc . There are several OTC drugs including NSAIDs that generate oxidative stress when administered. So there is a need to explore about these drugs. Therefore this study was designed to evaluate the oxidative stress potential of Acetaminophen, acetyl salicylic acid and Celecoxib NSAIDs. Objective: The present study is design to investigate the oxidative stress of NSAIDs of acetaminophen, aspirin and Celecoxib drug with reference to the hydrogen peroxide. Material and method: The Experimental protocol was designed for estimate the level of oxidative stress in NSAIDs treated animals against hydrogen peroxides. Animal of control group received only vehicle throughout experimental protocol. Rats of AAP group, ASA group ,CX group were exposed to acetaminophen (150mg/kg; orally) acetyl salicylic acid (300mg/kg ;orally) and Celecoxib (50mg/kg; orally) for forty two days . Rodent of HP group were challenged with Hydrogen peroxides (0.5%) with same schedule as above. At end of experimental protocols, all the animals were sacrificed and their organ were identified and collected for oxidative stress estimation and histological examination. Result: NSAIDs administration caused increase in oxidative stress measured in terms of SOD, CAT, MDA, GSH and GPx. HP administration produced maximum oxidative stress compare to all other groups. Oxidative parameter i.e. SOD, CAT, GSH and GPx were found to be decreased as compare to control rats. However MDA were found to be increased as compare to control rats. Additionally, CX produced less oxidative stress compare to other NDAIDs. Further, histological examinations support the biochemical results. Conclusion: From the above observations it can be concluded that NSAIDs have oxidative stress potential and generate oxidative stress and damage the organs when administrated chronically. Thus, these drugs should be used judiciously.

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