Some Approaches to Rapid and Pre-Symptom Diagnosis of Chemical Stress in Plants

2009 ◽  
pp. 333-333-13 ◽  
Author(s):  
RR Velagaleti ◽  
D Kramer ◽  
SS Marsh ◽  
NG Reichenbach ◽  
DE Fleischman
Keyword(s):  
Chemical Stress

Pathomorphological changes in the internal organs of rats after chronic effect of chemical stress factors

2018 ◽  
Vol 3 (5) ◽  
pp. 63-66
Author(s):  
S. A. Sherstyuk ◽  
◽  
S. A. Nakonechnaya ◽  
E. V. Nakonechnyi ◽  
Е. E. Koschiy ◽  
...  
Keyword(s):  
Stress Factors ◽  
Chemical Stress ◽  
Chronic Effect ◽  
Internal Organs

Furan Analysis of Oil Impregnated Paper Aged by Chemical Stress

2020 ◽  
Author(s):  
Dusit Suksawat ◽  
Kittiphot Atthaphotpong ◽  
Kriangsak Takboontam ◽  
Kanin Satirapattanakiat ◽  
Chiranuwat Raphephat ◽  
...  
Keyword(s):  
Chemical Stress

Chemical stress and GABAergic central system

1990 ◽  
Vol 21 (4) ◽  
pp. 517-520 ◽  
Author(s):  
G.B. Acosta ◽  
M.E. Otero Losada ◽  
M.C. Rubio
Keyword(s):  
Chemical Stress ◽  
Central System

scholarly journals Dihydroceramide Desaturase Functions as an Inducer and Rectifier of Apoptosis: Effect of Retinol Derivatives, Antioxidants and Phenolic Compounds

2021 ◽  
Author(s):  
Mariam Alsanafi ◽  
Ryan D. R. Brown ◽  
Jeongah Oh ◽  
David R. Adams ◽  
Federico Torta ◽  
...  
Keyword(s):  
Phenolic Compounds ◽  
De Novo ◽  
Dynamic Balance ◽  
Proteasomal Degradation ◽  
Parp Cleavage ◽  
Chemical Stress ◽  
De Novo Synthesis ◽  
Concomitant Increase ◽  
Native Form ◽  
Dihydroceramide Desaturase

AbstractDihydroceramide desaturase (Degs1) catalyses the introduction of a 4,5-trans double bond into dihydroceramide to form ceramide. We show here that Degs1 is polyubiquitinated in response to retinol derivatives, phenolic compounds or anti-oxidants in HEK293T cells. The functional predominance of native versus polyubiquitinated forms of Degs1 appears to govern cytotoxicity. Therefore, 4-HPR or celecoxib appear to stimulate the de novo ceramide pathway (with the exception of C24:0 ceramide), using native Degs1, and thereby promote PARP cleavage and LC3B-I/II processing (autophagy/apoptosis). The ubiquitin-proteasomal degradation of Degs1 is positively linked to cell survival via XBP-1s and results in a concomitant increase in dihydroceramides and a decrease in C24:0 ceramide levels. However, in the case of 4-HPR or celecoxib, the native form of Degs1 functionally predominates, such that the apoptotic programme is sustained. In contrast, 4-HPA or AM404 do not produce apoptotic ceramide, using native Degs1, but do promote a rectifier function to induce ubiquitin-proteasomal degradation of Degs1 and are not cytotoxic. Therefore, Degs1 appears to function both as an ‘inducer’ and ‘rectifier’ of apoptosis in response to chemical cellular stress, the dynamic balance for which is dependent on the nature of chemical stress, thereby determining cytotoxicity. The de novo synthesis of ceramide or the ubiquitin-proteasomal degradation of Degs1 in response to anti-oxidants, retinol derivatives and phenolic compounds appear to involve sensors, and for rectifier function, this might be Degs1 itself.

scholarly journals Chromosomal Response to Chemical Stress in Flesh Fly Sarcophaga ruficornis (Fab.) (Sarcophagidae: Diptera)

CYTOLOGIA ◽  
2009 ◽  
Vol 74 (2) ◽  
pp. 177-183
Author(s):  
Jaya Tripathi ◽  
Uma Rani Agrawal ◽  
Raghav Ram Tewari
Keyword(s):  
Chemical Stress ◽  
Flesh Fly ◽  
Sarcophaga Ruficornis

Chemical Stress Relaxation of NR Networks Prepared in the Swollen State

1986 ◽  
Vol 59 (4) ◽  
pp. 541-550 ◽  
Author(s):  
Kyung-Do Suh ◽  
Hidetoshi Oikawa ◽  
Kenkichi Murakami
Keyword(s):  
Stress Relaxation ◽  
Network Structure ◽  
Three Dimensional ◽  
Polymer Chains ◽  
Chemical Stress ◽  
Network Chain ◽  
Crosslinking Process ◽  
Dimensional Network ◽  
Chemical Relaxation ◽  
The Difference

Abstract From the experimental results of the present investigation, it is apparent that two kinds of networks which have a different three-dimensional network structure give quite different behavior of chemical stress relaxation, even if both networks have the same network chain density. The difference in three-dimensional network structure for the two kinds of rubber arises from the degree of entanglement, which changes with the concentration of the polymer chains prior to the crosslinking process. The direct cause of chemical relaxation is due to the scission of network chains by degradation, whereas the total relaxation is caused by the change of geometrical conformation of network chains. This then casts doubt on the basic concept of chemorheology which is represented by Equation 2.

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