scholarly journals An 89-year-old woman with cranial nerve dysfunction: diagnosis by observation

2005 ◽  
Vol 172 (5) ◽  
pp. 632-632 ◽  
Author(s):  
K. Milbrandt
Keyword(s):  
Cranial Nerve ◽  
Cranial Nerve Dysfunction ◽  
Nerve Dysfunction

Persistent triplegia and cranial nerve dysfunction after lysis of epidural adhesions

2021 ◽  
Vol 8 (4) ◽  
Author(s):  
Marco LA GRUA ◽  
Irene GRAZZINI ◽  
Gian L. CUNEO ◽  
Gianfranco SINDACO ◽  
Matteo ZANELLA ◽  
...  
Keyword(s):  
Cranial Nerve ◽  
Cranial Nerve Dysfunction ◽  
Nerve Dysfunction

scholarly journals Hemorrhagic vestibular schwannoma: an unusual clinical entity

1998 ◽  
Vol 5 (3) ◽  
pp. E14 ◽  
Author(s):  
Dean Chou ◽  
Prakash Sampath ◽  
Henry Brem
Keyword(s):  
Vestibular Schwannoma ◽  
Cranial Nerve ◽  
Unusual Case ◽  
Case Reports ◽  
Clinical Entity ◽  
Vestibular Schwannomas ◽  
Presenting Symptoms ◽  
Cranial Nerve Dysfunction ◽  
Nerve Dysfunction ◽  
Management Issues

Hemorrhagic vestibular schwannomas are rare entities, with only a few case reports in the literature during the last 25 years. The authors review the literature on vestibular schwannoma hemorrhage and the presenting symptoms of this entity, which include headache, nausea, vomiting, sudden cranial nerve dysfunction, and ataxia. A very unusual case is presented of a 36-year-old man, who unlike most of the patients reported in the literature, had clinically silent vestibular schwannoma hemorrhage. The authors also discuss the management issues involved in more than 1000 vestibular schwannomas treated at their institution during a 25-year period.

Rehabilitation of Lower Cranial Nerve Dysfunction

2005 ◽  
Vol 8 (1) ◽  
pp. 50-53
Author(s):  
Timothy D. Anderson ◽  
Eva Michalakis ◽  
Peter Catalano
Keyword(s):  
Cranial Nerve ◽  
Lower Cranial Nerve ◽  
Cranial Nerve Dysfunction ◽  
Nerve Dysfunction

Cranial nerve dysfunction following Gamma Knife surgery for pituitary adenomas: long-term incidence and risk factors

2012 ◽  
Vol 116 (6) ◽  
pp. 1304-1310 ◽  
Author(s):  
Christopher P. Cifarelli ◽  
David J. Schlesinger ◽  
Jason P. Sheehan
Keyword(s):  
Risk Factors ◽  
Gamma Knife ◽  
Pituitary Adenomas ◽  
Cranial Nerve ◽  
Maximal Dose ◽  
Visual Dysfunction ◽  
Cranial Nerve Dysfunction ◽  
Nerve Dysfunction

Object Gamma Knife surgery (GKS) has become a significant component of neurosurgical treatment for recurrent secretory and nonsecretory pituitary adenomas. Although the long-term risks of visual dysfunction following microsurgical resection of pituitary adenomas has been well studied, the comparable risk following radiosurgery is not well defined. This study evaluates the long-term risks of ophthalmological dysfunction following GKS for recurrent pituitary adenomas. Methods An analysis of 217 patients with recurrent secretory (n = 131) and nonsecretory (n = 86) pituitary adenomas was performed to determine the incidence of and risk factors for subsequent development of visual dysfunction. Patients underwent ophthalmological evaluation as part of post-GKS follow-up to assess for new or worsened cranial nerve II, III, IV, or VI palsies. The median follow-up duration was 32 months. The median maximal dose was 50 Gy, and the median peripheral dose was 23 Gy. A univariate analysis was performed to assess for risk factors of visual dysfunction post-GKS. Results Nine patients (4%) developed new visual dysfunctions, and these occurred within 6 hours to 34 months following radiosurgery. None of these 9 patients had tumor growth on post-GKS neuroimaging studies. Three of these patients had permanent deficits whereas in 6 the deficits resolved. Five of the 9 patients had prior GKS or radiotherapy, which resulted in a significant increase in the incidence of cranial nerve dysfunction (p = 0.0008). An increased number of isocenters (7.1 vs 5.0, p = 0.048) was statistically related to the development of visual dysfunction. Maximal dose, margin dose, optic apparatus dose, tumor volume, cavernous sinus involvement, and suprasellar extension were not significantly related to visual dysfunction (p >0.05). Conclusions Neurological and ophthalmological assessment in addition to routine neuroimaging and endocrinological follow-up are important to perform following GKS. Patients with a history of radiosurgery or radiation therapy are at higher risk of cranial nerve deficits. Also, a reduction in the number of isocenters delivered, along with volume treated, particularly in the patients with secretory tumors, appears to be the most reasonable strategy to minimize the risk to the visual system when treating recurrent pituitary adenomas with stereotactic radiosurgery.

Eighth Cranial Nerve Dysfunction in Hyperostosis Cranialis Interna

1992 ◽  
Vol 112 (1) ◽  
pp. 75-82 ◽  
Author(s):  
J. J. Manni ◽  
P. L. M. Huygen ◽  
J. F. P. Noten ◽  
W. Kuijpers
Keyword(s):  
Cranial Nerve ◽  
Eighth Cranial Nerve ◽  
Cranial Nerve Dysfunction ◽  
Nerve Dysfunction
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