scholarly journals Microvascular dysfunction in cardiac syndrome X: the role of inflammation

2006 ◽  
Vol 174 (13) ◽  
pp. 1833-1833 ◽  
Author(s):  
R. Arroyo-Espliguero
Keyword(s):  
Microvascular Dysfunction ◽  
Syndrome X ◽  
Cardiac Syndrome X ◽  
Cardiac Syndrome

Relation between cardiovascular risk factors and coronary microvascular dysfunction in cardiac syndrome X

2011 ◽  
Vol 12 (5) ◽  
pp. 322-327 ◽  
Author(s):  
Alfonso Sestito ◽  
Gaetano A Lanza ◽  
Antonio Di Monaco ◽  
Priscilla Lamendola ◽  
Giulia Careri ◽  
...  
Keyword(s):  
Risk Factors ◽  
Cardiovascular Risk ◽  
Cardiovascular Risk Factors ◽  
Microvascular Dysfunction ◽  
Syndrome X ◽  
Coronary Microvascular Dysfunction ◽  
Cardiac Syndrome X ◽  
Cardiac Syndrome

scholarly journals Evaluation of the coronary flow by the coronary clearance time in patients with cardiac syndrome X

2017 ◽  
Vol 46 (3) ◽  
pp. 1121-1129 ◽  
Author(s):  
Erkan Yildirim ◽  
Uygar Cagdas Yuksel ◽  
Murat Celik ◽  
Baris Bugan ◽  
Mutlu Gungor ◽  
...  
Keyword(s):  
Coronary Artery ◽  
Nuclear Imaging ◽  
Microvascular Dysfunction ◽  
Syndrome X ◽  
Control Group ◽  
Coronary Microvascular Dysfunction ◽  
Cardiac Syndrome X ◽  
Conventional Angiography ◽  
Cardiac Syndrome ◽  
Normal Coronary Angiography

Objective The vessels involved in the microcirculation are too small to be visualized by conventional angiography and no tools are currently available that can directly evaluate the coronary microcirculation. This study evaluated the coronary clearance frame count (CCFC) in patients with cardiac syndrome X (CSX). Methods The retrospective study enrolled patients with angina, who had a positive nuclear imaging test and normal coronary angiography; and a control group consisting of patients who underwent an angiogram to exclude coronary artery disease. Thrombosis in myocardial infarction frame count (TFC) and CCFC for each coronary artery (left anterior descending coronary artery [LAD], circumflex coronary artery [CFX] and right coronary artery [RCA]) were calculated offline. Results A total of 71 patients with CSX and 61 control patients were enrolled in the study. No significant differences were found between the two groups regarding the baseline demographic and clinical variables. The TFC of LAD, CFX and RCA were similar between the two groups. The mean CCFC-LAD, CCFC-CFX and CCFC-RCA were significantly longer in the CSX group compared with the control group. Conclusion CCFC is a simple, quantitative and highly reproducible method that might be used as a marker of coronary microvascular dysfunction.

scholarly journals TCTAP A-103 Cardiac Syndrome X – The Role of Endothelial Dysfunction

2016 ◽  
Vol 67 (16) ◽  
pp. S49-S50
Author(s):  
Ashwin Lysander ◽  
Sujai Nikhil Ramakrishnan ◽  
Raghava Chowdary ◽  
Ashish Kansal ◽  
Sri Vidhya
Keyword(s):  
Endothelial Dysfunction ◽  
Syndrome X ◽  
Cardiac Syndrome X ◽  
Cardiac Syndrome

scholarly journals Cardiac syndrome X. the possible role of Na+-Li+-countertransport activity and other pathophysiological mechanisms in pathogenesis

2013 ◽  
Vol 94 (3) ◽  
pp. 355-361
Author(s):  
V N Oslopov ◽  
Y V Oslopova ◽  
D V Borisov
Keyword(s):  
Coronary Heart Disease ◽  
Heart Disease ◽  
Vascular Dysfunction ◽  
Syndrome X ◽  
Cardiac Syndrome X ◽  
Pathophysiological Mechanisms ◽  
Cardiac Syndrome ◽  
Number Of Patients

There are numerous pathophysiological mechanisms unequally responsible for the cardiac syndrome X development. The most important is endothelium and smooth muscle cells dysfunction that can intensify vasoconstriction and depress both endothelium-dependant and endothelium-independent vasodilatation, finally leading to coronary micro vascular dysfunction as the basis of the cardiac syndrome X pathogenesis. Together with other possible mechanisms of pathogenesis, studying the importance of increased cell membrane Na+-Li+-countertransport activity seems promising. If was found that a significant number of patients with cardiac syndrome X have increased Na+-Li+-countertransport activity, which is an in vitro marker of Na+-H+-antiporter. Therefore, it is important to measure Na+-Li+-countertransport speed in patients with coronary heart disease, because its high levels increases the chance for cardiac syndrome X, which is a coronary heart disease with no anatomic signs of coronary arteries involvement.

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