Effect of Impurity Ions on Vanadium Precipitation in Vanadium-rich Solution

We characterized the two types of voltage-dependent inward currents in murine antral SMC. The HVA and LVA inward currents were identified when cells were bathed in Ca2+-containing physiological salt solution. We examined whether the LVA inward current was due to: 1) T-type Ca2+ channels, 2) Ca2+-activated Cl- channels, 3) non-selective cation channels (NSCC) or 4) voltage-dependent K+ channels with internal Cs+-rich solution. Replacement of external Ca2+ (2 mM) with equimolar Ba2+ increased the amplitude of the HVA current but blocked the LVA current. Nicardipine blocked the HVA current, and in the presence of nicardipine, T-type Ca2+ blockers failed to block LVA. The Cl- channel antagonist had little effect on LVA. Cation-free external solution completely abolished both HVA and LVA. Addition of Ca2+ in cation-free solution restored only HVA currents. Addition of K+ (5 mM) to cation-free solution induced LVA current that reversed at -20 mV. These data suggest that LVA is not due to T-type Ca2+ channels, Ca2+-activated Cl- channels or NSCC. Antral SMC express A-type K+ currents (KA) and delayed rectifying K+ currents (KV) with dialysis of high K+ (140 mM) solution. When cells were exposed to high K+ external solution with dialysis of Cs+-rich solution in the presence of nicardipine, LVA was evoked and reversed at positive potentials. These HK-induced inward currents were blocked by K+ channel blockers, 4-aminopyridine and TEA. In conclusion, LVA inward currents can be generated by K+ influx via KA and KV channels in murine antral SMC when cells were dialyzed with Cs+-rich solution.

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Effects of trapped electrons and impurity ions on ITG modes in reversed-field pinch plasmas

EPL (Europhysics Letters) ◽

10.1209/0295-5075/127/45002 ◽

2019 ◽

Vol 127 (4) ◽

pp. 45002

Author(s):

J. C. Li ◽

S. F. Liu ◽

W. Kong ◽

S. C. Guo ◽

J. Q. Dong

Keyword(s):

Reversed Field Pinch ◽

Trapped Electrons ◽

Impurity Ions ◽

Reversed Field

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Composition profiles of (Hg,Cd)Te liquid phase epitaxy layers grown from Te-rich solution

Journal of Crystal Growth ◽

10.1016/0022-0248(94)90053-1 ◽

1994 ◽

Vol 143 (3-4) ◽

pp. 176-183 ◽

Cited By ~ 2

Author(s):

V. Jović ◽

Z. Djurić ◽

Z. Jakšić ◽

M. Popović

Keyword(s):

Liquid Phase ◽

Liquid Phase Epitaxy ◽

Rich Solution ◽

Composition Profiles

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Optical Spectra of Cr3+Impurity Ions in Ferroelectric LiNbO3and LiTaO3

The Journal of Chemical Physics ◽

10.1063/1.1671234 ◽

1969 ◽

Vol 50 (4) ◽

pp. 1501-1510 ◽

Cited By ~ 149

Author(s):

A. M. Glass

Keyword(s):

Optical Spectra ◽

Impurity Ions

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Calcium channels and excitation–contraction coupling in cardiac cells. I. Two components of contraction in guinea-pig papillary muscle

Canadian Journal of Physiology and Pharmacology ◽

10.1139/y87-284 ◽

1987 ◽

Vol 65 (9) ◽

pp. 1821-1831 ◽

Cited By ~ 4

Author(s):

E. Honoré ◽

M. M. Adamantidis ◽

B. A. Dupuis ◽

C. E. Challice ◽

P. Guilbault

Keyword(s):

Membrane Potential ◽

Guinea Pig ◽

Papillary Muscle ◽

Cardiac Cells ◽

Resting Membrane Potential ◽

Tonic Component ◽

Contraction Coupling ◽

Rich Solution ◽

The Relationship ◽

Guinea Pig Atria

Biphasic contractions have been obtained in guinea-pig papillary muscle by inducing partial depolarization in K+-rich solution (17 mM) containing 0.3 μM isoproterenol; whereas in guinea-pig atria, the same conditions led to monophasic contractions corresponding to the first component of contraction in papillary muscle. The relationships between the amplitude of the two components of the biphasic contraction and the resting membrane potential were sigmoidal curves. The first component of contraction was inactivated for membrane potentials less positive than those for the second component. In Na+-low solution (25 mM), biphasic contraction became monophasic subsequent to the loss of the second component, but tetraethylammonium unmasked the second component of contraction. The relationship between the amplitude of the first component of contraction and the logarithm of extracellular Ca2+ concentration was complex, whereas for the second component it was linear. When Ca2+ ions were replaced by Sr2+ ions, only the second component of contraction was observed. It is suggested that the first component of contraction may be triggered by a Ca2+ release from sarcoplasmic reticulum, induced by the fast inward Ca2+ current and (or) by the depolarization. The second component of contraction may be due to a direct activation of contractile proteins by Ca2+ entering the cell along with the slow inward Ca2+ current and diffusing through the sarcoplasm. These results do not exclude the existence of a third "tonic" component, which could possibly be mixed with the second component of contraction.

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