scholarly journals Heat shock pretreatment inhibited the release of Smac/DIABLO from mitochondria and apoptosis induced by hydrogen peroxide in cardiomyocytes and C2C12 myogenic cells

2005 ◽  
Vol 10 (3) ◽  
pp. 252 ◽  
Author(s):  
Bimei Jiang ◽  
Weimin Xiao ◽  
Yongzhong Shi ◽  
Meidong Liu ◽  
Xianzhong Xiao
Keyword(s):  
Hydrogen Peroxide ◽  
Heat Shock ◽  
Myogenic Cells ◽  
C2c12 Myogenic Cells ◽  
Heat Shock Pretreatment

HEAT SHOCK PRETREATMENT PREVENTS HYDROGEN PEROXIDE INJURY OF PULMONARY ENDOTHELIAL CELLS AND MACROPHAGES IN CULTURE

Shock ◽  
1996 ◽  
Vol 6 (2) ◽  
pp. 134-141 ◽  
Author(s):  
Yan-Ru Wang ◽  
Xian-Zong Xiao ◽  
Sheng-Ning Huang ◽  
Fei-Jun Luo ◽  
Jia-Lu You ◽  
...  
Keyword(s):  
Hydrogen Peroxide ◽  
Endothelial Cells ◽  
Heat Shock ◽  
Pulmonary Endothelial Cells ◽  
Heat Shock Pretreatment

Role of Smac/DIABLO in hydrogen peroxide-induced apoptosis in C2C12 myogenic cells

2005 ◽  
Vol 39 (5) ◽  
pp. 658-667 ◽  
Author(s):  
Bimei Jiang ◽  
Weimin Xiao ◽  
Yongzhong Shi ◽  
Meidong Liu ◽  
Xianzhong Xiao
Keyword(s):  
Hydrogen Peroxide ◽  
Myogenic Cells ◽  
Induced Apoptosis ◽  
C2c12 Myogenic Cells

scholarly journals Attenuation of sepsis-induced apoptosis by heat shock pretreatment in rats

2000 ◽  
Vol 5 (3) ◽  
pp. 188 ◽  
Author(s):  
Hsiang-Wen Chen ◽  
Chin Hsu ◽  
Sheng-I Lue ◽  
Rei-Cheng Yang
Keyword(s):  
Heat Shock ◽  
Induced Apoptosis ◽  
Heat Shock Pretreatment

scholarly journals Genes for hydrogen peroxide detoxification and adaptation contribute to protection against heat shock in Xanthomonas campestris pv. campestris

2011 ◽  
Vol 317 (1) ◽  
pp. 60-66 ◽  
Author(s):  
Sarinya Buranajitpakorn ◽  
Anong Piwkam ◽  
Nisanart Charoenlap ◽  
Paiboon Vattanaviboon ◽  
Skorn Mongkolsuk
Keyword(s):  
Hydrogen Peroxide ◽  
Heat Shock ◽  
Xanthomonas Campestris ◽  
Xanthomonas Campestris Pv Campestris

Hydrogen peroxide induces midzonal heat shock protein 72 and apoptosis in sinusoidal endothelial cells of hypoxic rat liver

2000 ◽  
Vol 28 (5) ◽  
pp. 1509-1514 ◽  
Author(s):  
Satoru Motoyama ◽  
Satoshi Saito ◽  
Maria Elena Alojado ◽  
Hideaki Itoh ◽  
Michihiko Kitamura ◽  
...  
Keyword(s):  
Hydrogen Peroxide ◽  
Endothelial Cells ◽  
Heat Shock ◽  
Heat Shock Protein ◽  
Rat Liver ◽  
Heat Shock Protein 72 ◽  
Sinusoidal Endothelial Cells

A feedback inhibition between nitric oxide and hydrogen peroxide in the heat shock pathway in Arabidopsis seedlings

2014 ◽  
Vol 75 (2) ◽  
pp. 503-509 ◽  
Author(s):  
Dan Wu ◽  
Hongye Chu ◽  
Lixiu Jia ◽  
Kunming Chen ◽  
Liqun Zhao
Keyword(s):  
Nitric Oxide ◽  
Hydrogen Peroxide ◽  
Heat Shock ◽  
Feedback Inhibition

The efficacy of long-lasting residual drinking water disinfectants based on hydrogen peroxide and silver

2000 ◽  
Vol 42 (1-2) ◽  
pp. 293-298 ◽  
Author(s):  
R. Pedahzur ◽  
D. Katzenelson ◽  
N. Barnea ◽  
O. Lev ◽  
H.I. Shuval ◽  
...  
Keyword(s):  
Hydrogen Peroxide ◽  
Heat Shock ◽  
Stress Responses ◽  
Heat Shock Response ◽  
Chemical Properties ◽  
Bactericidal Effect ◽  
Physiological Effects ◽  
Mechanisms Of Toxicity ◽  
E Coli ◽  
Shock Response

The aim of the present work was to evaluate the disinfectant capacity and the possible fields of application of a combined silver and hydrogen peroxide (HP) water disinfectant. The findings demonstrated the high bactericidal action of silver (on E. coli) and its relatively ineffective virucidal effect (on MS-2 phage). HP was found to have a small bactericidal effect and a mild virucidal one. When combined, silver and HP usually exhibited a synergistic action on the viability of E. coli and on the luminescence of recombinant luminescent E. coli. In some instances, the combined bactericidal effects were 1000-fold higher than the sum of the separate ones. No increased virucidal action was observed. The biocidal action of the combination generally increased with increasing temperature and pH, and decreased in secondary and tertiary effluents. The physiological effects and mechanisms of toxicity of HP, silver and their combinations, were assessed by monitoring the induction of stress promoters upon exposure to the active agents, and by assessing the sensitivity of E. coli mutated in major stress responses to HP, silver and their combinations. The results showed that HP induced a wide array of stress responses, that both silver and HP induced promoters regulated by the heat shock response, and that the dnaK promoter (regulated by the heat shock response) was synergistically induced. The mutant sensitivity tests showed that bacteria deficient in the ability to activate central cellular stress responses (SOS, heat shock, stationary phase, oxidative) were hypersensitive to both HP and silver. These results imply that cellular proteins, and possibly the DNA, are the cellular moieties chiefly affected. The above findings suggest that the potentiated effect of HP and silver is a metabolically dependant/related process that stems from a combination and/or accumulation of physiological effects exerted by the active ingredients. The physico-chemical properties of the combined disinfectant, and its disinfection capacity, points to its potential application as a long-term secondary residual disinfectant for water of relatively high quality.

Sign in / Sign up

Export Citation Format

Share Document