scholarly journals Chlamydia trachomatis inclusion membrane protein MrcA interacts with the inositol 1,4,5-trisphosphate receptor type 3 (ITPR3) to regulate extrusion formation

2018 ◽  
Vol 14 (3) ◽  
pp. e1006911 ◽  
Author(s):  
Phu Hai Nguyen ◽  
Erika I. Lutter ◽  
Ted Hackstadt
Keyword(s):  
Chlamydia Trachomatis ◽  
Membrane Protein ◽  
Receptor Type ◽  
Inclusion Membrane ◽  
Inositol 1,4,5 Trisphosphate ◽  
Trisphosphate Receptor ◽  
Type 3

Differential modulation of inositol 1,4,5-trisphosphate receptor type 1 and type 3 by ATP

Cell Calcium ◽  
2000 ◽  
Vol 27 (5) ◽  
pp. 257-267 ◽  
Author(s):  
K. Maes ◽  
L. Missiaen ◽  
P. De Smet ◽  
S. Vanlingen ◽  
G. Callewaert ◽  
...  
Keyword(s):  
Receptor Type ◽  
Differential Modulation ◽  
Inositol 1,4,5 Trisphosphate ◽  
Trisphosphate Receptor ◽  
Type 3

scholarly journals Inositol 1,4,5‑trisphosphate receptor type 3 is involved in resistance to apoptosis and maintenance of human hepatocellular carcinoma

2021 ◽  
Vol 23 (1) ◽  
Author(s):  
Marcone Dos Santos ◽  
Andressa França ◽  
Antônio Lima Filho ◽  
Rodrigo Florentino ◽  
Paulo Diniz ◽  
...  
Keyword(s):  
Hepatocellular Carcinoma ◽  
Human Hepatocellular Carcinoma ◽  
Receptor Type ◽  
Inositol 1,4,5 Trisphosphate ◽  
Trisphosphate Receptor ◽  
Type 3

scholarly journals Inositol 1,4,5-Trisphosphate Receptor Type 3 Regulates Neuronal Growth Cone Sensitivity to Guidance Signals

iScience ◽  
2020 ◽  
Vol 23 (3) ◽  
pp. 100963 ◽  
Author(s):  
Carmen Chan ◽  
Noriko Ooashi ◽  
Hiroki Akiyama ◽  
Tetsuko Fukuda ◽  
Mariko Inoue ◽  
...  
Keyword(s):  
Growth Cone ◽  
Receptor Type ◽  
Neuronal Growth ◽  
Inositol 1,4,5 Trisphosphate ◽  
Neuronal Growth Cone ◽  
Cone Sensitivity ◽  
Trisphosphate Receptor ◽  
Type 3

Immunohistochemical study of inositol 1,4,5-trisphosphate receptor type 3 in rat central nervous system

Neuroreport ◽  
1995 ◽  
Vol 6 (2) ◽  
pp. 273-276 ◽  
Author(s):  
Miki Yamamoto-Hino ◽  
Atsushi Miyawaki ◽  
Hitoshi Kawano ◽  
Tomoyasu Sugiyama ◽  
Teiichi Furuichi ◽  
...  
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Immunohistochemical Study ◽  
Receptor Type ◽  
Inositol 1,4,5 Trisphosphate ◽  
Trisphosphate Receptor ◽  
Type 3

Promoter structure and gene expression of the mouse inositol 1,4,5-trisphosphate receptor type 3 gene

Gene ◽  
2001 ◽  
Vol 275 (1) ◽  
pp. 169-176 ◽  
Author(s):  
Taka-aki Tamura ◽  
Mitsuhiro Hashimoto ◽  
Jun Aruga ◽  
Yoshiyuki Konishi ◽  
Midori Nakagawa ◽  
...  
Keyword(s):  
Gene Expression ◽  
Receptor Type ◽  
Promoter Structure ◽  
Inositol 1,4,5 Trisphosphate ◽  
Trisphosphate Receptor ◽  
Type 3

scholarly journals Inositol 1,4,5-trisphosphate receptor type 3 plays a protective role in hepatocytes during hepatic ischemia-reperfusion injury

Cell Calcium ◽  
2020 ◽  
Vol 91 ◽  
pp. 102264
Author(s):  
Antônio Carlos Melo Lima Filho ◽  
Andressa França ◽  
Rodrigo M. Florentino ◽  
Marcone Loiola dos Santos ◽  
Fernanda de Oliveira Lemos ◽  
...  
Keyword(s):  
Reperfusion Injury ◽  
Ischemia Reperfusion Injury ◽  
Ischemia Reperfusion ◽  
Protective Role ◽  
Receptor Type ◽  
Hepatic Ischemia ◽  
Inositol 1,4,5 Trisphosphate ◽  
Hepatic Ischemia Reperfusion ◽  
Trisphosphate Receptor ◽  
Type 3

scholarly journals Functional analysis of the green fluorescent protein-tagged inositol 1,4,5-trisphosphate receptor type 3 in Ca2+ release and entry in DT40 B lymphocytes

2004 ◽  
Vol 382 (3) ◽  
pp. 793-801 ◽  
Author(s):  
Takao MORITA ◽  
Akihiko TANIMURA ◽  
Akihiro NEZU ◽  
Tomohiro KUROSAKI ◽  
Yosuke TOJYO
Keyword(s):  
Green Fluorescent Protein ◽  
Fluorescent Protein ◽  
Cell Receptor ◽  
Receptor Type ◽  
Intact Cells ◽  
Entry Pathway ◽  
Inositol 1,4,5 Trisphosphate ◽  
Green Fluorescent ◽  
Trisphosphate Receptor ◽  
Type 3

We examined the function of GFP-IP3R3 (green fluorescent protein-tagged inositol 1,4,5-trisphosphate receptor type 3) in Ca2+ release and entry using a mutant DT40 cell line (IP3R-KO) in which all three IP3R genes had been disrupted. GFP-IP3R3 fluorescence largely overlapped with the distribution of endoplasmic reticulum, whereas a portion of GFP-IP3R3 apparently co-localized with the plasma membrane. The application of IP3 to permeabilized WT (wild-type) DT40 cells induced Ca2+ release from internal stores. Although this did not occur in IP3R-KO cells it was restored by expression of GFP-IP3R3. In intact cells, application of anti-IgM, an activator of the BCR (B-cell receptor), or trypsin, a protease-activated receptor 2 agonist, did not cause any Ca2+ response in IP3R-KO cells, whereas these treatments induced oscillatory or transient Ca2+ responses in GFP-IP3R3-expressing IP3R-KO cells, as well as in WT cells. In addition, BCR activation elicited Ca2+ entry in WT and GFP-IP3R3-expressing IP3R-KO cells but not in IP3R-KO cells. This BCR-mediated Ca2+ entry was observed in the presence of La3+, which blocks capacitative Ca2+ entry. Thapsigargin depleted Ca2+ stores and led to Ca2+ entry in IP3R-KO cells irrespective of GFP-IP3R3 expression. In contrast with BCR stimulation, thapsigargin-induced Ca2+ entry was completely blocked by La3+, suggesting that the BCR-mediated Ca2+ entry pathway is distinct from the capacitative Ca2+ entry pathway. The present study demonstrates that GFP-IP3R3 could compensate for native IP3R in both IP3-induced Ca2+ release and BCR-mediated Ca2+ entry.

Protein kinase C decreases the apparent affinity of the inositol 1,4,5-trisphosphate receptor type 3 in RINm5F cells

Cell Calcium ◽  
2007 ◽  
Vol 42 (3) ◽  
pp. 323-331 ◽  
Author(s):  
Annabelle Z. Caron ◽  
Benoit Chaloux ◽  
Guillaume Arguin ◽  
Gaetan Guillemette
Keyword(s):  
Protein Kinase C ◽  
Protein Kinase ◽  
Receptor Type ◽  
Rinm5f Cells ◽  
Apparent Affinity ◽  
Kinase C ◽  
Inositol 1,4,5 Trisphosphate ◽  
Trisphosphate Receptor ◽  
Type 3
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