scholarly journals Prolonged Gallbladder Hydrops in a Kawasaki Disease Patient

2018 ◽  
Vol 24 (2) ◽  
pp. 107
Author(s):  
Kyung Lim Yoon ◽  
Do Hee Kim ◽  
Mi Young Han ◽  
Sung Ho Cha ◽  
Hyun Cheol Kim
2021 ◽  
Vol 71 (3) ◽  
pp. 199-201
Author(s):  
Yuiko Suzuki ◽  
Tetsuro Tamai ◽  
Shuhei Takahashi ◽  
Akiho Ueda ◽  
Rena Okada ◽  
...  

1997 ◽  
Vol 61 (2) ◽  
pp. 197-200 ◽  
Author(s):  
Koh Arakawa ◽  
Takako Akita ◽  
Kenya Nishizawa ◽  
Akira Kurita ◽  
Haruo Nakamura ◽  
...  

2003 ◽  
Vol 53 (1) ◽  
pp. 171-171
Author(s):  
Jun-Ichi Hirao ◽  
Osamu Arisaka ◽  
Ken-Ichi Sugita ◽  
Mitsuoki Eguchi

2021 ◽  
Vol 22 (11) ◽  
pp. 5655
Author(s):  
Heather Jackson ◽  
Stephanie Menikou ◽  
Shea Hamilton ◽  
Andrew McArdle ◽  
Chisato Shimizu ◽  
...  

The aetiology of Kawasaki disease (KD), an acute inflammatory disorder of childhood, remains unknown despite various triggers of KD having been proposed. Host ‘omic profiles offer insights into the host response to infection and inflammation, with the interrogation of multiple ‘omic levels in parallel providing a more comprehensive picture. We used differential abundance analysis, pathway analysis, clustering, and classification techniques to explore whether the host response in KD is more similar to the response to bacterial or viral infections at the transcriptomic and proteomic levels through comparison of ‘omic profiles from children with KD to those with bacterial and viral infections. Pathways activated in patients with KD included those involved in anti-viral and anti-bacterial responses. Unsupervised clustering showed that the majority of KD patients clustered with bacterial patients on both ‘omic levels, whilst application of diagnostic signatures specific for bacterial and viral infections revealed that many transcriptomic KD samples had low probabilities of having bacterial or viral infections, suggesting that KD may be triggered by a different process not typical of either common bacterial or viral infections. Clustering based on the transcriptomic and proteomic responses during KD revealed three clusters of KD patients on both ‘omic levels, suggesting heterogeneity within the inflammatory response during KD. The observed heterogeneity may reflect differences in the host response to a common trigger, or variation dependent on different triggers of the condition.


2017 ◽  
Vol 57 (3) ◽  
pp. 341-343 ◽  
Author(s):  
Qiufeng Sun ◽  
Jianmin Zhang ◽  
Yungang Yang

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