scholarly journals Effect of Treadmill Exercise and Hydrogen-rich Water Intake on Serum Oxidative and Anti-oxidative Metabolites in Serum of Thoroughbred Horses

2013 ◽  
Vol 24 (1) ◽  
pp. 1-8 ◽  
Author(s):  
Hirokazu TSUBONE ◽  
Masakazu HANAFUSA ◽  
Maiko ENDO ◽  
Noboru MANABE ◽  
Atsushi HIRAGA ◽  
...  
Keyword(s):  
Water Intake ◽  
Treadmill Exercise ◽  
Thoroughbred Horses ◽  
Oxidative Metabolites

Digestion characteristics of two forms of preserved lucerne forage fed to mature horses

2012 ◽  
Vol 1 ◽  
Author(s):  
L. A. Waldron ◽  
D. G. Thomas ◽  
I. Pryor
Keyword(s):  
New Zealand ◽  
Energy Intake ◽  
Water Intake ◽  
Reference Values ◽  
Dry Matter ◽  
Dry Matter Intake ◽  
Small Scale ◽  
Digestible Energy ◽  
Thoroughbred Horses ◽  
Nutritional Benefits

SummaryA trial was conducted, using 12 mature thoroughbred horses as a cross-over design, to compare the water and energy intake and the digestibility of dry lucerne chaff (LC) against a controlled fermented lucerne (CFL) product (HNF Fiber®; Fiber Fresh Feeds Ltd, Reporoa, New Zealand). Significant (P < 0.05) increases in dry matter intake (20%), energy intake (20%), retained energy (32%) and digestible energy (22%) were observed for the CFL compared to the LC. In a concurrent small scale study within the trial, numeric improvements in water intake (18%) for the CFL were recorded. The results demonstrated that the standard reference values underestimate the digestibility and nutritional contribution made by CFL when fed to horses, and that this type of forage has increased nutritional benefits.

Reliability of cardiorespiratory measurements with a new ergospirometer during intense treadmill exercise in Thoroughbred horses

2005 ◽  
Vol 169 (2) ◽  
pp. 223-231 ◽  
Author(s):  
R.A. Curtis ◽  
K. Kusano ◽  
D.L. Evans ◽  
N.H. Lovell ◽  
D.R. Hodgson
Keyword(s):  
Treadmill Exercise ◽  
Thoroughbred Horses

Cardiac output and oxygen consumption in exercising Thoroughbred horses

1987 ◽  
Vol 253 (6) ◽  
pp. R890-R895 ◽  
Author(s):  
J. M. Weber ◽  
G. P. Dobson ◽  
W. S. Parkhouse ◽  
D. Wheeldon ◽  
J. C. Harman ◽  
...  
Keyword(s):  
Heart Rate ◽  
Cardiac Output ◽  
Oxygen Consumption ◽  
Maximal Exercise ◽  
Treadmill Exercise ◽  
Aerobic Scope ◽  
O2 Uptake ◽  
Thoroughbred Racehorses ◽  
Thoroughbred Horses ◽  
Mean Heart Rate

This study characterizes the effects of exercise on the cardiac output (Q) and the metabolic rate (VO2) of trained Thoroughbred racehorses. Heart rate, Q, and arteriovenous (a-v)O2 difference were measured at rest and at three levels of submaximal treadmill exercise (1.6 m/s walk and 3-4 m/s trot at 6% incline, and 6.5 m/s horizontal canter). Heart rate and (a-v)O2 difference were also measured during maximal exercise (12.5 m/s gallop, 5% incline) to obtain an estimate of maximum O2 uptake (VO2max). The walk, trot, and canter represented 25, 45, and 55% VO2max. Mean heart rate went from 48.9 (rest) to 197 beats/min (gallop). Q ranged from 106 (rest) to 571 ml.min-1.kg-1 (canter), and stroke volume went from 1.34 (rest) to a maximum of 1.58 liters (walk). Thoroughbreds were able to bring hematocrit from 38 (rest) to 63% (gallop), and this adjustment allowed them to reach an impressive (a-v)O2 difference of 23 vol%, which represents a fivefold increase over resting values. These outstanding athletes probably support an aerobic scope of 40-fold.

Effect of prolonged heavy exercise on pulmonary gas exchange in horses

1998 ◽  
Vol 84 (5) ◽  
pp. 1723-1730 ◽  
Author(s):  
S. R. Hopkins ◽  
W. M. Bayly ◽  
R. F. Slocombe ◽  
H. Wagner ◽  
P. D. Wagner
Keyword(s):  
Gas Exchange ◽  
Oxygen Uptake ◽  
Gas Phase ◽  
Maximal Exercise ◽  
Treadmill Exercise ◽  
Submaximal Exercise ◽  
Pulmonary Gas Exchange ◽  
Diffusion Limitation ◽  
Short Term ◽  
Thoroughbred Horses

During short-term maximal exercise, horses have impaired pulmonary gas exchange, manifested by diffusion limitation and arterial hypoxemia, without marked ventilation-perfusion (V˙a/Q˙) inequality. Whether gas exchange deteriorates progressively during prolonged submaximal exercise has not been investigated. Six thoroughbred horses performed treadmill exercise at ∼60% of maximal oxygen uptake until exhaustion (28–39 min). Multiple inert gas, blood-gas, hemodynamic, metabolic rate, and ventilatory data were obtained at rest and 5-min intervals during exercise. Oxygen uptake, cardiac output, and alveolar-arterial[Formula: see text] gradient were unchanged after the first 5 min of exercise. Alveolar ventilation increased progressively during exercise, from increased tidal volume and respiratory frequency, resulting in an increase in arterial[Formula: see text] and decrease in arterial[Formula: see text]. At rest there was minimalV˙a/Q˙inequality, log SD of the perfusion distribution (log SDQ˙) = 0.20. This doubled by 5 min of exercise (log SDQ˙ = 0.40) but did not increase further. There was no evidence of alveolar-end-capillary diffusion limitation during exercise. However, there was evidence for gas-phase diffusion limitation at all time points, and enflurane was preferentially overretained. Horses maintain excellent pulmonary gas exchange during exhaustive, submaximal exercise. AlthoughV˙a/Q˙inequality is greater than at rest, it is less than observed in most mammals and the effect on gas exchange is minimal.

Association between muscle acetyl-CoA and acetylcarnitine levels in the exercising horse

1990 ◽  
Vol 69 (1) ◽  
pp. 42-45 ◽  
Author(s):  
J. I. Carlin ◽  
R. C. Harris ◽  
G. Cederblad ◽  
D. Constantin-Teodosiu ◽  
D. H. Snow ◽  
...  
Keyword(s):  
Mitochondrial Membrane ◽  
Treadmill Exercise ◽  
Close Correlation ◽  
The State ◽  
Acetyl Coa ◽  
Whole Cell ◽  
Thoroughbred Horses ◽  
Rate Limiting ◽  
High Work

Treadmill exercise of 2-min duration and increasing intensity resulted in increased formation of acetyl-CoA and acetylcarnitine in working muscle of Thoroughbred horses. At high work intensities a plateau was reached for both acetyl-CoA (approximately 50 mumols/kg dry muscle) and acetylcarnitine (approximately 20 mmol/kg dry muscle). Postexercise concentrations were significantly (P less than 0.001) correlated; [acetylcarnitine] = 349.[acetyl-CoA] + 2.4. The results indicate that approximately 350 mumols acetylcarnitine were accumulated for every 1 mumol acetyl-CoA. Under the conditions of exercise used it is probable that most of the acetyl-CoA formed is generated through the intramitochondrial decarboxylation of pyruvate. The acetyl groups of acetyl-CoA are apparently redistributed throughout the whole cell through formation of acetylcarnitine, which readily transverses the mitochondrial membrane. Despite the redistribution, however, the close correlation between acetylcarnitine and acetyl-CoA would indicate that equilibrium was maintained and that neither acetylcarnitine transferase nor carnitine/acetylcarnitine translocase were rate limiting. There is some question as to whether the changes observed relate directly to exercise itself or to the state in muscle 10 s or more after exercise.

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