scholarly journals Changes in Plasma Endothelin-1 Concentration during Blood Volume Depletion and Expansion: Role of the Cardiopulmonary Baroreflex.

1995 ◽  
Vol 18 (1) ◽  
pp. 43-46 ◽  
Author(s):  
Yuhei Kawano ◽  
Hiroki Yoshimi ◽  
Hiroaki Matsuoka ◽  
Teruo Omae
Keyword(s):  
Blood Volume ◽  
Volume Depletion ◽  
Endothelin 1 ◽  
Cardiopulmonary Baroreflex

Restricted postexercise pulmonary diffusion capacity and central blood volume depletion

1997 ◽  
Vol 83 (1) ◽  
pp. 11-17 ◽  
Author(s):  
Birgitte Hanel ◽  
Inge Teunissen ◽  
Alan Rabøl ◽  
Jørgen Warberg ◽  
Niels H. Secher
Keyword(s):  
Blood Volume ◽  
Electrical Impedance ◽  
Control Group ◽  
Central Blood Volume ◽  
Volume Depletion ◽  
Diffusion Capacity ◽  
Pulmonary Diffusion ◽  
Before And After ◽  
Pulmonary Diffusion Capacity

Hanel, Birgitte, Inge Teunissen, Alan Rabøl, Jørgen Warberg, and Niels H. Secher. Restricted postexercise pulmonary diffusion capacity and central blood volume depletion. J. Appl. Physiol. 83(1): 11–17, 1997.—Pulmonary diffusion capacity for carbon monoxide (Dl CO), regional electrical impedance (Z0), and the distribution of technetium-99m-labeled erythrocytes together with concentration of plasma atrial natriuretic peptide (ANP) were determined before and after a 6-min “all-out” row in nine oarsmen and in six control subjects. Two and one-half hours after exercise in the upright seated position, Dl CO was reduced by 6 (−2 to 21; median and range) %, the thoracic-to-thigh electrical impedance ratio (Z0 thorax/Z0 thigh) rose by 14 (−1 to 29) %, paralleled by a 7 (−3 to 11) % decrease and a 3 (−5 to 12) % increase in the thoracic and thigh blood volume, respectively. These responses were associated with a decrease in the plasma ANP concentration from 15 (13–31) to 12 (9–27) pmol/l ( P < 0.05). Similarly, in the supine position, Z0 thorax/Z0 thighincreased by 10 (−5 to 28) % when Dl CO was reduced 12 (6–26) % ( P < 0.05), whereas Dl COremained stable in the control group. The increase in Z0 thorax/Z0 thighand the corresponding redistribution of the blood volume in both body positions show that approximately one-half of the postexercise reduction of Dl CO is explained by a decrease in the pulmonary blood volume. The role of a reduced postexercise central blood volume is underscored by the lower plasma ANP, which aids in upregulating the blood volume after exercise in athletes.

scholarly journals THE ROLE OF VOLUME DEPLETION IN FUROSEMIDE ENHANCED AMINOGLYCOSIDE NEPHROTOXICITY

1980 ◽  
Vol 14 (8) ◽  
pp. 1007-1007
Author(s):  
R D Adelman ◽  
W Spangler ◽  
D Thomson ◽  
F Beasom
Keyword(s):  
Volume Depletion

scholarly journals Endothelin-1 inhibits adipogenesis: Role of phosphorylation of Akt and ERK1/2

FEBS Letters ◽  
2006 ◽  
Vol 580 (24) ◽  
pp. 5765-5771 ◽  
Author(s):  
Indranil Bhattacharya ◽  
Axel Ullrich
Keyword(s):  
Endothelin 1

ACUTE CONFUSIONAL STATES AND DEMENTIA IN THE ELDERLY: THE ROLE OF DEHYDRATION/VOLUME DEPLETION, PHYSICAL ILLNESS AND AGE

1980 ◽  
Vol 9 (3) ◽  
pp. 137-146 ◽  
Author(s):  
D. G. SEYMOUR ◽  
P. J. HENSCHKE ◽  
R. D. T. CAPE ◽  
A. J. CAMPBELL
Keyword(s):  
The Elderly ◽  
Physical Illness ◽  
Volume Depletion ◽  
Confusional States

Fluctuations in O2 stores and gas exchange with passive changes in posture

1975 ◽  
Vol 39 (1) ◽  
pp. 47-53 ◽  
Author(s):  
J. A. Loeppky ◽  
U. C. Luft
Keyword(s):  
Cardiac Output ◽  
Gas Exchange ◽  
Metabolic Rate ◽  
Blood Volume ◽  
Mass Spectrometer ◽  
Rapid Loss ◽  
Pulmonary Capillary ◽  
O2 Transfer

To clarify the role of O2 stores in the fluctuations in VO2 observed with changing posture, O2 intake (Veo2) and pulmonary capillary O2 transfer (Vpco2) were calculated breath by breath with a box-balloon sprometer and mass spectrometer. Changes in O2 stores of the lungs (O2L) and blood (O2b) were computed assuming metabolic rate (Vco2) constant (O2L = Veo2 - Vpco2; O2b = Vpco2 - Vco2). Measurements were made before, during, and after passive tilt to 60 degrees and on return to recumbency after 10 min erect. From supine to upright O2L increased rapidly and O2b dropped slowly, creating a net deficit in Veo2 of 130 ml in 10 min. Return to supine caused rapid loss in O2L and gain in O2b with a net Veo2 excess of 117 ml. Shifts in O2b were 2.5 times greater but opposite to shifts in O2L. Changes in O2b result from shifts in blood volume and flow more than from changes in cardiac output. Refilling of O2b, matching loss while upright, caused transient hypoxia with significant hyperpnea.

Role of Endothelin-1 in Myocardial Failure

1999 ◽  
Vol 111 (5) ◽  
pp. 417-422 ◽  
Author(s):  
Flora Sam ◽  
Wilson S. Colucci
Keyword(s):  
Endothelin 1 ◽  
Myocardial Failure
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