Epidemiologic characterization of sudden infant death syndrome in relation to climate factors and air pollution in Korea: a case-crossover study

2016 ◽  
Vol 2016 (1) ◽  
Author(s):  
Myung-Jae Hwang* ◽  
Hae-Kwan Cheong
2016 ◽  
Vol 2016 (1) ◽  
Author(s):  
Nuredin I Mohammed* ◽  
Jon G Ayres ◽  
Jouni J K Jaakkola ◽  
Ian J Litchfield

PEDIATRICS ◽  
1995 ◽  
Vol 96 (6) ◽  
pp. 1106-1110
Author(s):  
Harald H. Knöbel ◽  
Chien-Jen Chen ◽  
Kung-Yee Liang

Objective. To examine the possible role of weather and air pollution in sudden infant death syndrome (SIDS) and suffocation. Methods. Poisson regression analysis was carried out to measure the association between daily rates of SIDS per 1000 live births and daily average values of visibility and temperature in Taiwan between 1981 and 1991. The optimetrical measure of air pollution was used to represent pollution over a whole area rather than at a point source. Results. Mortality from SIDS per 1000 live births was 3.3 times greater in the lowest category of visibility on the day of death than in the highest category; this rate ratio was 3.4 for the average visibility during the 9 days before death. Adjustment for population size, season, level of urbanization, incidence of deaths from respiratory tract infections, temperature, air pressure, sunshine, rainfall, relative humidity, and windspeed increased these rate ratios to 3.8 and 5.1, respectively. This suggests that the relationship between air pollution and SIDS is not biased by ecological confounders. For temperature the rate ratios were between 3.3 and 4.0. Conclusions. Our findings confirm the association of climatic temperature and air pollution with SIDS.


PEDIATRICS ◽  
1996 ◽  
Vol 98 (4) ◽  
pp. 797-798
Author(s):  
Harald H. Knöbel ◽  
Chien-Jen Chen ◽  
Kung-Yee Liang

We are grateful to Steven Koehler for his comments on our study on sudden infant death syndrome (SIDS) and air pollution in Taiwan.1 The criticism focuses on the autopsy rate and the new, but well-referenced finding, that in Taiwan, and probably other Asian countries, the majority of classic SIDS is coded as suffocation. Following is an itemized reply to the issues raised. SIDS has been assumed to be a phenomenon of western countries. In China and Hong Kong it is thought to be virtually nonexistent2,3 Japan and Taiwan have officially published rates that are extremely low.4,5


2015 ◽  
Vol 123 (7) ◽  
pp. 712-716 ◽  
Author(s):  
Nathalie Auger ◽  
William D. Fraser ◽  
Audrey Smargiassi ◽  
Tom Kosatsky

Chemosphere ◽  
2020 ◽  
pp. 129515
Author(s):  
Yin-Ting Chen ◽  
Chia-Lin Liu ◽  
Chi-Jen Chen ◽  
Mei-Huei Chen ◽  
Chien-Yi Chen ◽  
...  

PEDIATRICS ◽  
2004 ◽  
Vol 113 (6) ◽  
pp. e628-e631 ◽  
Author(s):  
R. Dales ◽  
R. T. Burnett ◽  
M. Smith-Doiron ◽  
D. M. Stieb ◽  
J. R. Brook

Author(s):  
Myung-Jae Hwang ◽  
Hae-Kwan Cheong ◽  
Jong-Hun Kim

Sudden infant death syndrome (SIDS) is an occasional cause of unexpected mortality in infancy. While various etiological factors have been hypothesized, air pollution has been consistently presented as an environmental factor. In this study, we aimed to estimate the risk of SIDS in relation to exposure to air pollution and the effects of its modifying factors. A mortality dataset with supplementary infant mortality survey data from Statistics Korea was used and combined the concentration of ambient air pollution data from AirKorea based on the date of death and residential addresses of the SIDS cases. Odds ratios (ORs) were estimated according to birthweight, gestational age, maternal age, and infant age using a time-stratified case-crossover study design. The risk of exposure to particulate matter of less than 10 μm in diameter (PM10), nitrogen dioxide (NO2), carbon monoxide (CO), and sulfur dioxide was estimated. The number of deaths due to SIDS was 454 (253 males and 201 females). The OR per 27.8 µg/m3 increment of PM10 was 1.14 (95% confidence interval [CI]: 1.03–1.25) and that per 215.8 ppb of CO was 1.20 (95% CI: 1.03–1.40) in all infants. In females, an increase in NO2 and CO levels was associated with a higher risk of SIDS in low-birthweight and preterm infants. The OR per 15.7 ppb increment in NO2 was highest among preterm infants, with a value of 5.12 (95% CI: 1.27–20.63), and low-birthweight individuals, with a value of 4.11 (95% CI: 1.74–9.72), at a moving average of 0 to 3 days. In males, however, no significant association was found. In the present study, exposure to air pollution was associated with an increased risk of SIDS. This association was more evident in susceptible infants with a low-birthweight or in cases of preterm birth.


PEDIATRICS ◽  
1996 ◽  
Vol 98 (4) ◽  
pp. 796-797
Author(s):  
Steven A. Koehler

This letter is in response to the article by Knöbel et al entitled, "Sudden Infant Death Syndrome in Relation to Weather and Optimetrically Measured Air Pollution in Taiwan" in the December issue of Pediatrics (1995;96:1106-1110). The findings of this article are brought into question because the article suffers from several serious methodological and epidemiologic problems that greatly affect the validity of the results. Before proceeding, and because none was provided, a clear definition of sudden infant death syndrome (SIDS) needs to be given.


Sign in / Sign up

Export Citation Format

Share Document