Generation of Reactive Oxygen Species from Hinokitiol under Near-UV Irradiation

2003 ◽  
Vol 67 (9) ◽  
pp. 1996-1998 ◽  
Author(s):  
Hitoshi SHIBATA ◽  
Takayuki NAGAMINE ◽  
Yong WANG ◽  
Takahiro ISHIKAWA ◽  
Yoshihiro SAWA
Keyword(s):  
Reactive Oxygen Species ◽  
Uv Irradiation ◽  
Reactive Oxygen ◽  
Oxygen Species

scholarly journals Electrophilic Chemicals but not UV Irradiation or Reactive Oxygen Species Activate Nrf2 in Keratinocytes In Vitro and In Vivo

2007 ◽  
Vol 127 (3) ◽  
pp. 646-653 ◽  
Author(s):  
Moritz Durchdewald ◽  
Tobias A. Beyer ◽  
Delinda A. Johnson ◽  
Jeffrey A. Johnson ◽  
Sabine Werner ◽  
...  
Keyword(s):  
Reactive Oxygen Species ◽  
Uv Irradiation ◽  
Reactive Oxygen ◽  
Oxygen Species

Evaluation of carotenoids and reactive oxygen species in human skin after UV irradiation: a critical comparison betweenin vivoandex vivoinvestigations

2015 ◽  
Vol 24 (3) ◽  
pp. 194-197 ◽  
Author(s):  
Martina C. Meinke ◽  
Robert Müller ◽  
Anne Bechtel ◽  
Stefan F. Haag ◽  
Maxim E. Darvin ◽  
...  
Keyword(s):  
Reactive Oxygen Species ◽  
Human Skin ◽  
Uv Irradiation ◽  
Reactive Oxygen ◽  
Oxygen Species ◽  
Critical Comparison

Melatonin suppresses reactive oxygen species induced by UV irradiation in leukocytes

2004 ◽  
Vol 37 (2) ◽  
pp. 107-112 ◽  
Author(s):  
Tobias W. Fischer ◽  
Georg Scholz ◽  
Brunhilde Knoll ◽  
Uta-Christina Hipler ◽  
Peter Elsner
Keyword(s):  
Reactive Oxygen Species ◽  
Uv Irradiation ◽  
Reactive Oxygen ◽  
Oxygen Species

Mitochondria as a source of reactive oxygen species

2009 ◽  
pp. c3 ◽  
Author(s):  
Helena M. Cochemé ◽  
Michael P. Murphy
Keyword(s):  
Reactive Oxygen Species ◽  
Reactive Oxygen ◽  
Oxygen Species

Clinical aspects of reactive oxygen and nitrogen species

2004 ◽  
Vol 71 ◽  
pp. 121-133 ◽  
Author(s):  
Ascan Warnholtz ◽  
Maria Wendt ◽  
Michael August ◽  
Thomas Münzel
Keyword(s):  
Oxidative Stress ◽  
Reactive Oxygen Species ◽  
Risk Factor ◽  
Endothelial Dysfunction ◽  
Vascular Tissue ◽  
Rapid Development ◽  
Reactive Oxygen ◽  
Clinical Aspects ◽  
No Production ◽  
Oxygen Species

Endothelial dysfunction in the setting of cardiovascular risk factors, such as hypercholesterolaemia, hypertension, diabetes mellitus and chronic smoking, as well as in the setting of heart failure, has been shown to be at least partly dependent on the production of reactive oxygen species in endothelial and/or smooth muscle cells and the adventitia, and the subsequent decrease in vascular bioavailability of NO. Superoxide-producing enzymes involved in increased oxidative stress within vascular tissue include NAD(P)H-oxidase, xanthine oxidase and endothelial nitric oxide synthase in an uncoupled state. Recent studies indicate that endothelial dysfunction of peripheral and coronary resistance and conductance vessels represents a strong and independent risk factor for future cardiovascular events. Ways to reduce endothelial dysfunction include risk-factor modification and treatment with substances that have been shown to reduce oxidative stress and, simultaneously, to stimulate endothelial NO production, such as inhibitors of angiotensin-converting enzyme or the statins. In contrast, in conditions where increased production of reactive oxygen species, such as superoxide, in vascular tissue is established, treatment with NO, e.g. via administration of nitroglycerin, results in a rapid development of endothelial dysfunction, which may worsen the prognosis in patients with established coronary artery disease.

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