scholarly journals Functional and geometrical interference and interdependency between the right and left ventricle in cor pulmonale: An experimental study on simultaneous measurement of biventricular geometry of acute right ventricular pressure overload.

1989 ◽  
Vol 53 (10) ◽  
pp. 1237-1244 ◽  
Author(s):  
HIROHISA YAMASHITA ◽  
SOKICHI ONODERA ◽  
TETSURO IMAMOTO ◽  
ATSUSHI OBARA ◽  
SATOSHI TANAZAWA ◽  
...  
Keyword(s):  
Experimental Study ◽  
Left Ventricle ◽  
Right Ventricular ◽  
Simultaneous Measurement ◽  
Pressure Overload ◽  
Cor Pulmonale ◽  
Ventricular Pressure ◽  
The Right

scholarly journals Epigenetics, inflammation and metabolism in right heart failure associated with pulmonary hypertension

2017 ◽  
Vol 7 (3) ◽  
pp. 572-587 ◽  
Author(s):  
Nolwenn Samson ◽  
Roxane Paulin
Keyword(s):  
Left Ventricle ◽  
Right Ventricle ◽  
Right Ventricular ◽  
Pressure Overload ◽  
Right Heart Failure ◽  
Left Ventricular ◽  
Ventricular Failure ◽  
Pulmonary Arterial ◽  
Ventricle Hypertrophy ◽  
The Right

Right ventricular failure (RVF) is the most important prognostic factor for both morbidity and mortality in pulmonary arterial hypertension (PAH), but also occurs in numerous other common diseases and conditions, including left ventricle dysfunction. RVF remains understudied compared with left ventricular failure (LVF). However, right and left ventricles have many differences at the morphological level or the embryologic origin, and respond differently to pressure overload. Therefore, knowledge from the left ventricle cannot be extrapolated to the right ventricle. Few studies have focused on the right ventricle and have permitted to increase our knowledge on the right ventricular-specific mechanisms driving decompensation. Here we review basic principles such as mechanisms accounting for right ventricle hypertrophy, dysfunction, and transition toward failure, with a focus on epigenetics, inflammatory, and metabolic processes.

scholarly journals The left ventricle undergoes biomechanical and gene expression changes in response to increased right ventricular pressure overload

2020 ◽  
Vol 8 (9) ◽  
Author(s):  
Vitaly O. Kheyfets ◽  
Melanie J. Dufva ◽  
Mario Boehm ◽  
Xuefeit Tian ◽  
Xulei Qin ◽  
...  
Keyword(s):  
Gene Expression ◽  
Left Ventricle ◽  
Right Ventricular ◽  
Pressure Overload ◽  
Ventricular Pressure

Characterisation of the Right Ventricular Pressure-Volume Relationship: Comparison with the Left Ventricle

1986 ◽  
Vol 71 (s15) ◽  
pp. 55P-55P
Author(s):  
A.N. Redington ◽  
H. Gray ◽  
R.D. Dawson ◽  
M.E. Hodson ◽  
P.J. Oldershaw
Keyword(s):  
Left Ventricle ◽  
Right Ventricular ◽  
Ventricular Pressure ◽  
Volume Relationship ◽  
Pressure Volume Relationship ◽  
The Right

scholarly journals Changes in collagen and elastin in rabbit right-ventricular pressure overload

1989 ◽  
Vol 263 (3) ◽  
pp. 709-713 ◽  
Author(s):  
R B Low ◽  
W S Stirewalt ◽  
P Hultgren ◽  
E S Low ◽  
B Starcher
Keyword(s):  
Connective Tissue ◽  
Right Ventricular ◽  
Mechanical Performance ◽  
Age Groups ◽  
Pressure Overload ◽  
Ventricular Pressure ◽  
Type I ◽  
Collagen Concentration ◽  
Ventricular Tissue ◽  
The Right

Collagen content, the ratio of collagen types I and III and elastin content were measured in 5-6- and 10-12-week-old rabbits with and without right-ventricular pressure overload. Significant and equivalent hypertrophy occurred in both age groups. A 2-day pressure overload caused a fall in collagen concentration below control levels in right-ventricular tissue from the older animals, but no change in the younger ones. A 2-week pressure overload in the older animals resulted in a rise in collagen concentration, a decreased ratio of type III to type I plus III [III/(I + III)] collagens, a fall in desmosine concentration and a fall in the desmosine/hydroxyproline ratio in the right ventricle. None of these changes occurred in the younger age group. We hypothesize that the changes in connective-tissue proteins after overload in the older group may contribute to previously observed changes in mechanical performance. The divergent connective-tissue responses in the two groups suggest the importance of age in determining outcome, as well as the possibility of separate regulatory mechanisms for contractile and for architectural elements of the heart.

Regional Left Ventricular Systolic Function in Relation to the Cavity Geometry in Patients With Chronic Right Ventricular Pressure Overload

Circulation ◽  
1995 ◽  
Vol 91 (9) ◽  
pp. 2359-2370 ◽  
Author(s):  
Sheng-Jing Dong ◽  
Adrian P. Crawley ◽  
John H. MacGregor ◽  
Yael Fisher Petrank ◽  
Dale W. Bergman ◽  
...  
Keyword(s):  
Right Ventricular ◽  
Systolic Function ◽  
Pressure Overload ◽  
Left Ventricular Systolic Function ◽  
Left Ventricular ◽  
Ventricular Pressure ◽  
Ventricular Systolic Function

Decreased Myocyte Tension Development and Calcium Responsiveness in Rat Right Ventricular Pressure Overload

Circulation ◽  
1997 ◽  
Vol 95 (9) ◽  
pp. 2312-2317 ◽  
Author(s):  
Dongsheng Fan ◽  
Thomas Wannenburg ◽  
Pieter P. de Tombe
Keyword(s):  
Right Ventricular ◽  
Pressure Overload ◽  
Ventricular Pressure ◽  
Tension Development

scholarly journals Biventricular Remodeling in Murine Models of Right Ventricular Pressure Overload

PLoS ONE ◽  
2013 ◽  
Vol 8 (7) ◽  
pp. e70802 ◽  
Author(s):  
Navin K. Kapur ◽  
Vikram Paruchuri ◽  
Mark J. Aronovitz ◽  
Xiaoying Qiao ◽  
Emily E. Mackey ◽  
...  
Keyword(s):  
Right Ventricular ◽  
Pressure Overload ◽  
Ventricular Pressure ◽  
Murine Models
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