Fe(III), Cu(II), V(V)/TiO2 for Hydroxylation of Benzene to Phenol with Hydrogen Peroxide at Room Temperature

2007 ◽  
Vol 40 (5) ◽  
pp. 415-421 ◽  
Author(s):  
Garun Tanarungsun ◽  
Worapon Kiatkittipong ◽  
Suttichai Assabumrungrat ◽  
Hiroshi Yamada ◽  
Tomohiko Tagawa ◽  
...  
Keyword(s):  
Hydrogen Peroxide ◽  
Room Temperature ◽  
Hydroxylation Of Benzene

HYDROXYLATION OF BENZENE BY HYDROGEN PEROXIDE CATALYZED BY AN Fe3+-CATECHOL CATALYST SUPPORTED ON SILICA GEL

1982 ◽  
Vol 11 (5) ◽  
pp. 651-652 ◽  
Author(s):  
Seizo Tamagaki ◽  
Kazuhiko Hotta ◽  
Waichiro Tagaki
Keyword(s):  
Hydrogen Peroxide ◽  
Silica Gel ◽  
Hydroxylation Of Benzene

scholarly journals HYDROXYLATION OF BENZENE WITH HYDROGEN PEROXIDE BY THE USE OF HYDROPHOBIC CATECHOLS AND Fe3+COMPLEXES AS THE CATALYST

1981 ◽  
Vol 10 (6) ◽  
pp. 789-790 ◽  
Author(s):  
Kazuhiko Hotta ◽  
Seizo Tamagaki ◽  
Yusuke Suzuki ◽  
Waichiro Tagaki
Keyword(s):  
Hydrogen Peroxide ◽  
Hydroxylation Of Benzene

scholarly journals A Mn(iii) polyoxotungstate in the oxidation of organosulfur compounds by H2O2 at room temperature: an environmentally safe catalytic approach

2016 ◽  
Vol 6 (9) ◽  
pp. 3271-3278 ◽  
Author(s):  
Tiago A. G. Duarte ◽  
Sónia M. G. Pires ◽  
Isabel C. M. S. Santos ◽  
Mário M. Q. Simões ◽  
M. Graça P. M. S. Neves ◽  
...  
Keyword(s):  
Hydrogen Peroxide ◽  
Room Temperature ◽  
Organosulfur Compounds ◽  
Keggin Type ◽  
Environmentally Safe

A manganese monosubstituted Keggin-type polyoxometalate was used as a catalyst in the oxidation of recalcitrant organosulfur compounds by hydrogen peroxide at room temperature.

A Promising Coupled Process of Pd/γ-Al2O3–NH4VO3 Catalyzing the Hydroxylation of Benzene with Hydrogen Peroxide Produced In Situ by an Anthraquinone Redox Route

2007 ◽  
Vol 118 (3-4) ◽  
pp. 270-274 ◽  
Author(s):  
Guosheng Peng ◽  
Zaihui Fu ◽  
Dulin Yin ◽  
Sheng Zhong ◽  
Yan Yang ◽  
...  
Keyword(s):  
Hydrogen Peroxide ◽  
Hydroxylation Of Benzene ◽  
Coupled Process

scholarly journals Studies on the Preservation of Raw Cow’s Milk by Chemical Method

1970 ◽  
Vol 42 (3) ◽  
pp. 317-326 ◽  
Author(s):  
F Rokhsana ◽  
UK Das ◽  
R Yeasmin ◽  
A Nahar ◽  
S Parveen
Keyword(s):  
Hydrogen Peroxide ◽  
Chemical Method ◽  
Room Temperature ◽  
Storage Period ◽  
Total Coliform ◽  
Human Consumption ◽  
Milk Products ◽  
E Coli ◽  
Keeping Quality ◽  
Control Milk

Studies carried out to develop a technique for the preservation of cow's milk in raw condition using hydrogen peroxide (H2O2) as a preservative. Fresh cow’s milk was collected and experiments were conducted by four treatments in order to achieve the optimum condition of storage. The treatments were with various concentration of H2O2 starting from 0.05 %, 0.1 %, 0.2 %, 0.3 %, 0.4 %, & 0.5 %. Treated milk with 0.05 % concentration of H2O2 had storage period of 20 days compared to that of the control one (5 days only) in refrigerated temperature (±8°C). On the other hand hydrogen peroxide treated milk (0.05 %) had a storage period of 8 hours at room temperature (±28°C). Results also showed that the higher concentration of H2O2 had no effect on storage period than that of control. Milk products like kheer and halawa prepared by treated milk and stored for 20 days showed almost nil growth of total coliform and E. coli which means that food products prepared from hydrogen peroxide treated milk is safe for human consumption. Key words: Raw, Storage, Hydrogen peroxide, Preservative, keeping quality, Pasteurization, deteriorated, MPN. Bangladesh J. Sci. Ind. Res. 42(3), 317-326, 2007

scholarly journals Formation of Water-Soluble Fullerenes [C60,C70] under Ultrasonication and Antioxidant Effect

2017 ◽  
Vol 5 (1) ◽  
pp. 61
Author(s):  
Weon-Bae Ko ◽  
Hong-Seok Jeong ◽  
Sung-Ho Hwang
Keyword(s):  
Hydrogen Peroxide ◽  
Room Temperature ◽  
Antioxidant Effect ◽  
Water Soluble ◽  
Maldi Tof Ms ◽  
Pc 12 Cells ◽  
Effect Of Water ◽  
Maldi Tof ◽  
Line Following ◽  
Tof Ms

<p>The water-soluble fullerenes [C<sub>60</sub>, C<sub>70</sub>] are prepared with fullerenes [C<sub>60</sub>, C<sub>70</sub>] and a mixture of oxidants (v/v) at the ratio of 3:1 under ultrasonic condition at room temperature. The MALDI-TOF MS confirmed that the water-soluble compounds were C<sub>60</sub> and C<sub>70</sub>. The antioxidant effect of water-soluble fullerenes [C<sub>60</sub>, C<sub>70</sub>] in the PC 12 cells (Rat pheochromocytoma) line following exposure to hydrogen peroxide (H<sub>2</sub>O<sub>2</sub>) was investigated.</p>

Abstract 355: Effect of Poloxamer 188 on Rat Brain Isolated Mitochondria After Exposure to Hydrogen Peroxide

Circulation ◽  
2019 ◽  
Vol 140 (Suppl_2) ◽  
Author(s):  
Johannes A Pille ◽  
Michele M Salzman ◽  
Anna A Sonju ◽  
Felicia P Lotze ◽  
Josephine E Hees ◽  
...  
Keyword(s):  
Hydrogen Peroxide ◽  
Oxygen Consumption ◽  
Mitochondrial Function ◽  
Complex I ◽  
Room Temperature ◽  
In Vitro Model ◽  
Atp Synthesis ◽  
Complex Ii ◽  
Vitro Model

Introduction: In a pig model of myocardial infarction (MI), intracoronary delivered Poloxamer (P) 188 significantly reduces ischemia/reperfusion (IR) injury when given immediately upon reperfusion, with improved mitochondrial function as a predominant effect. As mitochondria are heavily damaged during IR, a direct effect of P188 on mitochondria may lead to better therapy options during reperfusion. To show not only a similar reduction of IR injury by P188 in the brain, but also a direct P188 effect on mitochondria, we established an in-vitro model of IR that consists of damaging isolated rat brain mitochondria with hydrogen peroxide (H 2 O 2 ), one component of ischemia, then applying P188, and analyzing mitochondrial function. Methods: Male Sprague-Dawley rat brains were removed, and the mitochondria isolated by differential centrifugation and Percoll gradients, then kept on ice to slow their bioenergetics prior to any experimental treatments. Mitochondria were exposed to 200 μM H 2 O 2 for 10 min at room temperature with slight agitation; controls received no H 2 O 2 . Samples were then diluted ½ with buffer ± P188 (250 μM after dilution) to simulate reperfusion and treatment, and kept at room temperature for 10 further minutes. ATP synthesis was measured in a luminometer using a luciferase enzymatic assay. Oxygen consumption was measured by closed cell respirometry with an oxygen meter. In both assays, Complex I and Complex II were examined; Complex I substrates glutamate and malate, Complex II substrate succinate plus the Complex I inhibitor rotenone. Statistics: Data are expressed as mean ± SEM. One-Way ANOVA, SNK-Test; Kruskal-Wallis-Test; α=0.05, * vs control. Results: In both Complex I and II, mitochondrial function was significantly impaired by H 2 O 2 , with ATP synthesis affected more at Complex I and oxygen consumption affected more at Complex II. Addition of P188 did not provide any significant improvement in mitochondrial function. Conclusions: Although P188 significantly reduced IR injury when given during reperfusion in a pig model of MI, it does not appear to provide direct protection to mitochondria in this in-vitro model. Whether the exposure to H 2 O 2 causes the appropriate injury for P188 to become effective remains to be elucidated.

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