scholarly journals Isotopic Exchange between Hydrogen Sulfide and Deuterium on the Surface of Cuprous Sulfide

1970 ◽  
Vol 43 (1) ◽  
pp. 23-30 ◽  
Author(s):  
Kazuo Fueki ◽  
Hideaki Inaba ◽  
Takashi Mukaibo
Keyword(s):  
Hydrogen Sulfide ◽  
Isotopic Exchange ◽  
Cuprous Sulfide

Structure and Reactions of Cuprous Thiobenzoate

1972 ◽  
Vol 50 (24) ◽  
pp. 4056-4058 ◽  
Author(s):  
Aldo Ferretti
Keyword(s):  
Hydrogen Sulfide ◽  
Benzoic Acid ◽  
Ethyl Benzoate ◽  
Prolonged Heating ◽  
Pyridine Solution ◽  
Excellent Yield ◽  
Cuprous Sulfide ◽  
Thiobenzoic Acid

Cuprous thiobenzoate is capable of displacing I− from aliphatic and activated aromatic iodides, in alcoholic or alcohol–pyridine solution, with formation of thiolbenzoates in excellent yield. The reaction fails with other halogenated substrates. In these cases, the products of prolonged heating of the reaction mixture are ethyl benzoate, dibenzoyl disulfide, benzoic acid, thiobenzoic acid, hydrogen sulfide, and cuprous sulfide.

Dissociation of dihydrogen and hydrogen sulfide over a sulfided NiMo-alumina catalyst as evidenced by D2S-H2 isotopic exchange

1995 ◽  
Vol 34 (3-4) ◽  
pp. 375-378 ◽  
Author(s):  
P. d'Araujo ◽  
C. Thomas ◽  
L. Vivier ◽  
D. Duprez ◽  
G. P�rot ◽  
...  
Keyword(s):  
Hydrogen Sulfide ◽  
Isotopic Exchange ◽  
Alumina Catalyst

Deuterium Tracer Studies on Hydrotreating Catalysts—Isotopic Exchange between Hydrogen and Hydrogen Sulfide on Sulfided NiMo/Al2O3

1997 ◽  
Vol 167 (1) ◽  
pp. 1-11 ◽  
Author(s):  
C. Thomas ◽  
L. Vivier ◽  
J.L. Lemberton ◽  
S. Kasztelan ◽  
G. Pérot
Keyword(s):  
Hydrogen Sulfide ◽  
Isotopic Exchange ◽  
Tracer Studies ◽  
Hydrotreating Catalysts

Endogenous hydrogen sulfide sulfhydrates IKKβ at cysteine 179 to control pulmonary artery endothelial cell inflammation

2019 ◽  
Vol 133 (20) ◽  
pp. 2045-2059 ◽  
Author(s):  
Da Zhang ◽  
Xiuli Wang ◽  
Siyao Chen ◽  
Selena Chen ◽  
Wen Yu ◽  
...  
Keyword(s):  
Hydrogen Sulfide ◽  
Endothelial Cell ◽  
Pulmonary Artery ◽  
Cell Model ◽  
Site Directed Mutagenesis ◽  
Critical Event ◽  
Hypertensive Rats ◽  
Pulmonary Artery Endothelial Cell

Abstract Background: Pulmonary artery endothelial cell (PAEC) inflammation is a critical event in the development of pulmonary arterial hypertension (PAH). However, the pathogenesis of PAEC inflammation remains unclear. Methods: Purified recombinant human inhibitor of κB kinase subunit β (IKKβ) protein, human PAECs and monocrotaline-induced pulmonary hypertensive rats were employed in the study. Site-directed mutagenesis, gene knockdown or overexpression were conducted to manipulate the expression or activity of a target protein. Results: We showed that hydrogen sulfide (H2S) inhibited IKKβ activation in the cell model of human PAEC inflammation induced by monocrotaline pyrrole-stimulation or knockdown of cystathionine γ-lyase (CSE), an H2S generating enzyme. Mechanistically, H2S was proved to inhibit IKKβ activity directly via sulfhydrating IKKβ at cysteinyl residue 179 (C179) in purified recombinant IKKβ protein in vitro, whereas thiol reductant dithiothreitol (DTT) reversed H2S-induced IKKβ inactivation. Furthermore, to demonstrate the significance of IKKβ sulfhydration by H2S in the development of PAEC inflammation, we mutated C179 to serine (C179S) in IKKβ. In purified IKKβ protein, C179S mutation of IKKβ abolished H2S-induced IKKβ sulfhydration and the subsequent IKKβ inactivation. In human PAECs, C179S mutation of IKKβ blocked H2S-inhibited IKKβ activation and PAEC inflammatory response. In pulmonary hypertensive rats, C179S mutation of IKKβ abolished the inhibitory effect of H2S on IKKβ activation and pulmonary vascular inflammation and remodeling. Conclusion: Collectively, our in vivo and in vitro findings demonstrated, for the first time, that endogenous H2S directly inactivated IKKβ via sulfhydrating IKKβ at Cys179 to inhibit nuclear factor-κB (NF-κB) pathway activation and thereby control PAEC inflammation in PAH.

Limnological characteristics, community metabolism and management strategies of a coastal sinkhole in Cuba (Cenote Jennifer)

2020 ◽  
Vol 56 ◽  
pp. 24
Author(s):  
Roberto González-De Zayas ◽  
Liosban Lantigua Ponce de León ◽  
Liezel Guerra Rodríguez ◽  
Felipe Matos Pupo ◽  
Leslie Hernández-Fernández
Keyword(s):  
Hydrogen Sulfide ◽  
Primary Productivity ◽  
Major Ions ◽  
Saline Water ◽  
Management Strategies ◽  
Tourist Destination ◽  
Community Metabolism ◽  
Morphological Studies ◽  
Made In ◽  
Limnological Parameters

The Cenote Jennifer is an important and unique aquatic sinkhole in Cayo Coco (Jardines del Rey Tourist Destination) that has brackish to saline water. Two samplings were made in 1998 and 2009, and 4 metabolism community experiments in 2009. Some limnological parameters were measured in both samplings (temperature, salinity, pH, dissolved oxygen major ions, hydrogen sulfide, nutrients and others). Community metabolism was measured through incubated oxygen concentration in clear and dark oxygen bottles. Results showed that the sinkhole limnology depends on rainfall and light incidence year, with some stratification episodes, due to halocline or oxycline presence, rather than thermocline. The sinkhole water was oligotrophic (total nitrogen of 41.5 ± 22.2 μmol l−1 and total phosphorus of 0.3 ± 0.2 μmol l−1) and with low productivity (gross primary productivity of 63.0 mg C m−2 d−1). Anoxia and hypoxia were present at the bottom with higher levels of hydrogen sulfide, lower pH and restricted influence of the adjacent sea (2 km away). To protect the Cenote Jennifer, tourist exploitation should be avoided and more resources to ecological and morphological studies should be allocated, and eventually use this aquatic system only for specialized diving. For conservation purposes, illegal garbage disposal in the surrounding forest should end.

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