The Nerves and Muscles of Medusae

1955 ◽  
Vol 32 (4) ◽  
pp. 642-648 ◽  
Author(s):  
G. A. HORRIDGE

1. The co-ordination of the swimming movement (beat) and feeding response in Aequorea forskalea has been studied with particular attention to the pathways taken by the excitation. 2. The rapid through-conducting system which co-ordinates the beat is sharply distinguished physiologically from the radial system which co-ordinates feeding. 3. The spontaneous origin of the beat and its rapid marginal conduction are both inhibited while the radial muscle is contracting. 4. Inhibition of this type has been observed in other genera of Hydromedusae

Heart ◽  
1976 ◽  
Vol 38 (2) ◽  
pp. 204-206 ◽  
Author(s):  
R Gray ◽  
V S Kaushik ◽  
W J Mandel

2001 ◽  
Vol 122 (2) ◽  
pp. 193-199 ◽  
Author(s):  
Mitchell F. Roitman ◽  
Gertjan van Dijk ◽  
Todd E. Thiele ◽  
Ilene L. Bernstein
Keyword(s):  

1991 ◽  
Vol 69 (7) ◽  
pp. 1552-1556 ◽  
Author(s):  
M. Muntanola-Cvetković ◽  
Jelena Vukojević ◽  
M. Mihaljčević

The systemic nature of the disease of sunflower plants caused by Diaporthe helianthi, the leaf–petiole–stem route of the host invasion by the fungus, and the plant tissues that were successively affected were demonstrated through histological studies. After penetration into the host, the infection hyphae invade the intercellular spaces and terminal veinlets of the lamina and spread toward larger branches of the conducting system, the midrib, and the petiole. Xylem elements are invaded but are affected less by the fungus attack than the phloem and the parenchyma tissues, which disintegrate completely. Hyphae spread through the leaf axil to the tissues of the stem cortex, where pycnidia of the Phomopsis anamorph are initiated from internal masses of mycelium. Key words: systemic plant diseases, sunflower diseases, Diaporthe helianthi, Phomopsis helianthi.


1965 ◽  
Vol 5 (02) ◽  
pp. 131-140 ◽  
Author(s):  
K.P. Fournier

Abstract This report describes work on the problem of predicting oil recovery from a reservoir into which water is injected at a temperature higher than the reservoir temperature, taking into account effects of viscosity-ratio reduction, heat loss and thermal expansion. It includes the derivation of the equations involved, the finite difference equations used to solve the partial differential equation which models the system, and the results obtained using the IBM 1620 and 7090–1401 computers. Figures and tables show present results of this study of recovery as a function of reservoir thickness and injection rate. For a possible reservoir hot water flood in which 1,000 BWPD at 250F are injected, an additional 5 per cent recovery of oil in place in a swept 1,000-ft-radius reservoir is predicted after injection of one pore volume of water. INTRODUCTION The problem of predicting oil recovery from the injection of hot water has been discussed by several researchers.1–6,19 In no case has the problem of predicting heat losses been rigorously incorporated into the recovery and displacement calculation problem. Willman et al. describe an approximate method of such treatment.1 The calculation of heat losses in a reservoir and the corresponding temperature distribution while injecting a hot fluid has been attempted by several authors.7,8 In this report a method is presented to numerically predict the oil displacement by hot water in a radial system, taking into account the heat losses to adjacent strata, changes in viscosity ratio with temperature and the thermal-expansion effect for both oil and water. DERIVATION OF BASIC EQUATIONS We start with the familiar Buckley-Leverett9 equation for a radial system:*Equation 1 This can be written in the formEquation 2 This is sometimes referred to as the Lagrangian form of the displacement equation.


2016 ◽  
Vol 18 (3) ◽  
pp. 221-227 ◽  
Author(s):  
Matthew Charlton ◽  
Mark Sims ◽  
Tim Coats ◽  
Jonathan P Thompson

The microcirculation describes the smallest elements of the cardiovascular conducting system and is pivotal in the maintenance of homeostasis. Microcirculatory dysfunction is present early in the pathophysiology of sepsis, with the extent of microcirculatory derangement relating to disease severity and prognosis in ICU patients. However, at present microcirculatory function is not routinely monitored at the bedside. This article describes the pathophysiology of microcirculatory derangements in sepsis, methods of its measurement and evidence to support their clinical use.


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