scholarly journals How does the 'ancient' asexual Philodina roseola (Rotifera: Bdelloidea) handle potential UVB-induced mutations?

2013 ◽  
Vol 216 (16) ◽  
pp. 3090-3095 ◽  
Author(s):  
C. Fischer ◽  
W. H. Ahlrichs ◽  
A. G. J. Buma ◽  
W. H. van de Poll ◽  
O. R. P. Bininda-Emonds
Keyword(s):  
Induced Mutations ◽  
Ancient Asexual ◽  
Philodina Roseola ◽  
Rotifera Bdelloidea

scholarly journals TAXAS DE FILTRAÇÃO E INGESTÃO DE UMA MICROALGA POR Philodina roseola (Rotifera: Bdelloidea)

2016 ◽  
Vol 21 (2) ◽  
pp. 325-333
Author(s):  
Raquel Aparecida Moreira ◽  
Adrislaine Da Silva Mansano ◽  
Odete Rocha
Keyword(s):  
Philodina Roseola ◽  
Rotifera Bdelloidea

scholarly journals First data on secondary production of Philodina roseola (Rotifera, Bdelloidea) grown in laboratory

Limnetica ◽  
2017 ◽  
pp. 55-65
Author(s):  
Aparecida Moreira, Raquel ◽  
Da Silva Mansano, Adrislaine ◽  
Rocha, Odete
Keyword(s):  
Secondary Production ◽  
Philodina Roseola ◽  
Rotifera Bdelloidea

scholarly journals Correction to: Mechanisms of UV-induced mutations and skin cancer

2021 ◽  
Author(s):  
Gerd P. Pfeifer
Keyword(s):  
Skin Cancer ◽  
Induced Mutations

A correction to this paper has been published: https://doi.org/10.1007/s42764-021-00037-y

Interaction Between Mutations in the suppressor of Hairy wing and modifier of mdg4 Genes of Drosophila melunogaster Affecting the Phenotype of gypsy-Induced Mutations

Genetics ◽  
1996 ◽  
Vol 142 (2) ◽  
pp. 425-436 ◽  
Author(s):  
Pavel Georgiev ◽  
Marina Kozycina
Keyword(s):  
Mutagenic Effect ◽  
Induced Mutations ◽  
Binding Region ◽  
Direct Inhibition ◽  
Amino Terminal ◽  
Acidic Domain ◽  
Complete Inactivation ◽  
Insulation Effect ◽  
Gypsy Retrotransposon ◽  
Carboxy Terminal

Abstract The suppressor of Hairy-wing [su(Hw)] protein mediates the mutagenic effect of the gypsy retrotransposon by repressing the function of transcriptional enhancers located distally from the promoter with respect to the position of the su(Hw)-binding region. Mutations in a second gene, modifier of mdg4, also affect the gypsy-induced phenotype. Two major effects of the mod(mdg4)lul mutation can be distinguished: the interference with insulation by the su(Hw)-binding region and direct inhibition of gene expression that is not dependent on the su(Hw)-binding region position. The mod(mdg4)lul mutation partially suppresses ct6, scD1 and Hw1 mutations, possibly by interfering with the insulation effect of the su(Hw)-binding region. An example of the second effect of mod(mdg4)lul is a complete inactivation of yellow expression in combination with the y  2 allele. Phenotypic analyses of flies with combinations of mod(mdg4)lul and different su(Hw) mutations, or with constructions carrying deletions of the acidic domains of the su(Hw) protein, suggest that the carboxy-terminal acidic domain is important for direct inhibition of yellow transcription in bristles, while the amino-terminal acidic domain is more essential for insulation.

scholarly journals MOLECULAR GENETIC ANALYSIS OF THE DILUTE-SHORT EAR (d-se) REGION OF THE MOUSE

Genetics ◽  
1986 ◽  
Vol 112 (2) ◽  
pp. 321-342
Author(s):  
Eugene M Rinchik ◽  
Liane B Russell ◽  
Neal G Copeland ◽  
Nancy A Jenkins
Keyword(s):  
Physical Map ◽  
Leukemia Virus ◽  
Molecular Genetic ◽  
Break Point ◽  
Virus Genome ◽  
Molecular Genetic Analysis ◽  
Chromosome 9 ◽  
Genetic Complementation ◽  
Induced Mutations ◽  
Radiation Induced

ABSTRACT Genes of the dilute-short ear (d-se) region of mouse chromosome 9 comprise an array of loci important to the normal development of the animal. Over 200 spontaneous, chemically induced and radiation-induced mutations at these loci have been identified, making it one of the most genetically well-characterized regions of the mouse. Molecular analysis of this region has recently become feasible by the identification of a dilute mutation that was induced by integration of an ecotropic murine leukemia virus genome. Several unique sequence cellular DNA probes flanking this provirus have now been identified and used to investigate the organization of wild-type chromosomes and chromosomes with radiation-induced d-se region mutations. As expected, several of these mutations are associated with deletions, and, in general, the molecular and genetic complementation maps of these mutants are concordant. Furthermore, a deletion break-point fusion fragment has been identified and has been used to orient the physical map of the d-se region with respect to the genetic complementation map. These experiments provide important initial steps for analyzing this developmentally important region at the molecular level, as well as for studying in detail how a diverse group of mutagens acts on the mammalian germline.

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