scholarly journals Induced synthesis of P450 aromatase and 17 -estradiol by D-aspartate in frog brain

2012 ◽  
Vol 215 (20) ◽  
pp. 3559-3565 ◽  
Author(s):  
L. Burrone ◽  
A. Santillo ◽  
C. Pinelli ◽  
G. C. Baccari ◽  
M. M. Di Fiore
Keyword(s):  
1997 ◽  
Vol 16 (6) ◽  
pp. 769-777 ◽  
Author(s):  
INHO CHOI ◽  
DERYL L. TROYER ◽  
DEAN L. CORNWELL ◽  
KATHY R. KIRBY-DOBBELS ◽  
WERNER R. COLLANTE ◽  
...  

2006 ◽  
Vol 26 (25) ◽  
pp. 6749-6760 ◽  
Author(s):  
J.-L. Do-Rego ◽  
S. Acharjee ◽  
J. Y. Seong ◽  
L. Galas ◽  
D. Alexandre ◽  
...  
Keyword(s):  

2006 ◽  
Vol 99 (4-5) ◽  
pp. 189-196 ◽  
Author(s):  
Marie-Noëlle Dieudonné ◽  
Anes Sammari ◽  
Esther Dos Santos ◽  
Marie-Christine Leneveu ◽  
Yves Giudicelli ◽  
...  

2006 ◽  
Vol 85 (5) ◽  
pp. 1516-1518 ◽  
Author(s):  
Jean-François Heilier ◽  
Olivier Donnez ◽  
Valérie Van Kerckhove ◽  
Dominique Lison ◽  
Jacques Donnez

2004 ◽  
Vol 21 (4) ◽  
pp. 417-425 ◽  
Author(s):  
Yasuhisa Kobayashi ◽  
Tohru Kobayashi ◽  
Masaru Nakamura ◽  
Tomoki Sunobe ◽  
Craig E. Morrey ◽  
...  

2002 ◽  
Vol 65 (2) ◽  
pp. 798-801 ◽  
Author(s):  
Jaideep Moitra ◽  
Huseyin Avni Öktem ◽  
Anna Borsodi

1997 ◽  
Vol 200 (22) ◽  
pp. 2913-2917 ◽  
Author(s):  
P L Lutz ◽  
R Reiners

This study investigated the relationship between energy failure and neurotransmitter release in the frog (Rana pipiens) brain during 1-3 h of anoxia. Unlike truly anoxia-tolerant species, the frog does not defend its brain energy charge. When exposed to anoxia at 25 degrees C, there is an immediate fall in brain ATP levels, which reach approximately 20% of normoxic levels in approximately 60 min. The frog, nevertheless, survives another 1-2 h of anoxia. At 100 min of anoxia, there is an increase in extracellular adenosine concentration, probably originating from the increased intracellular adenosine concentration caused by the breakdown of intracellular ATP. Increases in the levels of extracellular glutamate and GABA do not occur until 1-2 h after ATP depletion. This response is quite unlike that recorded for other vertebrates, anoxia-tolerant or anoxia-intolerant, where energy failure quickly results in an uncontrolled and neurotoxic release of excitatory neurotransmitters. In the frog, the delay in excitotoxic neurotransmitter release may be one of the factors that allow a period of survival after energy failure. Clearly, energy failure by itself is not a fatal event in the frog brain.


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