scholarly journals Decreased precision contributes to the hypoxic thermoregulatory response in lizards

2008 ◽  
Vol 212 (1) ◽  
pp. 137-144 ◽  
Author(s):  
V. Cadena ◽  
G. J. Tattersall
Keyword(s):  
Thermoregulatory Response

Thermoregulatory Response Thresholds During Spinal Anesthesia

1993 ◽  
Vol 77 (4) ◽  
pp. 721???726 ◽  
Author(s):  
Andrea Kurz ◽  
Daniel I. Sessler ◽  
Marc Schroeder ◽  
Martin Kurz
Keyword(s):  
Spinal Anesthesia ◽  
Thermoregulatory Response ◽  
Response Thresholds

Influence of sex, athletic performance and age differences on the acute cardiovascular and thermoregulatory response to incremental test in endurance runners

2016 ◽  
Vol 31 (5) ◽  
pp. e123-e129 ◽  
Author(s):  
F. García-Pinillos ◽  
V. Serrano-Huete ◽  
J. Salas-Sánchez ◽  
R. Ortega-García ◽  
F.J. Gómez-Rodríguez ◽  
...  
Keyword(s):  
Age Differences ◽  
Athletic Performance ◽  
Thermoregulatory Response ◽  
Incremental Test ◽  
Endurance Runners

scholarly journals Thermoregulatory response of Frankliniella occidentalis (Pergande) (Thysanoptera: Thripidae) to infection by Beauveria bassiana, and its effect on survivorship and reproductive success

2019 ◽  
Vol 9 (1) ◽  
Author(s):  
Xiaochen Liu ◽  
Stuart R. Reitz ◽  
Zhongren Lei ◽  
Haihong Wang
Keyword(s):  
Biological Control ◽  
Beauveria Bassiana ◽  
Frankliniella Occidentalis ◽  
Behavioral Thermoregulation ◽  
Thermoregulatory Response ◽  
Defensive Strategy ◽  
Control Programs ◽  
Severe Reduction ◽  
Seeking Behavior ◽  
History Of

Abstract Behavioral thermoregulation is a defensive strategy employed by some insects to counter infections by parasites and pathogens. Most reported examples of this type of thermoregulatory response involve behavioral fevering. However depending upon the life history of a host-insect and that of the parasite or pathogen, the host may respond by cold-seeking behavior. Thermoregulation is not only ecologically important; it may affect the success of parasites and pathogens as biological control agents. We examined if Frankliniella occidentalis (Pergande) thermoregulates in response to infection by Beauveria bassiana, a fungal pathogen commonly used for biological control. Fungal-infected thrips preferentially moved to cooler areas (~12 °C) while healthy thrips sought out warmer temperatures (~24 °C). This cold-seeking behavior suppressed the growth of B. bassiana in infected thrips, and significantly improved survivorship of infected thrips. At 24 °C, males only survived up to 10 d after infection and females up to 20 d after infection, which was substantially poorer survivorship than that of corresponding healthy individuals. However, individuals of both sexes survived up to 48 d after infection at 12 °C, which was a much less severe reduction in survivorship compared with the effect of B. bassiana infection at 24 °C. The proportion of females among progeny from infected thrips at 12 °C was higher than at 24 °C. Therefore, cold-seeking behavior is beneficial to F. occidentalis when infected by B. bassiana, and its effects should be considered in the use of B. bassiana in biological control programs.

Centrally acting vasopressin contributes to endotoxin tolerance

1990 ◽  
Vol 258 (2) ◽  
pp. R443-R449 ◽  
Author(s):  
M. F. Wilkinson ◽  
N. W. Kasting
Keyword(s):  
Escherichia Coli ◽  
Receptor Antagonist ◽  
Arginine Vasopressin ◽  
Endotoxin Tolerance ◽  
Thermoregulatory Response ◽  
Endotoxin Challenge ◽  
E Coli ◽  
Intravenous Injections ◽  
Refractory State ◽  
V2 Antagonist

Repeated daily intravenous injections of bacterial endotoxin induce a refractory state to their usual pyrogenic effects. The neuropeptide arginine vasopressin (AVP) has been implicated in natural fever suppression and may be involved in the process of pyrogenic tolerance to intravenous endotoxin. This study was conducted to test this hypothesis. Tolerance was induced by two successive daily intravenous injections of Escherichia coli endotoxin (50 micrograms/kg) into conscious unrestrained rats. This tolerance was maintained, unaltered, after a third or fourth subsequent injection. However, bilateral administration of an AVP V1-receptor antagonist (0.43-4.3 nmol) into the ventral septal area (VSA) of the rat brain markedly enhanced the thermoregulatory response to a third or fourth endotoxin challenge compared with saline controls. The effect of the V1 antagonist was dose related. In contrast, an AVP V2 antagonist (0.43 nmol) bilaterally injected into the VSA did not affect the tolerant reaction to endotoxin. Furthermore, neither saline nor the V1 antagonist significantly affected core temperature when administered within the VSA without subsequent endotoxin. These results are consistent with the hypothesis that AVP acts as an endogenous antipyretic within the VSA during fever. Moreover, the data suggest a possible role for centrally acting vasopressin during pyrogenic tolerance to E. coli endotoxin.

Exercise- and cold-induced changes in plasma beta-endorphin and beta-lipotropin in men and women

1987 ◽  
Vol 62 (2) ◽  
pp. 622-627 ◽  
Author(s):  
M. Viswanathan ◽  
J. P. Van Dijk ◽  
T. E. Graham ◽  
A. Bonen ◽  
J. C. George
Keyword(s):  
Exercise Bout ◽  
Bicycle Ergometer ◽  
Relative Increase ◽  
Thermoregulatory Response ◽  
O2 Uptake ◽  
Beta Endorphin ◽  
Exposure Levels ◽  
Induced Changes ◽  
Ergometer Test ◽  
Bicycle Ergometer Test

The plasma beta-endorphin (beta-EP) and beta-lipotropin (beta-LPH) response of men, eumenorrheic women, and amenorrheic women (n = 6) to 1 h of rest or to a bicycle ergometer test [20 min at 30% maximum O2 uptake (VO2max), 20 min at 60% VO2max, and at 90% VO2max to exhaustion] was studied in both normal (22 degrees C) and cold (5 degrees C) environments. beta-EP and beta-LPH was measured by radioimmunoassay in venous samples collected every 20 min during rest or after each exercise bout. Exhaustive exercise at ambient temperature (Ta) 22 degrees C induced significant increases in plasma beta-EP and beta-LPH in all subjects as did work at 60% VO2max in amenorrheic and eumenorrheic women. During work at Ta 5 degrees C, the relative increase in beta-EP and beta-LPH was suppressed in eumenorrheic women and completely prevented in amenorrheic women. Although significant lowering of beta-EP and beta-LPH was observed in men and eumenorrheic women during rest at 5 degrees C, amenorrheic women maintained precold exposure levels. These findings suggest that plasma beta-EP and beta-LPH may reflect a thermoregulatory response to heat load. There appears to be a sexual dimorphism in exercise- and cold-induced release of beta-EP and beta-LPH and amenorrhea may be accompanied by alterations in these responses.

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