LEVATOR SCAPULAE SYNDROME

2005 ◽  
pp. 361-362
Keyword(s):  
Levator Scapulae

scholarly journals An Unusual Anatomical Variation of the Levator Scapulae Muscle

2012 ◽  
Vol 30 (3) ◽  
pp. 866-869 ◽  
Author(s):  
Gabriel Varjão Lima ◽  
Richard Halti Cabral ◽  
Danilo Leite Andrade ◽  
Nayara Soares de Oliveira Lacerda ◽  
Vital Fernandes Araújo ◽  
...  
Keyword(s):  
Anatomical Variation ◽  
Levator Scapulae ◽  
Levator Scapulae Muscle

Transfer of the Levator Scapulae, Rhomboid Major, and Rhomboid Minor for Paralysis of the Trapezius*

1996 ◽  
Vol 78 (10) ◽  
pp. 1534-40 ◽  
Author(s):  
LOUIS U. BIGLIANI ◽  
CATHERINE A. COMPITO ◽  
XAVIER A. DURALDE ◽  
IRA N. WOLFE
Keyword(s):  
Levator Scapulae

scholarly journals The role of hyoid muscles in biotremor production in Chamaeleo calyptratus

2020 ◽  
Vol 223 (22) ◽  
pp. jeb227603
Author(s):  
Samuel M. Tegge ◽  
Christopher V. Anderson ◽  
Michael E. Smith ◽  
Steve Huskey
Keyword(s):  
Body Temperature ◽  
Muscle Activation ◽  
Mixed Effect ◽  
Anatomical Structures ◽  
Levator Scapulae ◽  
Effect Regression ◽  
Activation Data ◽  
Chamaeleo Calyptratus ◽  
Mixed Effect Regression Model

ABSTRACTThe production of biotremors has been described in veiled chameleons (Chamaeleo calyptratus), but the mechanism by which they are produced is unknown. We gathered muscle activation data via electromyography (EMG), with simultaneous recordings of biotremors using an accelerometer, to test for the role of hyoid muscles in biotremor production. We recorded a mean biotremor frequency of 150.87 Hz for females and 136.01 Hz for males. The durations of activity and the latencies to onset and offset for the M. sternohyoideus profundus (SP), M. sternohyoideus superficialis (SS), Mm. mandibulohyoideus (MH) and M. levator scapulae (LS) were all significantly correlated with biotremor durations and biotremor onset and offset, respectively. Linear mixed-effect regression model comparisons of biotremor duration indicated that models containing either the MH and/or the SP and LS account for the most variation in biotremor duration. Twitch times for the SP (100 ms) and the SS (132 ms) at field active body temperature, however, were individually too slow to produce the biotremors at the observed frequency without alteration after production by other anatomical structures. These results implicate the SP, SS, MH and LS in the production of biotremors, but the exact mechanism of production requires further study.

Levator Scapulae Syndrome

1989 ◽  
Vol 17 (10) ◽  
pp. 57-68 ◽  
Author(s):  
Joseph J. Estwanik
Keyword(s):  
Levator Scapulae

scholarly journals A Cadaveric Investigation of the Dorsal Scapular Nerve

2016 ◽  
Vol 2016 ◽  
pp. 1-5 ◽  
Author(s):  
Vuvi H. Nguyen ◽  
Hao (Howe) Liu ◽  
Armando Rosales ◽  
Rustin Reeves
Keyword(s):  
Upper Extremity ◽  
Transverse Plane ◽  
Spinal Root ◽  
Scalene Muscle ◽  
Levator Scapulae ◽  
Cause Of Pain ◽  
Rhomboid Muscles ◽  
Levator Scapulae Muscle

Compression of the dorsal scapular nerve (DSN) is associated with pain in the upper extremity and back. Even though entrapment of the DSN within the middle scalene muscle is typically the primary cause of pain, it is still easily missed during diagnosis. The purpose of this study was to document the DSN’s anatomy and measure the oblique course it takes with regard to the middle scalene muscle. From 20 embalmed adult cadavers, 23 DSNs were documented regarding the nerve’s spinal root origin, anatomical route, and muscular innervations. A transverse plane through the laryngeal prominence was established to measure the distance of the DSN from this plane as it enters, crosses, and exits the middle scalene muscle. Approximately 70% of the DSNs originated from C5, with 74% piercing the middle scalene muscle. About 48% of the DSNs supplied the levator scapulae muscle only and 52% innervated both the levator scapulae and rhomboid muscles. The average distances from a transverse plane at the laryngeal prominence where the DSN entered, crossed, and exited the middle scalene muscle were 1.50 cm, 1.79 cm, and 2.08 cm, respectively. Our goal is to help improve clinicians’ ability to locate the site of DSN entrapment so that appropriate management can be implemented.

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