Regional Oxygen Balance

2005 ◽  
pp. 361-390
Keyword(s):  
Oxygen Balance

Oxygen Balance of the Kiev Reservoir in Wintertime

2002 ◽  
Vol 38 (3) ◽  
pp. 15
Author(s):  
O. V. Timchenko ◽  
V. M. Yakushin
Keyword(s):  
Oxygen Balance ◽  
Kiev Reservoir

scholarly journals Oxygen balance during laser powder bed fusion of Alloy 718

2021 ◽  
Vol 201 ◽  
pp. 109511
Author(s):  
Camille Pauzon ◽  
Ahmad Raza ◽  
Eduard Hryha ◽  
Pierre Forêt
Keyword(s):  
Oxygen Balance ◽  
Powder Bed Fusion ◽  
Alloy 718 ◽  
Laser Powder Bed Fusion ◽  
Powder Bed

scholarly journals In-sewer Treatment System of Enhancing Self-Purification: Performance and Oxygen Balance in Pilot Tests

2015 ◽  
Vol 13 (6) ◽  
pp. 427-439 ◽  
Author(s):  
Tadashi SHOJI ◽  
Yoshiharu MATSUBARA ◽  
Satoshi TAMAKI ◽  
Katsuo MATSUZAKA ◽  
Hiroyasu SATOH ◽  
...  
Keyword(s):  
Treatment System ◽  
Oxygen Balance ◽  
Pilot Tests

Cerebral oxygenation monitoring in patients during and after cardiac arrest -- a narrative review of current methods and evidence

2021 ◽  
Keyword(s):  
Cardiac Arrest ◽  
Near Infrared ◽  
Cerebral Oxygenation ◽  
The Body ◽  
Narrative Review ◽  
Oxygen Balance ◽  
Monitoring Methods ◽  
Venous Oxygen Saturation ◽  
Oxygenation Monitoring ◽  
The Impact

Hypoxic-ischemic brain injury (HIBI) is a leading cause of mortality in post-cardiac arrest (post-CA) patients who successfully survive the initial cardiopulmonary resuscitation (CPR) but later die in the Intensive Care Unit (ICU). Therefore, a key priority of post-resuscitation ICU care is to prevent and limit the impact of HIBI by optimizing the balance between cerebral oxygen delivery and demand. Traditionally, an optimal systemic oxygen balance is considered to ensure the brain’s oxygen balance. However, the validity of this assumption is uncertain, as the brain constitutes only 2%of the body mass while accounting for approximately 20% of basal oxygen consumption at rest. Hence, there is a real need to monitor cerebral oxygenation realistically. Several imaging and bedside monitoring methods are available for cerebral oxygenation monitoring in post-CA patients. Unfortunately, each of them has its limitations. Imaging methods require transporting a critically ill unstable patient to the scanner. Moreover, they provide an assessment of the oxygenation state only at a particular moment, while brain oxygenation is dynamic. Bedside methods, specifically near-infrared spectroscopy (NIRS), brain tissue oxygen tension (PbtO2), and jugular venous oxygen saturation monitoring (SjvO2), have not often been used in studies involving post-CA patients. Hence there is ambiguity regarding clear recommendations for using these bedside monitors. Presently, the most promising option seems to be using the NIRS as an indicator of effective CPR. We present a narrative review focusing on bedside methods and discuss the evidence for their use in adult patients after cardiac arrest.

Oxygen Balance of an Estuary

1961 ◽  
Vol 126 (3) ◽  
pp. 556-575 ◽  
Author(s):  
Donald J. O'Connor
Keyword(s):  
Oxygen Balance

Photosynthesis and Oxygen Balance in Streams

1970 ◽  
Vol 96 (2) ◽  
pp. 547-571
Author(s):  
Donald J. O'Connor ◽  
Dominic M. Di Toro
Keyword(s):  
Oxygen Balance

Alteration in myocardial oxygen balance during exercise after beta-adrenergic blockade in dogs

1980 ◽  
Vol 49 (1) ◽  
pp. 28-33 ◽  
Author(s):  
G. R. Heyndrickx ◽  
J. L. Pannier ◽  
P. Muylaert ◽  
C. Mabilde ◽  
I. Leusen
Keyword(s):  
Heart Rate ◽  
Blood Flow ◽  
Oxygen Consumption ◽  
Myocardial Blood Flow ◽  
Coronary Blood Flow ◽  
Coronary Sinus ◽  
Myocardial Oxygen Consumption ◽  
Left Ventricular ◽  
Adrenergic Blockade ◽  
Oxygen Balance

The effects of beta-adrenergic blockade upon myocardial blood flow and oxygen balance during exercise were evaluated in eight conscious dogs, instrumented for chronic measurements of coronary blood flow, left ventricular pressure, aortic blood pressure, heart rate, and sampling of arterial and coronary sinus venous blood. The administration of propranolol (1.5 mg/kg iv) produced a decrease in heart rate, peak left ventricular (LV) dP/dt, LV (dP/dt/P, and an increase in LV end-diastolic pressure during exercise. Mean coronary blood flow and myocardial oxygen consumption were lower after propranolol than at the same exercise intensity in control conditions. The oxygen delivery-to-oxygen consumption ratio and the coronary sinus oxygen content were also significantly lower. It is concluded that the relationship between myocardial oxygen supply and demand is modified during exercise after propranolol, so that a given level of myocardial oxygen consumption is achieved with a proportionally lower myocardial blood flow and a higher oxygen extraction.

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