Role of invasive monitoring in heart failure: Pulmonary artery catheters in the post-ESCAPE era

Heart Failure ◽  
2012 ◽  
pp. 325-331
Keyword(s):  
Heart Failure ◽  
Pulmonary Artery ◽  
Invasive Monitoring ◽  
Pulmonary Artery Catheters

Heart failure in an elderly man: where is that coronary?

2021 ◽  
pp. 1-4
Author(s):  
Charlie J. Sang ◽  
Stephen A. Clarkson ◽  
Elizabeth A. Jackson ◽  
Firas Al Solaiman ◽  
Marc G. Cribbs
Keyword(s):  
Heart Failure ◽  
Coronary Artery ◽  
Pulmonary Artery ◽  
Medical Therapy ◽  
Coronary Arteries ◽  
Right Coronary Artery ◽  
Adult Population ◽  
Anomalous Coronary ◽  
Anomalous Coronary Arteries

Abstract Anomalous coronary arteries from the pulmonary artery are uncommon causes of heart failure in the adult population. This case demonstrates the unusual presentation in a patient with anomalous right coronary artery from the pulmonary artery and discusses the complex pathophysiology of this lesion and the role of guideline-directed medical therapy in the management of these patients.

scholarly journals High Mortality Without ESCAPE: The Registry of Heart Failure Patients Receiving Pulmonary Artery Catheters Without Randomization

2008 ◽  
Vol 14 (8) ◽  
pp. 661-669 ◽  
Author(s):  
Larry A. Allen ◽  
Joseph G. Rogers ◽  
J. Wayne Warnica ◽  
Thomas G. DiSalvo ◽  
Gudaye Tasissa ◽  
...  
Keyword(s):  
Heart Failure ◽  
Pulmonary Artery ◽  
High Mortality ◽  
Heart Failure Patients ◽  
Pulmonary Artery Catheters

scholarly journals Pulmonary artery catheters in acute heart failure: end of an era?

Critical Care ◽  
2009 ◽  
Vol 13 (6) ◽  
pp. 1003 ◽  
Author(s):  
Christopher Vernon ◽  
Charles R Phillips
Keyword(s):  
Heart Failure ◽  
Pulmonary Artery ◽  
Acute Heart Failure ◽  
Pulmonary Artery Catheters

scholarly journals Emerging Role of Galectin-3 in Pulmonary Artery Hypertension

2018 ◽  
Vol 1 (1) ◽  
pp. p35
Author(s):  
Alexander E. Berezin
Keyword(s):  
Heart Failure ◽  
Cell Proliferation ◽  
Pulmonary Artery ◽  
Short Communication ◽  
Clinical Status ◽  
Pulmonary Artery Hypertension ◽  
All Cause Mortality ◽  
Galectin 3 ◽  
Pulmonary Arterial

Pulmonary arterial hypertension (PAH) is multifactorial disadptive disease with poor clinical outcomes associated with increased pulmonary artery pressure resulting in primary small-to-moderate pulmonary artery remodeling. Numerous factors, including smooth muscle cell proliferation, vasospasm, vascular fibrosis and occlusion, direct vascular injury and inflammation, impaired repair of vasculature, are involved in the pathogenesis of PAH. It has been suggested that galectin-3 as a biomarker of excessive fibrosis and inflammation can be useful predictor of both severity and prognosis in patient with PAH. The short communication is reported that elevated Gal-3 levels were found in majority patients with PAH depending on clinical status and of the disease. Although elevated Gal-3 levels were associated with a higher risk of all-cause mortality, cardiovascular mortality, and right ventricle heart failure, the value of this biomarker in PAH patients at high risk stratification is uncertain and requires to be investigated in large clinical trials.

Protective role of peroxiredoxin-4 in heart failure

2020 ◽  
Vol 134 (1) ◽  
pp. 71-72
Author(s):  
Naseer Ahmed ◽  
Masooma Naseem ◽  
Javeria Farooq
Keyword(s):  
Heart Failure ◽  
Cardiac Fibroblasts ◽  
Protective Role ◽  
Oxidative Stress Markers ◽  
Stress Markers ◽  
Total Antioxidant ◽  
Galectin 3 ◽  
Consequent Increase ◽  
And Oxidative Stress

Abstract Recently, we have read with great interest the article published by Ibarrola et al. (Clin. Sci. (Lond.) (2018) 132, 1471–1485), which used proteomics and immunodetection methods to show that Galectin-3 (Gal-3) down-regulated the antioxidant peroxiredoxin-4 (Prx-4) in cardiac fibroblasts. Authors concluded that ‘antioxidant activity of Prx-4 had been identified as a protein down-regulated by Gal-3. Moreover, Gal-3 induced a decrease in total antioxidant capacity which resulted in a consequent increase in peroxide levels and oxidative stress markers in cardiac fibroblasts.’ We would like to point out some results stated in the article that need further investigation and more detailed discussion to clarify certain factors involved in the protective role of Prx-4 in heart failure.

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