- Thyrotoxic Crisis

2012 ◽  
pp. 410-415
Keyword(s):  
Thyrotoxic Crisis

E-041 Is there an Interventional Role in Thyrotoxic Crisis?

2013 ◽  
Vol 5 (Suppl 2) ◽  
pp. A52.1-A52
Author(s):  
R Morgan ◽  
S Best ◽  
C Connor ◽  
P Johnson ◽  
J Madarang
Keyword(s):  
Thyrotoxic Crisis

Reserpine in Thyrotoxic Crisis

1970 ◽  
Vol 283 (19) ◽  
pp. 1020-1023 ◽  
Author(s):  
Pauline T. Dillon ◽  
John Babe ◽  
C. R. Meloni ◽  
John I. Canary
Keyword(s):  
Thyrotoxic Crisis

Thyroid Storm (Thyrotoxic Crisis)

2019 ◽  
pp. 357-366 ◽  
Author(s):  
Leonard Wartofsky ◽  
Joanna Klubo-Gwiezdzinska
Keyword(s):  
Thyroid Storm ◽  
Thyrotoxic Crisis

Thyrotoxic Crisis and Diabetic Ketoacidosis

1957 ◽  
Vol 257 (15) ◽  
pp. 697-701 ◽  
Author(s):  
Donald H. Hanscom ◽  
Robert J. Ryan
Keyword(s):  
Diabetic Ketoacidosis ◽  
Thyrotoxic Crisis

Iodine Metabolism in a Case of Thyrotoxic Crisis

1963 ◽  
Vol 23 (4) ◽  
pp. 397-400 ◽  
Author(s):  
PETER WAHLBERG ◽  
B.-A. LAMBERG
Keyword(s):  
Iodine Metabolism ◽  
Thyrotoxic Crisis

scholarly journals Unmasking of Gitelman Syndrome during Pregnancy in an Adolescent with Thyrotoxic Crisis

2021 ◽  
Vol 13 (4) ◽  
pp. 632-638
Author(s):  
Ratna Acharya ◽  
Kiran Upadhyay
Keyword(s):  
Thyroid Nodules ◽  
Hyperemesis Gravidarum ◽  
Thyroid Stimulating Hormone ◽  
Gitelman Syndrome ◽  
Structural Homology ◽  
Case Summary ◽  
Beta Human Chorionic Gonadotropin ◽  
Thyrotoxic Crisis ◽  
High Index Of Suspicion ◽  
Thyroid Dysfunctions

Background. Gitelman syndrome (GS) is an inherited salt-losing renal tubulopathy characterized by hypokalemic metabolic alkalosis with hypomagnesemia and hypocalciuria. Patients can be asymptomatic until late adolescence or adulthood, and hence may be discovered incidentally during presentation with other illnesses. GS has been described in association with thyroid disorders and should be considered in patients with hyperthyroidism and persistent hypokalemia, especially in those with associated hypomagnesemia and hypocalciuria. Case summary. In this report, we describe an 18-year-old female who presented with hyperemesis gravidarum and thyrotoxicosis, and was incidentally found to have GS, confirmed by the sequence analysis of SLC12A3. Conclusions. Thyroid dysfunctions, such as hypothyroidism, thyrotoxicosis, and thyroid nodules, may develop during pregnancy. A structural homology between the beta-human chorionic gonadotropin and thyroid stimulating hormone molecules, as well as their receptors is probably the basis for the transient thyrotoxicosis crisis during pregnancy. Since hyperemesis in pregnancy can also lead to hypokalemia and alkalosis, a high index of suspicion for GS during pregnancy is required for timely diagnosis and management.

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