Epigenetic Mechanisms in Lung In•ammation and Chronic Airway Diseases and Intervention by Dietary Polyphenols

2010 ◽  
pp. 202-231
Keyword(s):  
Epigenetic Mechanisms ◽  
Dietary Polyphenols ◽  
Airway Diseases ◽  
Chronic Airway

scholarly journals Evaluation of Life Quality and Dyspnea Intensity on Chronic Airway Diseases Patients in Pulmonary Rehabilitation Program

2016 ◽  
Vol 04 (01) ◽  
pp. 10-15
Author(s):  
Kathleen Julia Silva ◽  
Giseli Domingues Cordeiro ◽  
Caroline Rossinoli ◽  
Maurício Longo Galhardo ◽  
Márcia Maria Faganello
Keyword(s):  
Pulmonary Rehabilitation ◽  
Life Quality ◽  
Rehabilitation Program ◽  
Airway Diseases ◽  
Pulmonary Rehabilitation Program ◽  
Chronic Airway

The Frequency of Small Airway Dysfunction and Its Relationship with Disease Control in Chronic Airway Diseases

2021 ◽  
Author(s):  
Demet Polat Yulug ◽  
Sibel Atış Naycı ◽  
Bahar Taşdelen ◽  
Eylem Sercan Ozgur ◽  
Cengiz Ozge
Keyword(s):  
Disease Control ◽  
Small Airway ◽  
Airway Diseases ◽  
Chronic Airway

scholarly journals Molecular mechanisms of action of naringenin in chronic airway diseases

2020 ◽  
Vol 879 ◽  
pp. 173139 ◽  
Author(s):  
Li Hian Chin ◽  
Chian Ming Hon ◽  
Dinesh Kumar Chellappan ◽  
Jestin Chellian ◽  
Thiagarajan Madheswaran ◽  
...  
Keyword(s):  
Molecular Mechanisms ◽  
Mechanisms Of Action ◽  
Airway Diseases ◽  
Chronic Airway

scholarly journals Staphylococcus Aureus in chronic airway diseases: An overview

2019 ◽  
Vol 155 ◽  
pp. 66-71 ◽  
Author(s):  
Martina Flora ◽  
Fabio Perrotta ◽  
Ambra Nicolai ◽  
Rosalba Maffucci ◽  
Anna Pratillo ◽  
...  
Keyword(s):  
Staphylococcus Aureus ◽  
Airway Diseases ◽  
Chronic Airway

scholarly journals Fourteen-Member Macrolides Suppress Interleukin-8 Production but Do Not Promote Apoptosis of Activated Neutrophils

2002 ◽  
Vol 46 (4) ◽  
pp. 1101-1104 ◽  
Author(s):  
Yoshiko Tsuchihashi ◽  
Kazunori Oishi ◽  
Hiroyuki Yoshimine ◽  
Shoichi Suzuki ◽  
Atsushi Kumatori ◽  
...  
Keyword(s):  
Clinical Efficacy ◽  
Interleukin 8 ◽  
Airway Diseases ◽  
Activated Neutrophils ◽  
Chronic Airway

ABSTRACT A 14-member macrolide was found to inhibit interleukin-8 (IL-8) synthesis in lipopolysaccharide-stimulated neutrophils but did not accelerate apoptosis in activated neutrophils. These data suggest that 14-member macrolides achieve clinical efficacy for chronic airway diseases partly by suppressing IL-8 production by activated neutrophils, but not by enhancing apoptosis in these cells.

Angiogenesis: A potentially critical part of remodelling in chronic airway diseases?

2008 ◽  
Vol 118 (1) ◽  
pp. 128-137 ◽  
Author(s):  
Eugene Haydn Walters ◽  
David Reid ◽  
Amir Soltani ◽  
Chris Ward
Keyword(s):  
Critical Part ◽  
Airway Diseases ◽  
Chronic Airway

scholarly journals Vanadium pentoxide (V2O5) induced mucin production by airway epithelium

2011 ◽  
Vol 301 (1) ◽  
pp. L31-L39 ◽  
Author(s):  
Dongfang Yu ◽  
Dianne M. Walters ◽  
Lingxiang Zhu ◽  
Pak-Kei Lee ◽  
Yin Chen
Keyword(s):  
Vanadium Pentoxide ◽  
Mapk Pathway ◽  
Mechanistic Study ◽  
Chronic Obstructive ◽  
In Vivo Models ◽  
Mucin Production ◽  
Airway Diseases ◽  
Chronic Airway

Exposure to environmental pollutants has been linked to various airway diseases and disease exacerbations. Almost all chronic airway diseases such as chronic obstructive pulmonary disease and asthma are caused by complicated interactions between gene and environment. One of the major hallmarks of those diseases is airway mucus overproduction (MO). Excessive mucus causes airway obstruction and significantly increases morbidity and mortality. Metals are major components of environmental particulate matters (PM). Among them, vanadium has been suggested to play an important role in PM-induced mucin production. Vanadium pentoxide (V2O5) is the most common commercial source of vanadium, and it has been associated with occupational chronic bronchitis and asthma, both of which are MO diseases. However, the underlying mechanism is not entirely clear. In this study, we used both in vitro and in vivo models to demonstrate the robust inductions of mucin production by V2O5. Furthermore, the follow-up mechanistic study revealed a novel v-raf-1 murine leukemia viral oncogene homolog 1-IKK-NF-κB pathway that mediated V2O5-induced mucin production. Most interestingly, the reactive oxygen species and the classical mucin-inducing epidermal growth factor receptor (EGFR)-MAPK pathway appeared not to be involved in this process. Thus the V2O5-induced mucin production may represent a novel EGFR-MAPK-independent and environmental toxicant-associated MO model. Complete elucidation of the signaling pathway in this model will not only facilitate the development of the treatment for V2O5-associated occupational diseases but also advance our understanding on the EGFR-independent mucin production in other chronic airway diseases.

MicroRNAs in chronic airway diseases: Clinical correlation and translational applications

2020 ◽  
Vol 160 ◽  
pp. 105045 ◽  
Author(s):  
Bryce W.Q. Tan ◽  
Wei Liang Sim ◽  
Jit Kong Cheong ◽  
Win Sen Kuan ◽  
Thai Tran ◽  
...  
Keyword(s):  
Clinical Correlation ◽  
Airway Diseases ◽  
Chronic Airway
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