scholarly journals Respiratory variations in aortic blood flow velocity and inferior vena cava diameter as predictors of fluid responsiveness in mechanically ventilated children using transthoracic echocardiography in a pediatric PICU

Critical Care ◽  
2015 ◽  
Vol 19 (Suppl 1) ◽  
pp. P181 ◽  
Author(s):  
K Halimi ◽  
M Negadi ◽  
H Bouguetof ◽  
L Zemour ◽  
D Boumendil ◽  
...  
Keyword(s):  
Blood Flow ◽  
Inferior Vena Cava ◽  
Blood Flow Velocity ◽  
Inferior Vena ◽  
Vena Cava ◽  
Aortic Blood Flow ◽  
Mechanically Ventilated ◽  
Aortic Blood ◽  
Inferior Vena Cava Diameter ◽  
Aortic Blood Flow Velocity

Changes in Arterial Pressure during Mechanical Ventilation

2005 ◽  
Vol 103 (2) ◽  
pp. 419-428 ◽  
Author(s):  
Frédéric Michard
Keyword(s):  
Mechanical Ventilation ◽  
Blood Flow ◽  
Arterial Pressure ◽  
Vena Cava ◽  
Cardiac Preload ◽  
Physical Sign ◽  
Aortic Blood Flow ◽  
Mechanically Ventilated ◽  
Aortic Blood ◽  
Cyclic Changes

Mechanical ventilation induces cyclic changes in vena cava blood flow, pulmonary artery blood flow, and aortic blood flow. At the bedside, respiratory changes in aortic blood flow are reflected by "swings" in blood pressure whose magnitude is highly dependent on volume status. During the past few years, many studies have demonstrated that arterial pressure variation is neither an indicator of blood volume nor a marker of cardiac preload but a predictor of fluid responsiveness. That is, these studies have demonstrated the value of this physical sign in answering one of the most common clinical questions, Can we use fluid to improve hemodynamics?, while static indicators of cardiac preload (cardiac filling pressures but also cardiac dimensions) are frequently unable to correctly answer this crucial question. The reliable analysis of respiratory changes in arterial pressure is possible in most patients undergoing surgery and in critically ill patients who are sedated and mechanically ventilated with conventional tidal volumes.

Respiratory Variation in Aortic Blood Flow Velocity in Hemodynamically Unstable, Ventilated Neonates

2020 ◽  
Vol Publish Ahead of Print ◽  
Author(s):  
Ignacio Oulego-Erroz ◽  
Sandra Terroba-Seara ◽  
Paula Alonso-Quintela ◽  
Antonio Rodríguez-Núñez
Keyword(s):  
Blood Flow ◽  
Flow Velocity ◽  
Blood Flow Velocity ◽  
Aortic Blood Flow ◽  
Respiratory Variation ◽  
Aortic Blood ◽  
Aortic Blood Flow Velocity

Doppler Measurement of Parasternal Aortic Blood Flow Velocity

2017 ◽  
Vol 49 (5S) ◽  
pp. 726
Author(s):  
Evan L. Matthews ◽  
Stephanie A. Guarino ◽  
Jennifer M. Masiddo ◽  
Peter A. Hosick
Keyword(s):  
Blood Flow ◽  
Flow Velocity ◽  
Blood Flow Velocity ◽  
Aortic Blood Flow ◽  
Doppler Measurement ◽  
Aortic Blood ◽  
Aortic Blood Flow Velocity

Cardiac afferents attenuate the muscle metaboreflex in the rat

1993 ◽  
Vol 75 (1) ◽  
pp. 114-120 ◽  
Author(s):  
H. L. Collins ◽  
S. E. DiCarlo
Keyword(s):  
Blood Flow ◽  
Flow Velocity ◽  
Blood Flow Velocity ◽  
Volume Expansion ◽  
Aortic Blood Flow ◽  
Muscle Metaboreflex ◽  
Aortic Blood ◽  
Blood Volume Expansion ◽  
Cardiac Afferents ◽  
Aortic Blood Flow Velocity

The influence of cardiac afferents on the muscle metaboreflex was examined in 16 rats instrumented with a Silastic-tipped catheter in the pericardial space and right atrium, Doppler ultrasonic flow probe and a pneumatic vascular occluder around the terminal aorta, and a Teflon catheter in the thoracic aorta. In protocol I (cardiac efferent and afferent blockade), the muscle metaboreflex was examined under three experimental conditions: 1) control, 2) cardiac autonomic efferent blockade [intrapericardial methylscopolamine (10 micrograms/kg) and propranolol (50 micrograms/kg)], and 3) combined cardiac autonomic efferent and afferent blockade (intrapericardial procainamide, 2%). In protocol II (blood volume expansion), the muscle metaboreflex was examined before and after 15% blood volume expansion. Mild treadmill exercise (9 m/min, 10% grade) increased heart rate (71 +/- 9.4 beats/min), mean arterial pressure (12 +/- 2.0 mmHg), and terminal aortic blood flow velocity (6 +/- 1.0 kHz). During exercise, a reduction of terminal aortic blood flow velocity (10.5 +/- 1.1%) reduced mixed venous PO2 18 +/- 6%. The gain of the muscle metaboreflex in the control condition was 14.6 +/- 2.9 mmHg/kHz. Efferent blockade reduced the gain 51 +/- 7%. However, combined cardiac efferent and afferent blockade increased the gain 207 +/- 64% above the efferent blocked condition and restored the gain to levels above those obtained in the control condition (18.3 +/- 4.6 mmHg/kHz). In addition, 15% blood volume expansion reduced the gain of the muscle metaboreflex regulation of mean arterial pressure and heart rate (44 +/- 9.5% and 41 +/- 12.0%, respectively). Thus cardiac afferents tonically inhibit the pressor response to a reduction in terminal aortic blood flow velocity during exercise.

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