scholarly journals Serological Cross-Reactions Between Different Brucella Species and Yersinia Enterocolitica Immunodiffusion and Immunoelectrophoresis

1972 ◽  
Vol 13 (4) ◽  
pp. 472-483
Author(s):  
B. Hurvell
2003 ◽  
Vol 10 (4) ◽  
pp. 710-714 ◽  
Author(s):  
Janchivdorj Erdenebaatar ◽  
Balgan Bayarsaikhan ◽  
Masahisa Watarai ◽  
Sou-ichi Makino ◽  
Toshikazu Shirahata

ABSTRACT Enzyme-linked immunosorbent assays using antigens extracted from Brucella abortus with n-lauroylsarcosine differentiated natural Brucella-infected animals from Brucella-vaccinated or Yersinia enterocolitica O9-infected animals. A field trial in Mongolia showed cattle, sheep, goat, reindeer, camel, and human sera without infection could be distinguished from Brucella-infected animals by conventional serological tests.


1991 ◽  
Vol 107 (2) ◽  
pp. 349-356 ◽  
Author(s):  
H. Chart ◽  
T. Cheasty ◽  
D. Cope ◽  
R. J. Gross ◽  
B. Rowe

SUMMARYSera from patients with yersiniosis. shown to contain antibodies toYersinia enterocoliticaO9; and sera from patients with haemolytic uraemic syndrome (HUS) caused byEscherichia coliO157, were used to investigate serological cross-reactions betweenY. enterocoliticaO9 andE. coliO157. Lipopolysaccharide (LPS) was isolated from strains ofY. enterocoliticaO9 andE. coliO157 and reacted with sera by immunoblotting and ELISA. Sera from patients with HUS contained antibodies to the LPS ofE. coliO157 only; 80% of sera from patients with yersiniosis contained antibodies to the LPS ofY. enterocoliticaO9 andE. coliO157. This one-way cross-reaction was also detected using hyperimmune rabbit antisera.


Author(s):  
Gerhard Dobler

• TBE appears with non-characteristic clinical symptoms, which cannot be distinguished from oth-er forms of viral encephalitis or other diseases. • Cerebrospinal fluid and neuro-imaging may give some evidence of TBE, but ultimately cannot confirm the diagnosis. • Thus, proving the diagnosis “TBE” necessarily requires confirmation of TBEV-infection by detec-tion of the virus or by demonstration of specific antibodies from serum and/or cerebrospinal fluid. • During the phase of clinic symptoms from the CNS, the TBEV can only rarely be detected in the cerebrospinal fluid of patients. • Most routinely used serological tests for diagnosing TBE (ELISA, HI, IFA) show cross reactions resulting from either Infection with other flaviviruses or with other flavivirus vaccines.


TBE appears with non-characteristic clinical symptoms, which cannot be distinguished from other forms of viral encephalitis or other diseases. Cerebrospinal fluid and neuro-imaging may give some evidence of TBE, but ultimately cannot confirm the diagnosis. Thus, proving the diagnosis “TBE” necessarily requires confirmation of TBEV-infection by detection of the virus or by demonstration of specific antibodies from serum and/or cerebrospinal fluid. During the phase of clinic symptoms from the CNS, the TBEV can only rarely be detected in the cerebrospinal fluid of patients. Most routinely used serological tests for diagnosing TBE (ELISA, HI, IFA) show cross reactions resulting from either infection with other flaviviruses or with other flavivirus vaccines.


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