scholarly journals Ambient air pollution, traffic noise and adult asthma prevalence: a BioSHaRE approach

2016 ◽  
Vol 49 (1) ◽  
pp. 1502127 ◽  
Author(s):  
Yutong Cai ◽  
Wilma L. Zijlema ◽  
Dany Doiron ◽  
Marta Blangiardo ◽  
Paul R. Burton ◽  
...  

We investigated the effects of both ambient air pollution and traffic noise on adult asthma prevalence, using harmonised data from three European cohort studies established in 2006–2013 (HUNT3, Lifelines and UK Biobank).Residential exposures to ambient air pollution (particulate matter with aerodynamic diameter ≤10 µm (PM10) and nitrogen dioxide (NO2)) were estimated by a pan-European Land Use Regression model for 2007. Traffic noise for 2009 was modelled at home addresses by adapting a standardised noise assessment framework (CNOSSOS-EU). A cross-sectional analysis of 646 731 participants aged ≥20 years was undertaken using DataSHIELD to pool data for individual-level analysis via a “compute to the data” approach. Multivariate logistic regression models were fitted to assess the effects of each exposure on lifetime and current asthma prevalence.PM10 or NO2 higher by 10 µg·m−3 was associated with 12.8% (95% CI 9.5–16.3%) and 1.9% (95% CI 1.1–2.8%) higher lifetime asthma prevalence, respectively, independent of confounders. Effects were larger in those aged ≥50 years, ever-smokers and less educated. Noise exposure was not significantly associated with asthma prevalence.This study suggests that long-term ambient PM10 exposure is associated with asthma prevalence in western European adults. Traffic noise is not associated with asthma prevalence, but its potential to impact on asthma exacerbations needs further investigation.

Author(s):  
Abhishek Adkine ◽  
Kalyani Mahajan

Vehicular traffic contributes significantly to the pollution of air and neighborhood noise. Since there is increasing affirmation that ambient air pollution hurts reproductive health, which is less known regarding the relationship between traffic noise and pregnancy outcomes. Many researchers have investigated all the possible adverse effects of ambient air pollution on birth outcomes during the last decade or so. We examined these studies, which were discovered by thoroughly searching the major scientific databases. Overall, research suggests that air pollution and noise pollution significantly impact birth outcomes, while the quality of the deposition varies. Researchers utilized connected administrative health data sources to discover pregnancy outcomes with detailed covariate data (gender, contemporary lifestyle, total no of pregnancies, birth month and year, and so on), earnings and education, and mother housing history. Using the deterministic model (CadnaA) and air pollutants exposure, they assessed noise exposure during pregnancy by assessing temporally adjusted land-use regression. Noise exposure is related to reduced birth weight. We also looked into various processes to prevent air pollution and noise. As a result of the policy change and technical innovation, the government, authorities, and industry have been at the forefront of combating pollution. Pollution levels in and around people's homes and workplaces are typically beyond their control. However, few things may be done to mitigate the impacts of contaminated air.


Thorax ◽  
2021 ◽  
pp. thoraxjnl-2020-216142
Author(s):  
Dany Doiron ◽  
Jean Bourbeau ◽  
Kees de Hoogh ◽  
Anna L Hansell

BackgroundFew large studies have assessed the relationship of long-term ambient air pollution exposure with the prevalence and incidence of symptoms of chronic bronchitis and cough.MethodsWe leveraged Lifelines cohort data on 132 595 (baseline) and 65 009 (second assessment) participants linked to ambient air pollution estimates. Logistic regression models adjusted for sex, age, educational attainment, body mass index, smoking status, pack-years smoking and environmental tobacco smoke at home were used to assess associations of air pollution with prevalence and incidence of chronic bronchitis (winter cough and sputum almost daily for ≥3 months/year), chronic cough (winter cough almost daily for ≥3 months/year) and prevalence of cough and sputum symptoms, irrespective of duration.ResultsAssociations were seen for all pollutants for prevalent cough or sputum symptoms. However, for prevalent and incident chronic bronchitis, statistically significant associations were seen for nitrogen dioxide (NO2) and black carbon (BC) but not for fine particulate matter (PM2.5). For prevalent chronic bronchitis, associations with NO2 showed OR: 1.05 (95% CI: 1.02 to 1.08) and with BC OR: 1.06 (95% CI: 1.03 to 1.09) expressed per IQR; corresponding results for incident chronic bronchitis were NO2 OR: 1.07 (95% CI: 1.02 to 1.13) and BC OR: 1.07 (95% CI: 1.02 to 1.13). In subgroup analyses, slightly stronger associations were observed among women, never smokers and younger individuals.ConclusionThis is the largest analysis to date to examine cross-sectional and longitudinal associations between ambient air pollution and chronic bronchitis. NO2 and BC air pollution was associated with increased odds of prevalent and incident chronic bronchitis.


2021 ◽  
Vol 152 ◽  
pp. 106464 ◽  
Author(s):  
Shuo Liu ◽  
Youn-Hee Lim ◽  
Marie Pedersen ◽  
Jeanette T. Jørgensen ◽  
Heresh Amini ◽  
...  

2020 ◽  
Vol 223 ◽  
pp. 117268 ◽  
Author(s):  
Zahra Namvar ◽  
Masud Yunesian ◽  
Mansour Shamsipour ◽  
Mohammad Sadegh Hassanvand ◽  
Kazem Naddafi ◽  
...  

2019 ◽  
Vol 34 (2) ◽  
pp. 211-218
Author(s):  
Toluwanimi Mobolade Oni ◽  
Godson R.E.E. Ana

Abstract Background There is an increasing range of adverse health effects associated with air pollution at very low concentrations. Few studies have assessed respiratory parameters among filling station attendants. Objectives This study assessed air pollutants; particulate matter (PM10) and total volatile organic compounds (TVOC) concentrations at filling stations as well as determined forced expiratory volume in one second (FEV1) and peak expiratory flow rate (PEFR) levels among filling station attendants. Methods A cross-sectional study was conducted to assess PM10 and TVOC concentrations at 20 systematically selected filling stations in Ibadan North Local Government Area, Ibadan for 2 months using a Thermo Scientific pDR 1500 PM10 monitor and SF2000-TVOC meter. FEV1 and PEFR levels were measured in order to assess the effect of exposure to PM10 and TVOC on lung function of 100 filling station attendants using a PIKO-1 Electronic peakflow/FEV1 meter. Results Total mean PM10 concentrations (μg/m3) in the morning (43.7±16.5) and afternoon (27.8±7.9) were significantly lower (p<0.01) than the World Health Organization (WHO) guideline limit (50 μg/m3). Total mean TVOC concentrations (ppm) in the morning (12.0±3.4) and afternoon (5.6±2.4) were however significantly higher (p<0.01) than the Occupational Safety and Health Administration (OSHA) guideline limit (3 ppm). Mean FEV1 for filling station attendants was 1.63±0.39 and PEFR was 171.7±45.9. Conclusion Filling stations are hotspots for the emission of VOCs and PM10. However, filling station attendants in this study are at risk of exposure to high concentrations of VOCs but not PM10. FEV1 and PEFR values among filling station attendants were very low which could possibly be attributed to extended exposure to air pollutants. Regular medical examinations should also be conducted on filling station attendants in order to aid early detection of deviations in their health status.


2019 ◽  
Vol 76 (Suppl 1) ◽  
pp. A97.2-A97
Author(s):  
Ching-chun Huang ◽  
Yue Leon Guo

BackgroundEvidence regarding whether prenatal exposure to air pollution increases the risk of hypospadias is limited.ObjectivesThe aim of the study is to evaluate the association between exposure to ambient air pollution during early pregnancy and occurrence of hypospadias.MethodsWe conducted a 1:10 case-control study using the Taiwanese Birth Registry database. Those male births reported to have hypospadias were defined as cases; while controls were randomly, matched by birth year, selected from those male births without any congenital anomaly. Monthly average of ambient air pollutants, including PM10, PM2.5, NO2, NOx, and O3, from three months pre- to six months post-conception were retrieved from the 76 air quality monitoring stations and interpolated to the level of township using empirical bayesian kriging. Potential covariates to be adjusted included gestational age, birth weight, birth season, maternal age, maternal diabetes and hypertension, maternal smoking, annual household income and population density of the residential township.ResultsDuring 2007–2014, a total of 265 hypospadias was reported, and 230 (87%) of them were full-term births. Results of multivariate logistic regression models revealed that for per IQR increase of O3 (8.0 p.p.b) exposure during the first months after conception increased the risk of hypospadias (aOR=1.38, 95% CI=1.07–1.78). In subgroup analysis of full-term births, we further found that PM2.5 exposure during the first three months post-conception significantly increased the risk of developing hypospadias (aOR=1.29, 95% CI=1.01–1.65, per IQR=15.4 ug/m3).ConclusionsThe results of the study suggested that early gestational exposure to ambient air pollution increased the risk of hypospadias occurrence.


Thorax ◽  
2019 ◽  
Vol 74 (11) ◽  
pp. 1063-1069 ◽  
Author(s):  
Mary B Rice ◽  
Wenyuan Li ◽  
Joel Schwartz ◽  
Qian Di ◽  
Itai Kloog ◽  
...  

BackgroundAmbient air pollution accelerates lung function decline among adults, however, there are limited data about its role in the development and progression of early stages of interstitial lung disease.AimsTo evaluate associations of long-term exposure to traffic and ambient pollutants with odds of interstitial lung abnormalities (ILA) and progression of ILA on repeated imaging.MethodsWe ascertained ILA on chest CT obtained from 2618 Framingham participants from 2008 to 2011. Among 1846 participants who also completed a cardiac CT from 2002 to 2005, we determined interval ILA progression. We assigned distance from home address to major roadway, and the 5-year average of fine particulate matter (PM2.5), elemental carbon (EC, a traffic-related PM2.5 constituent) and ozone using spatio-temporal prediction models. Logistic regression models were adjusted for age, sex, body mass index, smoking status, packyears of smoking, household tobacco exposure, neighbourhood household value, primary occupation, cohort and date.ResultsAmong 2618 participants with a chest CT, 176 (6.7%) had ILA, 1361 (52.0%) had no ILA, and the remainder were indeterminate. Among 1846 with a preceding cardiac CT, 118 (6.4%) had ILA with interval progression. In adjusted logistic regression models, an IQR difference in 5-year EC exposure of 0.14 µg/m3 was associated with a 1.27 (95% CI 1.04 to 1.55) times greater odds of ILA, and a 1.33 (95% CI 1.00 to 1.76) times greater odds of ILA progression. PM2.5 and O3 were not associated with ILA or ILA progression.ConclusionsExposure to EC may increase risk of progressive ILA, however, associations with other measures of ambient pollution were inconclusive.


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