scholarly journals Role of T lymphocyte subsets in the pathogenesis of primary infection and rechallenge with respiratory syncytial virus in mice.

1991 ◽  
Vol 88 (3) ◽  
pp. 1026-1033 ◽  
Author(s):  
B S Graham ◽  
L A Bunton ◽  
P F Wright ◽  
D T Karzon
Keyword(s):  
Respiratory Syncytial Virus ◽  
T Lymphocyte ◽  
Primary Infection ◽  
Lymphocyte Subsets ◽  
T Lymphocyte Subsets ◽  
Syncytial Virus

Role of T-lymphocyte subsets in facial nerve paralysis owing to the reactivation of herpes simplex virus type 1

2005 ◽  
Vol 125 (3) ◽  
pp. 316-321 ◽  
Author(s):  
Hisanobu Kisaki ◽  
Naohito Hato ◽  
Mutsuhiko Mizobuchi ◽  
Nobumitsu Honda ◽  
Hirotaka Takahashi ◽  
...  
Keyword(s):  
Herpes Simplex Virus ◽  
Herpes Simplex ◽  
T Lymphocyte ◽  
Lymphocyte Subsets ◽  
Facial Nerve Paralysis ◽  
T Lymphocyte Subsets ◽  
Nerve Paralysis ◽  
Simplex Virus

Expression and role of phosphatidylcholine-specific phospholipase C in human NK and T lymphocyte subsets

2006 ◽  
Vol 36 (12) ◽  
pp. 3277-3287 ◽  
Author(s):  
Francesca Spadaro ◽  
Serena Cecchetti ◽  
Massimo Sanchez ◽  
Clara Maria Ausiello ◽  
Franca Podo ◽  
...  
Keyword(s):  
Phospholipase C ◽  
T Lymphocyte ◽  
Lymphocyte Subsets ◽  
T Lymphocyte Subsets

scholarly journals Role of T Lymphocyte Subsets in Protection and Recovery from Hantaan Virus Infection in Mice

1987 ◽  
Vol 68 (7) ◽  
pp. 1961-1969 ◽  
Author(s):  
H. Asada ◽  
M. Tamura ◽  
K. Kondo ◽  
Y. Okuno ◽  
Y. Takahashi ◽  
...  
Keyword(s):  
Virus Infection ◽  
T Lymphocyte ◽  
Lymphocyte Subsets ◽  
Hantaan Virus ◽  
T Lymphocyte Subsets

scholarly journals Role of T-lymphocyte subsets in Rhodococcus equi infection.

1992 ◽  
Vol 60 (7) ◽  
pp. 2748-2752 ◽  
Author(s):  
P Nordmann ◽  
E Ronco ◽  
C Nauciel
Keyword(s):  
T Lymphocyte ◽  
Lymphocyte Subsets ◽  
Rhodococcus Equi ◽  
T Lymphocyte Subsets

scholarly journals The role of T-lymphocyte subsets and interleukin-5 blood levels among Indian subjects with autoimmune thyroid disease

HORMONES ◽  
2010 ◽  
Vol 9 (1) ◽  
pp. 76-81 ◽  
Author(s):  
Sripathy Gopalakrishnan ◽  
Sourav Sen ◽  
Jawahar Singh Adhikari ◽  
Pradeep Kumar Chugh ◽  
Tarun Sekhri ◽  
...  
Keyword(s):  
T Lymphocyte ◽  
Thyroid Disease ◽  
Autoimmune Thyroid Disease ◽  
Lymphocyte Subsets ◽  
Blood Levels ◽  
T Lymphocyte Subsets ◽  
Autoimmune Thyroid ◽  
Interleukin 5

scholarly journals Herpes simplex virus-induced stromal keratitis: role of T-lymphocyte subsets in immunopathology.

1989 ◽  
Vol 63 (2) ◽  
pp. 769-775 ◽  
Author(s):  
C K Newell ◽  
S Martin ◽  
D Sendele ◽  
C M Mercadal ◽  
B T Rouse
Keyword(s):  
Herpes Simplex Virus ◽  
Herpes Simplex ◽  
T Lymphocyte ◽  
Lymphocyte Subsets ◽  
T Lymphocyte Subsets ◽  
Stromal Keratitis ◽  
Simplex Virus

scholarly journals Inability To Evoke a Long-Lasting Protective Immune Response to Respiratory Syncytial Virus Infection in Mice Correlates with Ineffective Nasal Antibody Responses

2003 ◽  
Vol 77 (21) ◽  
pp. 11303-11311 ◽  
Author(s):  
Richard Singleton ◽  
Nathalie Etchart ◽  
Sam Hou ◽  
Lisa Hyland
Keyword(s):  
Bone Marrow ◽  
Respiratory Syncytial Virus ◽  
Lymphoid Tissue ◽  
Primary Infection ◽  
Plasma Cells ◽  
Local Protection ◽  
Rsv Infection ◽  
Syncytial Virus ◽  
Antibody Levels

ABSTRACT Long-lasting protective antibody is not normally generated in children following primary respiratory syncytial virus (RSV) infection, frequently leading to reinfection. We used the BALB/c mouse model to examine the role of the nasal-associated lymphoid tissue and the bone marrow in the generation of RSV-specific long-lasting plasma cells, with a view to further understanding the mechanisms responsible for the poorly sustained RSV antibody levels following primary infection. We show here that substantial numbers of RSV-specific plasma cells were generated in the bone marrow following challenge, which were maintained thereafter. In contrast, in the nasal-associated lymphoid tissue, RSV-specific plasma cell numbers waned quickly both after primary infection and after challenge and were not maintained at a higher level after boosting. These data indicate that the inability to generate a robust local mucosal response in the nasal tissues may contribute substantially to the likelihood of subsequent reinfection and that the presence of serum anti-RSV antibody without local protection is not enough to protect against reinfection.

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