scholarly journals On the role of antidiuretic hormone in the inhibition of acute water diuresis in adrenal insufficiency and the effects of gluco- and mineralocorticoids in reversing the inhibition

1970 ◽  
Vol 49 (9) ◽  
pp. 1724-1736 ◽  
Author(s):  
Howard H. Green ◽  
Avery R. Harrington ◽  
Heinz Valtin
Keyword(s):  
Adrenal Insufficiency ◽  
Antidiuretic Hormone ◽  
Water Diuresis

The Role of IL-1, IL-6, TNF-α and MIF in Relative Adrenal Insufficiency in Septic Shock: A Study in Piglet Model

2018 ◽  
Author(s):  
Bina Akura ◽  
Jose Batubara ◽  
Zakiudin Munasir ◽  
Gunati unknown ◽  
Joedo Prihartono ◽  
...  
Keyword(s):  
Septic Shock ◽  
Adrenal Insufficiency ◽  
Relative Adrenal Insufficiency ◽  
Tnf Α

The role of antidiuretic hormone in cold‐induced diuresis in the anaesthetized rat

1998 ◽  
Vol 162 (4) ◽  
pp. 475-480 ◽  
Author(s):  
BROMAN ◽  
KÄLLSKOG ◽  
NYGREN ◽  
WOLGAST
Keyword(s):  
Antidiuretic Hormone ◽  
Anaesthetized Rat

THE ROLE OF SODIUM IN ADRENAL INSUFFICIENCY

JAMA ◽  
1935 ◽  
Vol 104 (24) ◽  
pp. 2149 ◽  
Author(s):  
ROBERT F. LOEB
Keyword(s):  
Adrenal Insufficiency

Effects of vasopressin administration on diuresis of water immersion in normal humans

1981 ◽  
Vol 51 (6) ◽  
pp. 1384-1387 ◽  
Author(s):  
M. Epstein ◽  
A. G. DeNunzio ◽  
R. D. Loutzenhiser
Keyword(s):  
Antidiuretic Hormone ◽  
Flow Rate ◽  
Water Immersion ◽  
Normal Subjects ◽  
Urinary Flow Rate ◽  
Urinary Flow ◽  
Relative Role ◽  
Normal Humans

Although previous studies have demonstrated that water immersion to the neck (NI) results in a significant diuresis, the mechanisms are incompletely delineated. Because recent studies in our laboratory have demonstrated that NI is associated with a suppression of antidiuretic hormone (ADH), it is possible that such a suppression mediates the encountered diuresis. The present study was undertaken to assess more directly the relative role of ADH suppression by determining the effects of vasopressin administration. Six hydrated normal subjects were studied on two occasions while undergoing 6 h of NI. During the second NI study, aqueous vasopressin (20 mU/h) was infused for the initial 4 h of study (NI + vasopressin). NI resulted in a significant increase in urinary flow rate beginning during hour 1 and persisting throughout NI. In contrast, during NI + vasopressin, the anticipated diuresis was abolished throughout the 4 h of vasopressin administration. Cessation of vasopressin administration during the final 2 h of NI + vasopressin resulted in a marked and prompt diuresis. The present observations are consistent with the formulation that ADH suppression participates importantly in mediating the diuresis of NI in hydrated normal subjects.

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