scholarly journals CLINICAL STUDIES OF THE BLOOD VOLUME. VII. CHANGES IN BLOOD VOLUME IN BRIGHT'S DISEASE WITH OR WITHOUT EDEMA, RENAL INSUFFICIENCY, OR CONGESTIVE HEART FAILURE, AND IN HYPERTENSION 1

1939 ◽  
Vol 18 (5) ◽  
pp. 527-536 ◽  
Author(s):  
Alfred W. Harris ◽  
John G. Gibson
Keyword(s):  
Heart Failure ◽  
Congestive Heart Failure ◽  
Renal Insufficiency ◽  
Blood Volume ◽  
Clinical Studies

The Clinical Studies on Congestive Heart Failure

1979 ◽  
Vol 9 (2) ◽  
pp. 99
Author(s):  
Soon Nam Ahn ◽  
Nan Ho Kyung
Keyword(s):  
Heart Failure ◽  
Congestive Heart Failure ◽  
Clinical Studies

Blood volume and brain natriuretic peptide in congestive heart failure: A pilot study

2005 ◽  
Vol 150 (5) ◽  
pp. 984.e1-984.e6 ◽  
Author(s):  
Karen B. James ◽  
Richard W. Troughton ◽  
Joseph Feldschuh ◽  
Daniel Soltis ◽  
Delsa Thomas ◽  
...  
Keyword(s):  
Heart Failure ◽  
Congestive Heart Failure ◽  
Pilot Study ◽  
Brain Natriuretic Peptide ◽  
Natriuretic Peptide ◽  
Blood Volume

scholarly journals BLOOD VOLUME ALTERATIONS IN CONGESTIVE HEART FAILURE

1954 ◽  
Vol 33 (4) ◽  
pp. 578-586 ◽  
Author(s):  
Sidney S. Schreiber ◽  
Arthur Bauman ◽  
Rosalyn S. Yalow ◽  
Solomon A. Berson
Keyword(s):  
Heart Failure ◽  
Congestive Heart Failure ◽  
Blood Volume

Magnesium Homeostasis and Clinical Disorders of Magnesium Deficiency

1994 ◽  
Vol 28 (2) ◽  
pp. 220-226 ◽  
Author(s):  
Robert Whang ◽  
Edward M. Hampton ◽  
David D. Whang
Keyword(s):  
Myocardial Infarction ◽  
Heart Failure ◽  
Acute Myocardial Infarction ◽  
Congestive Heart Failure ◽  
Critically Ill ◽  
Clinical Studies ◽  
Magnesium Deficiency ◽  
Idiopathic Dilated Cardiomyopathy ◽  
Digitalis Toxicity ◽  
Mg Deficiency

OBJECTIVE: To survey the causes of clinical hypomagnesemia and Mg deficiency. The relationship of hypomagnesemia to digitalis toxicity, congestive heart failure, arrhythmias, and acute myocardial infarction is discussed, as is the clinical interrelationship of Mg and K concentrations, the principal intracellular cations. DATA SOURCES: A MEDLINE search and retrieval was used to identify relevant references. STUDY SELECTION: Clinical reports, as well as studies, were selected for this review. DATA EXTRACTION: There were very few placebo-controlled clinical studies. Clinical observations were related primarily to compilation of series in which Mg was administered and clinical results reported. In addition, conclusions derived from review articles on the subject of clinical Mg depletion were used. DATA SYNTHESIS: Clinical diagnosis of Mg deficiency is ascertained most expeditiously by estimating serum Mg concentrations. Although available on order by physicians, the lack of routine serum Mg analysis as part of the “electrolyte panel” impedes the diagnosis of clinical Mg deficiency. Renal loss of Mg resulting from the widespread use of loop diuretics is responsible for significant numbers of patients with Mg deficiency and hypomagnesemia. Life threatening cardiac arrhythmias and seizures represent the most serious manifestations of clinical hypomagnesemia and Mg depletion. In the most critically ill patients, treatment with intravenous Mg is recommended. Oral repletion of Mg is reserved for the less critically ill hospitalized patients and ambulatory patients. Close attention must be paid to optimizing K replenishment in hypokalemic patients by concurrent treatment of any accompanying hypomagnesemia to avoid the problem of refractory K repletion. CONCLUSIONS: Hypomagnesemia is one of the most frequent serum electrolyte abnormalities in current clinical practice. Routine inclusion of serum Mg analysis in the electrolyte panel will enhance the clinical recognition and treatment of hypomagnesemic Mg-depleted patients. Failure to respond to treatment of recurrent ventricular tachycardia/fibrillation to usual antiarrhythmic therapy in patients with acute myocardial infarction, idiopathic dilated cardiomyopathy, and congestive heart failure should alert the clinician to consider administering intravenous Mg. Repair of coexisting hypomagnesemia in hypokalemic patients is essentialto avoid the problem of refractory K repletion caused by coexisting Mg depletion. More controlled clinical studies of Mg deficiency are necessary to ascertain the cost-effectiveness of Mg replacement therapy.

scholarly journals Pulmonary Blood Volume in Congestive Heart Failure

Circulation ◽  
1966 ◽  
Vol 34 (2) ◽  
pp. 249-259 ◽  
Author(s):  
B. F. SCHREINER ◽  
G. W. MURPHY ◽  
P. N. YU
Keyword(s):  
Heart Failure ◽  
Congestive Heart Failure ◽  
Blood Volume ◽  
Pulmonary Blood Volume

CHLOROTHIAZIDE

PEDIATRICS ◽  
1958 ◽  
Vol 22 (2) ◽  
pp. 236-237
Author(s):  
JOHN D. CRAWFORD
Keyword(s):  
Heart Failure ◽  
Congestive Heart Failure ◽  
Carbonic Anhydrase ◽  
Renal Insufficiency ◽  
Chronic Renal Insufficiency ◽  
Free Water ◽  
Carbonic Anhydrase Inhibitor ◽  
Diuretic Agent

CHLOROTHIAZIDE is a new, orally effective diuretic agent chemically related to acetazolamide. Curiously, it is a considerably less potent carbonic anhydrase inhibitor, at least in vitro and, in vivo, its effect has been found additive to that of acetazolamide as it is to the action of the mercurials. Laragh's observations suggest that chlorothiazide inhibits the process of solute reabsorption which normally gives rise to "free" water in the urine. Thus, it may well have a locus of action in the kidney different from that of its chemical cousin or the mercury derivities. There have been optimistic reports of its efficacy in a variety of edema states including nephrosis, cirrhosis of the liver, congestive heart failure, acute hemorrhagic nephritis and chronic renal insufficiency.

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