Inner Ear Symptoms Are Prevalent in Patients with High Head Abbreviated Injury Scale Scores after Blunt Head Trauma

2021 ◽  
pp. 1-8
Author(s):  
Ji Eun Choi ◽  
Ye Rim Chang ◽  
In-Kwon Mun ◽  
Jae Yun Jung ◽  
Min Young Lee ◽  
...  
Keyword(s):  
Inner Ear ◽  
Head Trauma ◽  
Bed Rest ◽  
Abbreviated Injury Scale ◽  
Temporal Bone Fracture ◽  
Early Evaluation ◽  
Blunt Head Trauma ◽  
Scale Scores ◽  
High Head ◽  
Ear Symptoms

<b><i>Introduction:</i></b> The purpose of this article was to determine the prevalence of inner ear symptoms in patients with blunt head trauma and to explore whether the severity of head trauma was associated with the incidence of such symptoms. <b><i>Methods:</i></b> We performed a retrospective review of 56 patients admitted with blunt head trauma who underwent audiovestibular evaluation within 1 month after injury. Two scales were used to measure the severity of trauma; these were the Glasgow Coma Scale (GCS) and the Head Abbreviated Injury Scale (H-AIS). Patients with sensorineural-type hearing loss, or dizziness with nystagmus, were considered to have inner ear symptoms. <b><i>Results:</i></b> About half of all patients (45%) with blunt head trauma showed trauma-related inner ear symptoms. Patients with inner ear symptoms were significantly more likely to have H-AIS scores ≥4 than those without inner ear symptoms (<i>p</i> = 0.004), even without concomitant temporal bone fracture (<i>p</i> &#x3e; 0.05). Also, patients with inner ear symptoms required a statistically significantly longer time (measured from admission) before undergoing their ontological evaluations than did those without such symptoms (<i>p</i> = 0.002), possibly due to prolonged bed rest and use of sedatives. <b><i>Conclusion:</i></b> Thus, detailed history-taking and early evaluation using trauma scales are essential for all patients suffering from severe head trauma. It may be necessary to initiate early treatment of traumatic inner ear diseases.

scholarly journals KONTUSIO LABIRIN PADA PASCA CEDERA KEPALA

2019 ◽  
Vol 4 (1) ◽  
pp. 32
Author(s):  
Ari Astuti
Keyword(s):  
Traumatic Brain Injury ◽  
Brain Injury ◽  
Temporal Bone ◽  
Head Trauma ◽  
Bone Fracture ◽  
Symptomatic Therapy ◽  
Membranous Labyrinth ◽  
Temporal Bone Fracture ◽  
Blunt Head Trauma ◽  
Post Traumatic

Background : Blunt head trauma without any temporal bone fracture or longitudinal temporal bone fracture, with an associated fracture of the labyrinth may cause labyrinthine injury with hearing loss and vertigo because of a concusive injury to the membranous labyrinth. Objective : To explain a case of labyrinthine concussion in patient with post traumatic brain injury experienced in Dr. Sardjito Hospital Yogyakarta. Case Description : A patient who treated in the hospital with spinning sensation after crush injury. She also had fracture of the left collum femur. Head CT scan and laboratory examination was initiated within normal limit. Patient then diagnosed with labyrinthine concussion and treated with symptomatic therapy and physiotherapy. Conclusion : Labyrinthine concussion can occur to the patient of blunt head trauma. Pharmacotherapy and physiotherapy should be initiated to reduce symptomps and uncomfortable sensation. Keywords : labyrinthine concussion, traumatic brain injury, mixed type vertigo

scholarly journals Contralateral Cochlear Labyrinthine Concussion without Temporal Bone Fracture: Unusual Posttraumatic Consequence

2016 ◽  
Vol 2016 ◽  
pp. 1-4 ◽  
Author(s):  
I. M. Villarreal ◽  
D. Méndez ◽  
J. M. Duque Silva ◽  
P. Ortega del Álamo
Keyword(s):  
Hearing Loss ◽  
Head Injury ◽  
Sensorineural Hearing Loss ◽  
Temporal Bone ◽  
Head Trauma ◽  
Bone Fractures ◽  
Contralateral Side ◽  
Sensorineural Hearing ◽  
Temporal Bone Fracture ◽  
Blunt Head Trauma

Introduction. Labyrinthine concussion is a term used to describe a rare cause of sensorineural hearing loss with or without vestibular symptoms occurring after head trauma. Isolated damage to the inner ear without involving the vestibular organ would be designated as a cochlear labyrinthine concussion. Hearing loss is not a rare finding in head trauma that involves petrous bone fractures. Nevertheless it generally occurs ipsilateral to the side of the head injury and extraordinarily in the contralateral side and moreover without the presence of a fracture.Case Report. The present case describes a 37-year-old patient with sensorineural hearing loss and tinnitus in his right ear after a blunt head trauma of the left-sided temporal bone (contralateral). Otoscopy and radiological images showed no fractures or any abnormalities. A severe sensorineural hearing loss was found in his right ear with a normal hearing of the left side.Conclusion. The temporal bone trauma requires a complete diagnostic battery which includes a neurotologic examination and a high resolution computed tomography scan in the first place. Hearing loss after a head injury extraordinarily occurs in the contralateral side of the trauma as what happened in our case. In addition, the absence of fractures makes this phenomenon even more unusual.

Controversies in the evaluation and management of minor blunt head trauma in children

2007 ◽  
Vol 19 (3) ◽  
pp. 258-264 ◽  
Author(s):  
David Schnadower ◽  
Hector Vazquez ◽  
June Lee ◽  
Peter Dayan ◽  
Cindy Ganis Roskind
Keyword(s):  
Head Trauma ◽  
Blunt Head Trauma

Prevalence and Prognosis of Traumatic Intraventricular Hemorrhage in Patients with Blunt Head Trauma

2006 ◽  
Vol 60 (5) ◽  
pp. 1010-1017 ◽  
Author(s):  
Clare Atzema ◽  
William R. Mower ◽  
Jerome R. Hoffman ◽  
James F. Holmes ◽  
Anthony J. Killian ◽  
...  
Keyword(s):  
Head Trauma ◽  
Intraventricular Hemorrhage ◽  
Blunt Head Trauma

Diffuse Axonal Injury in Infants With Nonaccidental Craniocerebral Trauma

1999 ◽  
Vol 123 (2) ◽  
pp. 146-151 ◽  
Author(s):  
Aaron M. Gleckman ◽  
Michael D. Bell ◽  
Richard J. Evans ◽  
Thomas W. Smith
Keyword(s):  
Head Trauma ◽  
Axonal Injury ◽  
Diffuse Axonal Injury ◽  
Craniocerebral Trauma ◽  
Shaken Baby Syndrome ◽  
Normal Brain ◽  
Accurate Identification ◽  
Blunt Head Trauma ◽  
Hematoxylin Eosin ◽  
Axonal Swellings

Abstract Objective.—Accurate identification of diffuse axonal injury is important in the forensic investigation of infants who have died from traumatic brain injury. β-Amyloid precursor protein (β-APP) immunohistochemical staining is highly sensitive in identifying diffuse axonal injury. However, the effectiveness of this method in brain-injured infants has not been well established. The present study was undertaken to assess the utility of β-APP immunohistochemistry in detecting diffuse axonal injury in infants with either shaken baby syndrome or blunt head trauma. Materials and Methods.—Archival formalin-fixed, paraffin-embedded blocks from infants (&lt;1 year old) with shaken baby syndrome (7 cases) and blunt head trauma (3) and blocks from 7 control cases that included nontraumatic cerebral edema (1), acute hypoxic-ischemic encephalopathy (1), and normal brain (5) were immunostained for β-APP. A semiquantitative assessment of the severity of axonal staining was made. Corresponding hematoxylin-eosin–stained sections were examined for the presence of axonal swellings. Results.—Immunostaining for β-APP identified diffuse axonal injury in 5 of 7 infants with shaken baby syndrome and 2 of 3 infants with blunt head trauma. Immunoreactive axons were easily identified and were present in the majority of the sections examined. By contrast, hematoxylin-eosin staining revealed axonal swellings in only 3 of 7 infants with shaken baby syndrome and 1 of 3 infants with blunt head trauma. Most of these sections had few if any visible axonal swellings, which were often overlooked on initial review of the slides. No β-APP immunoreactivity was observed in any of the 7 control cases. Conclusions.—Immunostaining for β-APP can easily and reliably identify diffuse axonal injury in infants younger than 1 year and is considerably more sensitive than routine hematoxylin-eosin staining. We recommend its use in the forensic evaluation of infants with fatal craniocerebral trauma.

Olfactory dysfunction: A sequela of pediatric blunt head trauma

2006 ◽  
Vol 70 (6) ◽  
pp. 1015-1025 ◽  
Author(s):  
Amanda A. Sandford ◽  
Terence M. Davidson ◽  
Norma Herrera ◽  
Paul Gilbert ◽  
Anthony E. Magit ◽  
...  
Keyword(s):  
Head Trauma ◽  
Olfactory Dysfunction ◽  
Blunt Head Trauma

scholarly journals Initial clinical presentation of children with acute and chronic versus acute subdural hemorrhage resulting from abusive head trauma

2015 ◽  
Vol 16 (2) ◽  
pp. 177-185 ◽  
Author(s):  
Kenneth W. Feldman ◽  
Naomi F. Sugar ◽  
Samuel R. Browd
Keyword(s):  
Head Trauma ◽  
Clinical Presentation ◽  
Abusive Head Trauma ◽  
Group Differences ◽  
Similar Proportion ◽  
Subdural Hemorrhage ◽  
Retinal Hemorrhages ◽  
Clinical Presentations ◽  
Scale Scores ◽  
Multicenter Retrospective Review

OBJECT At presentation, children who have experienced abusive head trauma (AHT) often have subdural hemorrhage (SDH) that is acute, chronic, or both. Controversy exists whether the acute SDH associated with chronic SDH results from trauma or from spontaneous rebleeding. The authors compared the clinical presentations of children with AHT and acute SDH with those having acute and chronic SDH (acute/chronic SDH). METHODS The study was a multicenter retrospective review of children who had experienced AHT during 2004–2009. The authors compared the clinical and radiological characteristics of children with acute SDH to those of children with acute/chronic SDH. RESULTS The study included 383 children with AHT and either acute SDH (n = 291) or acute/chronic SDH (n = 92). The children with acute/chronic SDH were younger, had higher initial Glasgow Coma Scale scores, fewer deaths, fewer skull fractures, less parenchymal brain injury, and fewer acute noncranial fractures than did children with acute SDH. No between-group differences were found for the proportion with retinal hemorrhages, healing noncranial fractures, or acute abusive bruises. A similar proportion (approximately 80%) of children with acute/chronic SDH and with acute SDH had retinal hemorrhages or acute or healing extracranial injures. Of children with acute/chronic SDH, 20% were neurologically asymptomatic at presentation; almost half of these children were seen for macrocephaly, and for all of them, the acute SDH was completely within the area of the chronic SDH. CONCLUSIONS Overall, the presenting clinical and radiological characteristics of children with acute SDH and acute/chronic SDH caused by AHT did not differ, suggesting that repeated abuse, rather than spontaneous rebleeding, is the etiology of most acute SDH in children with chronic SDH. However, more severe neurological symptoms were more common among children with acute SDH. Children with acute/chronic SDH and asymptomatic macrocephaly have unique risks and distinct radiological and clinical characteristics.

Sign in / Sign up

Export Citation Format

Share Document