Lamellar Corneal Grafting with Autologous and Homologous Tissue

Author(s):  
J. Draeger ◽  
M. Kohlhaas
2017 ◽  
Vol 10 (3) ◽  
pp. 93-100 ◽  
Author(s):  
E.V. Chentsova ◽  
◽  
E.N. Verigo ◽  
P.V. Makarov ◽  
A.I. Khazamova ◽  
...  

2016 ◽  
Vol 100 (3) ◽  
pp. 481-482 ◽  
Author(s):  
Geoff Y. Zhang ◽  
Min Hu ◽  
Yuan Min Wang ◽  
Stephen I. Alexander
Keyword(s):  
Low Dose ◽  

2014 ◽  
Vol 2014 ◽  
pp. 1-5 ◽  
Author(s):  
Deepa Viswanathan ◽  
Nikhil L. Kumar ◽  
John J. Males

Purpose. To evaluate the efficacy of corneal collagen crosslinking for progressive keratoconus in paediatric patients.Methods. This prospective study included 25 eyes of 18 patients (aged 18 years or younger) who underwent collagen crosslinking performed using riboflavin and ultraviolet-A irradiation (370 nm, 3 mW/cm2, 30 min).Results. The mean patient age was 14.3 ± 2.4 years (range 8–17) and mean followup duration was 20.1 ± 14.25 months (range 6–48). Crosslinked eyes demonstrated a significant reduction of keratometry values. The mean baseline simulated keratometry values were 46.34 dioptres (D) in the flattest meridian and 50.06 D in the steepest meridian. At 20 months after crosslinking, the values were 45.67 D (P=0.03) and 49.34 D (P=0.005), respectively. The best spectacle corrected visual acuity (BSCVA) and topometric astigmatism improved after crosslinking. Mean logarithm of the minimum angle of resolution (logMAR) BSCVA decreased from 0.24 to 0.21 (P=0.89) and topometric astigmatism reduced from mean 3.50 D to 3.25 D (P=0.51).Conclusions. Collagen crosslinking using riboflavin and ultraviolet-A is an effective treatment option for progressive keratoconus in paediatric patients. Crosslinking stabilises the condition and, thus, reduces the need for corneal grafting in these young patients.


1989 ◽  
Vol 34 (1) ◽  
pp. 5-8 ◽  
Author(s):  
I. Baumgartner ◽  
W. R. Mayr ◽  
G. Grabner
Keyword(s):  

1972 ◽  
Vol 74 (6) ◽  
pp. 1222
Author(s):  
Claes H. Dohlman
Keyword(s):  

1996 ◽  
Vol 17 (8) ◽  
pp. 521-528
Author(s):  
Dominique Dormont

AbstractTransmissible spongiform encephalopathies are rare lethal diseases induced in humans and animals by unconventional agents called transmissible spongiform encephalopathy agents (TSEAs), virions, or prions. Several cases of iatrogenic Creutzfeldt-Jakob disease (CJD) have been reported in the literature after neuro-surgery, treatment with pituitary-derived hormones, corneal grafting, and use of dura mater lyophilisates. In a given infected individual, TSEA-associated infectiousness depends on the nature of the organ: the central nervous system has the highest infectiousness, spleen and lymph nodes a medium infectiousness, and organs such as bone, skin, or skeletal muscles do not harbor any detectable infectiousness in experimental models. Transmissible spongiform encephalopathy/prions have unconventional properties; in particular, they resist almost all the chemical and physical processes that inactivate conventional viruses. Therefore, prevention of CJD agent transmission must be taken into account in daily hospital practice. Efficient sterilization procedures should be determined. In tissue and blood donation, donors with a neurologic history must be excluded, and patients treated with pituitary-derived hormones should be considered potentially infected with TSEA and excluded.


The Lancet ◽  
1943 ◽  
Vol 242 (6267) ◽  
pp. 453
Keyword(s):  

1983 ◽  
Vol 95 (5) ◽  
pp. 711-712 ◽  
Author(s):  
David M. Meisler ◽  
David M. Meisler ◽  
Z. Nicholas Zakov ◽  
Roger H.S. Langston

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