Primary Position Upbeat Nystagmus in Man and Cats

Author(s):  
Koji Harada ◽  
Isao Kato ◽  
Yoshio Koike ◽  
Yo Kimura ◽  
Tadashi Nakamura ◽  
...  
1991 ◽  
Vol 1991 (Supplement48) ◽  
pp. 128-132
Author(s):  
Atsushi Sakuma ◽  
Isao Kato ◽  
Sadao Ogino ◽  
Tomoyuki Okada ◽  
Isamu Takeyama

Neurology ◽  
1977 ◽  
Vol 27 (3) ◽  
pp. 294-294 ◽  
Author(s):  
N. GILMAN ◽  
R. W. BALOH ◽  
U. TOMIYASU

1989 ◽  
Vol 82 (4) ◽  
pp. 543-553
Author(s):  
Hiromitsu Saito ◽  
Tsunemasa Sato

2021 ◽  
Vol 238 (11) ◽  
pp. 1186-1195
Author(s):  
Michael Leo Strupp ◽  
Dominik Straumann ◽  
Christoph Helmchen

AbstractNystagmus is defined as rhythmic, most often involuntary eye movements. It normally consists of a slow (pathological) drift of the eyes, followed by a fast central compensatory movement back to the primary position (refixation saccade). The direction, however, is reported according to the fast phase. The cardinal symptoms are, on the one hand, blurred vision, jumping images (oscillopsia), reduced visual acuity and, sometimes, double vision; many of these symptoms depend on the eye position. On the other hand, depending on the etiology, patients may suffer from the following symptoms: 1. permanent dizziness, postural imbalance, and gait disorder (typical of downbeat and upbeat nystagmus); 2. if the onset of symptoms is acute, the patient may experience spinning vertigo with a tendency to fall to one side (due to ischemia in the area of the brainstem or cerebellum with central fixation nystagmus or as acute unilateral vestibulopathy with spontaneous peripheral vestibular nystagmus); or 3. positional vertigo. There are two major categories: the first is spontaneous nystagmus, i.e., nystagmus which occurs in the primary position as upbeat or downbeat nystagmus; and the second includes various types of nystagmus which are induced or modified by certain factors. Examples are gaze-evoked nystagmus, head-shaking nystagmus, positional nystagmus, and hyperventilation-induced nystagmus. In addition, there are disorders similar to nystagmus, such as ocular flutter or opsoclonus. The most common central types of spontaneous nystagmus are downbeat and upbeat, infantile, pure torsional, pendular fixation, periodic alternating, and seesaw nystagmus. Many types of nystagmus allow a precise neuroanatomical localization: for instance, downbeat nystagmus, which is most often caused by a bilateral floccular lesion or dysfunction, or upbeat nystagmus, which is caused by a lesion in the midbrain or medulla. Examples of drug treatment are the use of 4-aminopyridine for downbeat and upbeat nystagmus, memantine or gabapentin for pendular fixation nystagmus, or baclofen for periodic alternating nystagmus. In this article we are focusing on nystagmus. In a second article we will focus on central ocular motor disorders, such as saccade or gaze palsy, internuclear ophthalmoplegia, and gaze-holding deficits. Therefore, these types of eye movements will not be described here in detail.


2012 ◽  
Vol 19 (1) ◽  
pp. 161-162 ◽  
Author(s):  
Ivan Adamec ◽  
Tereza Gabelić ◽  
Magdalena Krbot ◽  
David Ozretić ◽  
Iva Milivojević ◽  
...  

Eye ◽  
2004 ◽  
Vol 18 (1) ◽  
pp. 106-106 ◽  
Author(s):  
S F Osborne ◽  
A J Vivian

1987 ◽  
Vol 21 (1) ◽  
pp. 53-58 ◽  
Author(s):  
Patrick A. Sibony ◽  
Craig Evinger ◽  
Karen A. Manning

1996 ◽  
Vol 3 (3) ◽  
pp. 260-263 ◽  
Author(s):  
A. Iwata ◽  
F. Takao ◽  
M. Kunimoto ◽  
K. Inoue

1994 ◽  
Vol 87 (3) ◽  
pp. 301-315
Author(s):  
Hiromitsu SAITO ◽  
Tsunemasa SATO ◽  
Shigeyo NAGASE ◽  
Hiroyuki FUJITA ◽  
Soutaro FUNASAKA

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