Oxygen Poisoning

Journal of Biological Chemistry ◽

10.1016/s0021-9258(18)43066-9 ◽

1946 ◽

Vol 164 (1) ◽

pp. 257-263

Author(s):

William C. Stadie ◽

Niels Haugaard

Keyword(s):

Oxygen Poisoning

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OXYGEN POISONING

Journal of Biological Chemistry ◽

10.1016/s0021-9258(17)41532-8 ◽

1945 ◽

Vol 161 (1) ◽

pp. 153-174 ◽

Cited By ~ 7

Author(s):

William C. Stadie ◽

Niels Haugaard

Keyword(s):

Oxygen Poisoning

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Oxygen Poisoning in a Hyperbaric Environment

Journal of The Royal Naval Medical Service ◽

10.1136/jrnms-56-39 ◽

1970 ◽

Vol 56 (1) ◽

pp. 39-47

Author(s):

J. Murray Young

Keyword(s):

Partial Pressure ◽

Sea Water ◽

Signs And Symptoms ◽

Pressure Chamber ◽

Partial Pressure Of Oxygen ◽

Oxygen Exposure ◽

Rapid Decompression ◽

Hyperbaric Environment ◽

Biochemical Mechanisms ◽

Oxygen Poisoning

AbstractThe biochemical mechanisms of oxygen poisoning are discussed together with the signs and symptoms produced clinically. Investigation of 35 subjects performing work (mean 02 consumption 1.4 l./min) for 40 minutes in a pressure chamber at simulated depths of 20 to 47 feet of sea water showed that this degree of exercise markedly reduced the oxygen exposure tolerance of the subjects. This investigation also showed that rapid decompression of subjects breathing oxygen can produce an exacerbation of existing symptoms and these results are discussed.It is stressed that any breathing mixture containing a partial pressure of oxygen in excess of 150 torr constitutes an environment which is hyperbaric with respect to oxygen and caution is advised in the administration of oxygen to any patient.

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Oxygen poisoning of diamond film growth

Applied Physics Letters ◽

10.1063/1.110767 ◽

1993 ◽

Vol 63 (19) ◽

pp. 2641-2643 ◽

Cited By ~ 11

Author(s):

William N. Howard ◽

Karl E. Spear ◽

Michael Frenklach

Keyword(s):

Diamond Film ◽

Film Growth ◽

Oxygen Poisoning ◽

Diamond Film Growth

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Oxygen poisoning of NAD biosynthesis: A proposed site of cellular oxygen toxicity

Biochemical and Biophysical Research Communications ◽

10.1016/0006-291x(79)91976-4 ◽

1979 ◽

Vol 91 (3) ◽

pp. 982-990 ◽

Cited By ~ 17

Author(s):

Olen R. Brown ◽

Frederick Yein ◽

Daniel Boehme ◽

Laurie Foudin ◽

Cheng Shu Song

Keyword(s):

Oxygen Toxicity ◽

Nad Biosynthesis ◽

Cellular Oxygen ◽

Oxygen Poisoning

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Oxygen Poisoning in Insects1

Annals of the Entomological Society of America ◽

10.1093/aesa/52.6.637 ◽

1959 ◽

Vol 52 (6) ◽

pp. 637-640 ◽

Cited By ~ 5

Author(s):

Arnold M. Clark

Keyword(s):

Oxygen Poisoning

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Studies in the respiratory and carbohydrate metabolism of plant tissues. VII. Experimental studies with potato tubers of an inhibition of the respiration and of a ‘block’ in the tricarboxylic acid cycle induced by ‘oxygen poisoning’

Proceedings of the Royal Society of London Series B Biological Sciences ◽

10.1098/rspb.1955.0027 ◽

1955 ◽

Vol 143 (913) ◽

pp. 523-549 ◽

Cited By ~ 23

Keyword(s):

Tricarboxylic Acid Cycle ◽

Experimental Studies ◽

General Term ◽

Tricarboxylic Acid ◽

Pure Oxygen ◽

Prolonged Treatment ◽

Potato Tubers ◽

Acid Cycle ◽

Oxidation Reduction ◽

Oxygen Poisoning

Prolonged treatment of potato tubers at 1° C with an atmosphere of pure oxygen eventually induces a marked inhibition of the rate of CO 2 output; there is also an accumulation of pyruvate and of 'citrate’ and a decrease in the contents of α -ketoglutarate and of malate as compared with potatoes held in air. These changes in the acids appear to be in accord with the development during sojourn in pure oxygen of a ‘block’ in the tricarboxylic acid cycle between ‘citrate’ and α -ketoglutarate. The indications in previous work (Barron, Link, Klein & Michel 1950; Barker & Mapson 1953 b ) that the tricarboxylic acid cycle may operate in potato tubers under certain metabolic conditions are thus supported. The treatment with pure oxygen also results in a progressive shift to the more oxidized state in the ascorbic acid and glutathione oxidation-reduction systems; finally, the potato tissue develops a brown discoloration presumably due to polyphenolase action. The change in the balance of the two oxidation-reduction systems towards oxidation may be caused, in part, by a reduced rate of regeneration of coenzyme II because of the ‘block’ in the tricarboxylic acid cycle. The paper also contains the results of preliminary experiments on the reversibility of the above changes. The data add to the knowledge of the varied metabolic phenomena which have been observed in many different types of living tissue, both plant and animal, and which are conveniently classified under the general term ‘oxygen poisoning’ (Stadie, Riggs & Haugaard 1944).

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