Effects of Intracranial Implantation of Ovarian and Adrenal Steroids and Hypothalamic Deafferentation in Newborn Female Rats on Onset of Biological Rhythmicity1

Author(s):  
Shinji Hayashi ◽  
Eiichi Ooya ◽  
Susumu Miyabo
1977 ◽  
Vol 73 (1) ◽  
pp. 165-170 ◽  
Author(s):  
G. FINK ◽  
S. R. HENDERSON

SUMMARY The effect of oestrogen and progesterone on the spontaneous release of gonadotrophins and on the gonadotrophin response to luteinizing hormone releasing factor has been examined in female rats subjected to hypothalamic deafferentation or pituitary stalk section. The results indicate that both steroids act at the level of the medial basal hypothalamus as well as the anterior pituitary gland; progesterone, for its full effect, also requires the integrity of connexions of the hypothalamus with other areas of the brain.


1980 ◽  
Vol 86 (1) ◽  
pp. 69-77 ◽  
Author(s):  
AGNETA MODE ◽  
PETER ENEROTH ◽  
JAN-ÅKE GUSTAFSSON ◽  
TOMAS HÖKFELT ◽  
PAUL SKETT

The metabolism of 4-[4-14C]androstene-3,17-dione in the microsomal fraction of livers from male and female rats was investigated after hypothalamic deafferentation at two levels. It was found that frontal deafferentation at the retrochiasmatic level caused a complete 'feminization' of hepatic steroid metabolism in the male rat but was without effect in the female animal. Transection rostral to the suprachiasmatic nuclei was without effect in both sexes. A complete transition from male to female hepatic steroid metabolism after retrochiasmatic deafferentation was reached on day 4 after the operation and persisted for at least 10 weeks. The present results, taken together with previous investigations, indicate that the release of a 'feminizing' factor from the pituitary gland of the male rat is inhibited by a factor produced in, or transported through, the periventricular anterior hypothalamic region including the suprachiasmatic area. No effect on the hepatic steroid metabolism was observed after blinding of the rats suggesting that a diurnal rhythm is not essential to this control mechanism.


1958 ◽  
Vol 195 (2) ◽  
pp. 373-380 ◽  
Author(s):  
F. Eugene Yates ◽  
John Urquhart ◽  
Arthur L. Herbst

Triiodothyronine increases total hepatic capacity for in vitro reduction of ring A of cortisone by 38% in both male and female rats. Thyro-parathyroidectomy decreases it 56% in males and 48% in females. These alterations in thyroid state influence hepatic reduction of corticosteroids in several ways: a) the total amount of Δ4-steroid hydrogenases in the liver may be altered, both by changes in liver size and by changes in amount of enzyme per gram of liver; and, b) the activity per gram of liver can also be altered through variations in coenzyme (TPNH) availability. These effects of thyroid hormones provide an enzymatic basis for the alterations in biological half-life of adrenal steroids observed in hyper- and hypothyroidism. A very close correlation ( r = 0.97) between total hepatic capacity to inactivate cortisone and the size of the adrenal glands was found. It is suggested that the rate of ring A reduction of corticosteroids by liver determines the rate of ACTH secretion in unstressed animals.


1978 ◽  
Vol 89 (3) ◽  
pp. 417-424 ◽  
Author(s):  
H. Kato ◽  
M. E. Velasco ◽  
I. Rothchild

ABSTRACT To investigate where oestrogens act to induce an increase in the tonic release of prolactin, the effect of ovariectomy alone or of ovariectomy plus oestradiol benzoate treatment (10 μg/day for 7 days) (OeB10) on the morning prolactin serum level in adult female rats was determined after each of the following procedures: frontal hypothalamic deafferentation (FHD) (a semi-lunar cut at the caudal border of the optic chiasma, separating the anterior hypothalamus from the preoptic area); medial hypothalamic deafferentation (MHD) (a similar cut, but separating the anterior from the posterior half of the hypothalamus); sham deafferentation; an electrolytic lesion of the arcuate-median eminence area (Arcuate-ME lesion); hypophysectomy and pituitary autotransplantation beneath the kidney capsule. The rats were ovariectomized 1 week after the latter procedure, or 3 weeks after the others, and OeB10 or oil vehicle treatment was begun one week after ovariectomy. FHD, the Arcuate-ME lesion and pituitary autotransplantation markedly increased the basal prolactin level; MHD induced a much smaller increase. The level fell after ovariectomy only in the FHD group. A sharp and equal increase in the prolactin level occurred in the FHD, MHD and sham operation groups in response to OeB10; a much smaller and more variable increase occurred in the Arcuate-ME and pituitary autotransplantation groups. The results suggest that oestrogens increase the tonic release of prolactin only in part by a direct action on the pituitary, and more importantly by acting on the posterior half of the hypothalamus or on areas with lateral, superior, or posterior afferent connections to this site.


Author(s):  
I. Russo ◽  
J. Saby ◽  
J. Russo

It has been previously demonstrated that DMBA-induced rat mammary carcinoma originates in the terminal end bud (TEB) of the mammary gland by proliferation of intermediate type cells (1). The earliest lesion identified is the intraductal proliferation (IDP), which gives rise to intraductal carcinomas. These evolve to cribriform, papillary and comedo types (2). In the present work, we report the ultrastructural changes that take place in the IDP for the formation of a cribriform pattern.Fifty-five-day-old Sprague Dawley virgin female rats were inoculated intra- gastrically with 20 mg 7,12-dimethylbenz(a)anthracene (DMBA) in 1 ml sesame oil. Non-inoculated, age-matched females were used as controls. Mammary glands from both control and experimental rats were removed weekly from the time of inoculation until 86 days post-inoculation. The glands were fixed and processed for electron microscopy (2).The first change observed in IDP's was the widening of intercellular spaces and the secretion of an electron dense material into these spaces (Fig. 1).


Author(s):  
P. Bagavandoss ◽  
JoAnne S. Richards ◽  
A. Rees Midgley

During follicular development in the mammalian ovary, several functional changes occur in the granulosa cells in response to steroid hormones and gonadotropins (1,2). In particular, marked changes in the content of membrane-associated receptors for the gonadotropins have been observed (1).We report here scanning electron microscope observations of morphological changes that occur on the granulosa cell surface in response to the administration of estradiol, human follicle stimulating hormone (hFSH), and human chorionic gonadotropin (hCG).Immature female rats that were hypophysectcmized on day 24 of age were treated in the following manner. Group 1: control groups were injected once a day with 0.1 ml phosphate buffered saline (PBS) for 3 days; group 2: estradiol (1.5 mg/0.2 ml propylene glycol) once a day for 3 days; group 3: estradiol for 3 days followed by 2 days of hFSH (1 μg/0.1 ml) twice daily, group 4: same as in group 3; group 5: same as in group 3 with a final injection of hCG (5 IU/0.1 ml) on the fifth day.


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