Post-Traumatic Acute Renal Failure: Clinical Experience after the Earthquake in Southern Italy

2015 ◽  
pp. 219-239
Author(s):  
V. E. Andreucci ◽  
A. Dal Canton ◽  
M. Usberti ◽  
S. Federico ◽  
V. Calderaro ◽  
...  
1996 ◽  
Vol 7 (11) ◽  
pp. 2320-2326 ◽  
Author(s):  
A M Alkhunaizi ◽  
L Chan

Oxalosis, or calcium oxalate deposition in the tissues, may develop in patients with inherited disorders of oxalate metabolism or can occur secondary to other diseases. In this study, a case of renal oxalosis probably secondary to excessive parenteral vitamin C administration in a patient with acute post-traumatic oliguric renal failure is reported. Oxalate deposits may have contributed to further worsening and delayed recovery of renal function. The elimination of the source of excess vitamin C and its presumed effect on oxalate production, together with enhanced removal of oxalate during aggressive dialysis, resulted in prompt recovery of renal function. Secondary oxalosis represents a possible cause of delayed recovery of renal function in patients with acute renal failure who are receiving vitamin C supplementation if excess dosage of that supplementation is given. Vitamin C supplementation, if utilized, should be carefully monitored in patients receiving artificial renal replacement therapy.


1974 ◽  
Vol 111 (4) ◽  
pp. 425-428 ◽  
Author(s):  
Gerald W. Irelad ◽  
Alexander S. Cass

1976 ◽  
Vol 11 (2) ◽  
pp. 213
Author(s):  
Chung Nam Kang ◽  
Ki Hong Choi ◽  
Jong Hee Kim ◽  
Kang Hong Jung

Blood ◽  
1969 ◽  
Vol 33 (2) ◽  
pp. 234-239 ◽  
Author(s):  
PHILLIP E. HOFFSTEN ◽  
HUGH CHAPLIN

Abstract An acute hemolytic transfusion reaction due to ABO-incompatibility occurred in a patient during the fifth week of oliguria secondary to post-traumatic acute renal failure. Resort to "plasma-exchange" transfusion to reduce the high level of free hemoglobin in the circulation was considered and rejected. Sequential measurements of free and haptoglobin-bound hemoglobin documented rapid disappearance of both components from the patient’s plasma in the absence of hemoglobinuria. Within one week, the patient entered the diuretic phase, and ultimately stabilized at normal levels of urine volume, with a creatinine clearance of 53 ml per minute. The implications of free hemoglobinemia in the oliguric phase of acute renal failure are discussed.


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