Behavioral Effects of Prenatal Exposure to Opiates1

2015 ◽  
pp. 159-168
Author(s):  
I. S. Zagon ◽  
P. J. McLaughlin
Keyword(s):  
Prenatal Exposure ◽  
Behavioral Effects

Prenatal exposure to dichlorvos: physical and behavioral effects on rat offspring

2004 ◽  
Vol 26 (4) ◽  
pp. 607-614 ◽  
Author(s):  
C.A Lazarini ◽  
R.Y Lima ◽  
A.P Guedes ◽  
M.M Bernardi
Keyword(s):  
Prenatal Exposure ◽  
Behavioral Effects ◽  
Rat Offspring

Behavioral effects of prenatal exposure to lidocaine in the rat: Effects of dosage and of gestational age at administration

1989 ◽  
Vol 11 (4) ◽  
pp. 395-403 ◽  
Author(s):  
Robert F. Smith ◽  
Maura F. Kurkjian ◽  
Kathleen M. Mattran ◽  
Steven L. Kurtz
Keyword(s):  
Prenatal Exposure ◽  
Gestational Age ◽  
Behavioral Effects

Behavioral effects of prenatal exposure to caffeine in rats

Life Sciences ◽  
1986 ◽  
Vol 38 (10) ◽  
pp. 861-868 ◽  
Author(s):  
R.N. Hughes ◽  
I.J. Beveridge
Keyword(s):  
Prenatal Exposure ◽  
Behavioral Effects

Prenatal testosterone exposure, left-handedness, and high school delinquency

1998 ◽  
Vol 21 (3) ◽  
pp. 369-370 ◽  
Author(s):  
Stanley Coren
Keyword(s):  
High School ◽  
Antisocial Behavior ◽  
Prenatal Exposure ◽  
Indirect Evidence ◽  
Behavioral Effects ◽  
Prenatal Testosterone ◽  
Left Handedness

Prenatal exposure to high levels of testosterone may lead to increased probability of left-handedness. Extrapolating from arguments by Mazur & Booth leads to a prediction of increased incidence of antisocial behavior among left-handers. Six hundred ninety-four males were tested for seven indicators of delinquency in high school. Left-handers were more likely to display such behaviors, providing indirect evidence for the hypothesized behavioral effects of testosterone.

Behavioral effects of prenatal exposure to pulsed-wave ultrasound in unanesthetized rats

Teratology ◽  
1996 ◽  
Vol 54 (2) ◽  
pp. 65-72 ◽  
Author(s):  
J. Edward Fisher ◽  
Karen D. Acuff-Smith ◽  
Mary A. Schilling ◽  
Richard A. Meyer ◽  
Nadine B. Smith ◽  
...  
Keyword(s):  
Prenatal Exposure ◽  
Behavioral Effects

Reversal of cardiomyopathy resulting from prenatal exposure to ethanol

1984 ◽  
Vol 42 ◽  
pp. 716-717
Author(s):  
C. Uphoff ◽  
C. Nyquist-Battie
Keyword(s):  
Prenatal Exposure ◽  
Heart Development ◽  
Membrane Integrity ◽  
Ethanol Exposure ◽  
Prenatal Ethanol Exposure ◽  
Pathological Changes ◽  
Prenatally Exposed ◽  
Inner Mitochondrial Membrane ◽  
Fetal Alcohol ◽  
Newborn Mice

Fetal Alcohol Syndrone (FAS) is a syndrome with characteristic abnormalities resulting from prenatal exposure to ethanol. In many children with FAS syndrome gross pathological changes in the heart are seen with septal defects the most prevalent abnormality recorded. Few studies in animal models have been performed on the effects of ethanol on heart development. In our laboratory, it has been observed that prenatal ethanol exposure of Swiss albino mice results in abnormal cardiac muscle ultrastructure when mice were examined at birth and compared to pairfed and normal controls. Fig. 1 is an example of the changes that are seen in the ethanol-exposed animals. These changes include enlarged mitochondria with loss of inner mitochondrial membrane integrity and loss of myofibrils. Morphometric analysis substantiated the presence of these alterations from normal cardiac ultrastructure. The present work was undertaken to determine if the pathological changes seen in the newborn mice prenatally exposed to ethanol could be reversed with age and abstinence.

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