Chemical Diabetes in Children with Idiopathic Hypoglycemia of Infancy and Idiopathic Glucagon Unresponsive (Ketotic) Hypoglycemia

2015 ◽  
pp. 94-96
Author(s):  
A. L. Rosenbloom
Keyword(s):  
Chemical Diabetes ◽  
Ketotic Hypoglycemia

Surface epithelial ultrastructure of the polycystic ovary in the BB wistar rat

1986 ◽  
Vol 44 ◽  
pp. 252-253
Author(s):  
R. C. Kaufmann ◽  
F. K. Khosho ◽  
K. S. Amankwah
Keyword(s):  
Electron Microscopy ◽  
Scanning Electron Microscopy ◽  
Wistar Rats ◽  
Polycystic Ovary ◽  
Wistar Rat ◽  
Polycystic Ovaries ◽  
Chemical Diabetes ◽  
Scanning Electron

Diabetes decreases the fertility of females, but the mechanisms are not completely understood. In our investigations, we have found that 13% of the female BB Wistar rats that spontaneously developed chemical diabetes had persistent estrous. In this study the ovaries of these rats were examined by scanning electron microscopy(SEM) and compared to normal-cycling controls as well as to rats that had developed polycystic ovaries(PCO) by exposure to constant 1ight.

Case Report: Liver Glycogen Synthase Deficiency A Cause of Ketotic Hypoglycemia

PEDIATRICS ◽  
2001 ◽  
Vol 108 (2) ◽  
pp. 495-497 ◽  
Author(s):  
S. L. Rutledge ◽  
J. Atchison ◽  
N. U. Bosshard ◽  
B. Steinmann
Keyword(s):  
Case Report ◽  
Glycogen Synthase ◽  
Liver Glycogen ◽  
Ketotic Hypoglycemia

Chemical diabetes in childhood. Integrated concentrations of glucose, insulin, and growth hormone

Diabetes ◽  
1978 ◽  
Vol 27 (9) ◽  
pp. 909-915 ◽  
Author(s):  
R. J. Winter ◽  
L. P. Plotnick ◽  
R. G. Thompson
Keyword(s):  
Growth Hormone ◽  
Chemical Diabetes ◽  
Diabetes In Childhood

Ketotic hypoglycemia in the Penta X and other chromosome imbalance syndromes

2008 ◽  
Vol 14 (6) ◽  
pp. 367-369 ◽  
Author(s):  
Sri Moedjono ◽  
William S. Sly ◽  
Lawrence B. Mauldin ◽  
Morey Haymond
Keyword(s):  
Ketotic Hypoglycemia ◽  
Chromosome Imbalance

Insulin reserve in children with chemical diabetes

1969 ◽  
Vol 74 (5) ◽  
pp. 831-832
Author(s):  
D.Y.N. Murthy ◽  
R.A. Guthrie ◽  
W.N. Womack ◽  
R.L. Jackson
Keyword(s):  
Chemical Diabetes

Chemical diabetes in the juvenile patient

Metabolism ◽  
1973 ◽  
Vol 22 (2) ◽  
pp. 345-349 ◽  
Author(s):  
Eleanor Colle ◽  
Mimi M. Belmonte
Keyword(s):  
Chemical Diabetes ◽  
Juvenile Patient

Cutaneous Microangiopathy in Prediabetes, Chemical Diabetes, and Overt Diabetes

1976 ◽  
pp. 300-301
Author(s):  
L. R. Rouen ◽  
R. A. Camerini-Davalos ◽  
E. N. Terry ◽  
W. Redisch
Keyword(s):  
Chemical Diabetes ◽  
Overt Diabetes ◽  
Cutaneous Microangiopathy

scholarly journals Glutaric Acidemia, Pathogenesis and Nutritional Therapy

2021 ◽  
Vol 8 ◽  
Author(s):  
Qian Li ◽  
Chunlan Yang ◽  
Lijuan Feng ◽  
Yazi Zhao ◽  
Yong Su ◽  
...  
Keyword(s):  
Management Practices ◽  
Genetic Diseases ◽  
Management Strategies ◽  
Nutritional Therapy ◽  
Nutrition Therapy ◽  
Neurological Injury ◽  
Type I ◽  
Type Ii ◽  
Recessive Mutations ◽  
Ketotic Hypoglycemia

Glutaric acidemia (GA) are heterogeneous, genetic diseases that present with specific catabolic deficiencies of amino acid or fatty acid metabolism. The disorders can be divided into type I and type II by the occurrence of different types of recessive mutations of autosomal, metabolically important genes. Patients of glutaric acidemia type I (GA-I) if not diagnosed very early in infanthood, experience irreversible neurological injury during an encephalopathic crisis in childhood. If diagnosed early the disorder can be treated successfully with a combined metabolic treatment course that includes early catabolic emergency treatment and long-term maintenance nutrition therapy. Glutaric acidemia type II (GA- II) patients can present clinically with hepatomegaly, non-ketotic hypoglycemia, metabolic acidosis, hypotonia, and in neonatal onset cardiomyopathy. Furthermore, it features adult-onset muscle-related symptoms, including weakness, fatigue, and myalgia. An early diagnosis is crucial, as both types can be managed by simple nutraceutical supplementation. This review discusses the pathogenesis of GA and its nutritional management practices, and aims to promote understanding and management of GA. We will provide a detailed summary of current clinical management strategies of the glutaric academia disorders and highlight issues of nutrition therapy principles in emergency settings and outline some specific cases.

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