Effects of High-Sucrose and High-Fructose Diets on Non-Obese and Congenitally Obese Hyperglycemic Bar Harbor Mice1

2015 ◽  
pp. 83-91
Author(s):  
J. Winand ◽  
J. Christophe
Keyword(s):  
High Fructose ◽  
High Sucrose

scholarly journals Eucalyptus leaf extract inhibits intestinal fructose absorption, and suppresses adiposity due to dietary sucrose in rats

2005 ◽  
Vol 93 (6) ◽  
pp. 957-963 ◽  
Author(s):  
Keiichiro Sugimoto ◽  
Junko Suzuki ◽  
Kazuya Nakagawa ◽  
Shuichi Hayashi ◽  
Toshiki Enomoto ◽  
...  
Keyword(s):  
Eucalyptus Globulus ◽  
Leaf Extract ◽  
Elevated Concentration ◽  
Dependent Manner ◽  
High Fructose ◽  
Sucrose Diet ◽  
High Sucrose Diet ◽  
Hepatic Triacylglycerol ◽  
Dose Dependent ◽  
High Sucrose

Sucrose is more lipogenic than starch, and the extreme ingestion of sucrose induces adiposity and obesity. The aim of this study was to examine the effect of the eucalyptus (Eucalyptus globulus) leaf extract (ELE) on adiposity due to dietary sucrose in rats. In addition, in this study, the effect of ELE on intestinal fructose absorption was also examined. Rats were fed a high-sucrose diet (75 % in calorie base) with or without ELE (10 g/kg diet) for 5 weeks. Body weight was lower in the rats receiving ELE than in the controls (342 (sd 37·9)v. 392 (sd 26·0) g (n7);P<0·05). Furthermore, ELE resulted in decreases in the triacylglycerol concentrations in the plasma (1·44 (sd 0·448)v.2·79 (sd 0·677) mmol/l (n7);P<0·05) and liver (19·1 (sd 5·07)v.44·1 (sd 16·28) μmol/g (n7);P<0·05). In contrast, ELE did not show any significant effects in the rats fed a starch diet. When rats were orally given ELE 10 min before fructose administration, the intestinal fructose absorption, which was examined by measuring the elevated concentration of fructose in the portal vein at 30 min after the fructose administration, was significantly inhibited in a dose-dependent manner. Furthermore, in rats fed a high-fructose diet, the plasma and hepatic triacylglycerol concentrations were significantly decreased by ELE. These results indicate that ELE, which inhibits the intestinal fructose absorption, can suppress adiposity in rats that ingest large amounts of sucrose or fructose.

scholarly journals Effect of High‐Sucrose and High‐Fructose Diets on Adipose Tissue Dysfunction and Metabolic Syndrome

2018 ◽  
Vol 32 (S1) ◽  
Author(s):  
Anil Sakamuri ◽  
Sugeedha Jeyapal ◽  
Suryam Reddy Kona ◽  
Sailaja Pothana ◽  
Ahamed Ibrahim
Keyword(s):  
Metabolic Syndrome ◽  
Adipose Tissue ◽  
Adipose Tissue Dysfunction ◽  
High Fructose ◽  
High Sucrose

scholarly journals Differential effects of a High‐Fructose diet or a High‐Fat/High‐Sucrose diet on the mitochondrial ROS production and respiration in rat liver and skeletal muscle

2012 ◽  
Vol 26 (S1) ◽  
Author(s):  
Karine Couturier ◽  
Marie Le Guen ◽  
Charles Coudray ◽  
Christine Feillet-Coudray ◽  
Chantal Wrutniak-Cabello ◽  
...  
Keyword(s):  
Skeletal Muscle ◽  
Rat Liver ◽  
Ros Production ◽  
High Fat ◽  
High Fructose Diet ◽  
Mitochondrial Ros ◽  
High Fructose ◽  
Sucrose Diet ◽  
High Sucrose Diet ◽  
High Sucrose

Hepatic Transcriptomic Signature of High-Sucrose Diet in Nonobese Model of Metabolic Syndrome and Prediabetes

Diabetes ◽  
2018 ◽  
Vol 67 (Supplement 1) ◽  
pp. 1723-P
Author(s):  
IRENA MARKOVÁ ◽  
MARTINA HÜTTL ◽  
HANA MALINSKA ◽  
ONDREJ SEDA ◽  
LUDMILA KAZDOVA
Keyword(s):  
Metabolic Syndrome ◽  
Sucrose Diet ◽  
Transcriptomic Signature ◽  
High Sucrose Diet ◽  
High Sucrose

The Use of High - Sucrose Culture Media for the Identification of Oral Streptococci in Infant- Mother Pairs

2017 ◽  
Vol 68 (11) ◽  
pp. 2691-2693
Author(s):  
Krisztina Martha ◽  
Cristina Bica ◽  
Edva Anna Frunda
Keyword(s):  
Culture Media ◽  
Bacterial Flora ◽  
Mutans Streptococci ◽  
Resistant Bacteria ◽  
Oral Streptococci ◽  
Streptococcus Sobrinus ◽  
Sugar Intake ◽  
Tooth Surfaces ◽  
Refined Carbohydrates ◽  
High Sucrose

By the end of the 60�s, the theory that refined carbohydrates promotes the absorption of saccharolytic Gram-positive microbial species on the tooth surfaces has become generally. Mutans streptococci (Streptococcus mutans and Streptococcus sobrinus) were key players in this theory. On agar plates, Str. mutans produces small, circular colonies, in the presence of glucose, and in the presence of sucrose large, sticky, gelatinous colonies. This gelatinous texture is due to the shell material: mutant 1 � 3 glucose polymers and dextran 1 �! 6 glucose polymers. Str. mutans are able to survive in the oral cavity with a pH lower than 5.5. That is why consecutive multiple sugar intake promotes the colonization of Str. mutans, which results in dental caries in stagnant zones. As oral pH is continuously shifted to acid, more acid-resistant bacteria appear. Our aim was to identify species in infant-mother pair gingival crevicular bacterial flora, which can be detected on high-sucrose culture media and to underline the jeopardy of vertical oral contamination from mother to infant.

THE INTENSITY OF OXIDATIVE MODIFICATION OF PROTEINS AND PEROXIDATION OF LIPIDS IN THE BLOOD SERUM, BRAIN, LIVER AND PANCREAS TISSUES OF RATS WITH INSULIN RESISTANCE AND CONGENITAL IODINE DEFICIENCY

2019 ◽  
pp. 115-120
Author(s):  
V. B. Stetsevyat ◽  
N. M. Voronych-Semchenko
Keyword(s):  
Insulin Resistance ◽  
Iodine Deficiency ◽  
Thiobarbituric Acid ◽  
Oxidative Modification ◽  
Peroxidation Of Lipids ◽  
Diene Conjugates ◽  
Oxidative Modification Of Proteins ◽  
High Fructose ◽  
Liver And Pancreas ◽  
Dependent Processes

The results of the study about the nature of oxygen-dependent processes in rats that were on a high-fructose diet for 8 weeks under conditions of adequate iodine supply and congenital iodine deprivation is presented in the article. The significant activation of peroxide oxidation of proteins (by increasing the aldo- and keto-derivates of a neutral nature) and lipids (increased of diene conjugates content and products, that are responsible to thiobarbituric acid) of animals with insulin resistance was found. An aggravating factor in the course of these processes is a congenital iodine deficiency. The changes of oxidative modification of proteins in animals with congenital iodine deficiency had multidirectional character, and processes of lipid peroxidation mainly increased. The most pronounced changes of the studied processes were observed in the liver and pancreas of animals with insulin resistance against the background of congenital iodine deficiency.

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